Modulation of hepatitis C virus RNA abundance and the isoprenoid biosynthesis pathway by microRNA miR-122 involves distinct mechanisms - sprookjes - yvandenbroek - TriplyDB

Modulation of hepatitis C virus RNA abundance and the isoprenoid biosynthesis pathway by microRNA miR-122 involves distinct mechanisms

Modulation of hepatitis C virus RNA abundance and the isoprenoid biosynthesis pathway by microRNA miR-122 involves distinct mechanisms

http://www.wikidata.org/entity/Q24653950

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Regulation of hepatitis C virus translation and infectious virus production by the microRNA miR-122 MicroRNA biogenesis and their functions in regulating stem cell potency and differentiation Metabolic alterations and hepatitis C: From bench to bedside Targeting viral infection by microRNA inhibition microRNA-122 abundance in hepatocellular carcinoma and non-tumor liver tissue from Japanese patients with persistent HCV versus HBV infection.MicroRNAs in metabolism and metabolic disorders Human metapneumovirus infection induces significant changes in small noncoding RNA expression in airway epithelial cells.miR-139-5p modulates cortical neuronal migration by targeting Lis1 in a rat model of focal cortical dysplasia miR-122 does not modulate the elongation phase of hepatitis C virus RNA synthesis in isolated replicase complexes.Alcohol facilitates HCV RNA replication via up-regulation of miR-122 expression and inhibition of cyclin G1 in human hepatoma cells Inhibition of alpha interferon (IFN-α)-induced microRNA-122 negatively affects the anti-hepatitis B virus efficiency of IFN-α.Unconventional miR-122 binding stabilizes the HCV genome by forming a trimolecular RNA structure.MiR-125b reduces porcine reproductive and respiratory syndrome virus replication by negatively regulating the NF-κB pathway Masking the 5' terminal nucleotides of the hepatitis C virus genome by an unconventional microRNA-target RNA complex.End game: getting the most out of microRNAs Liver-specific microRNA-122: Biogenesis and function Coxsackievirus B3 engineered to contain microRNA targets for muscle-specific microRNAs displays attenuated cardiotropic virulence in mice.miR-122 stimulates hepatitis C virus RNA synthesis by altering the balance of viral RNAs engaged in replication versus translation Transient replication of Hepatitis C Virus sub-genomic RNA in murine cell lines is enabled by miR-122 and varies with cell passage miR-122 activates hepatitis C virus translation by a specialized mechanism requiring particular RNA components.Targeting microRNA-122 to Treat Hepatitis C Virus Infection.Dual regulation of hepatitis C viral RNA by cellular RNAi requires partitioning of Ago2 to lipid droplets and P-bodies The porcine microRNA transcriptome response to transmissible gastroenteritis virus infection The emerging role of microRNAs in drug responses Stabilization of hepatitis C virus RNA by an Ago2-miR-122 complex.Regulation of Hepatitis C Virus Genome Replication by Xrn1 and MicroRNA-122 Binding to Individual Sites in the 5' Untranslated Region.Relationships of PBMC microRNA expression, plasma viral load, and CD4+ T-cell count in HIV-1-infected elite suppressors and viremic patients Lipid metabolite profiling identifies desmosterol metabolism as a new antiviral target for hepatitis C virus.MicroRNA-122 plays a critical role in liver homeostasis and hepatocarcinogenesis Modulation of hepatitis C virus RNA abundance and virus release by dispersion of processing bodies and enrichment of stress granules.Competing and noncompeting activities of miR-122 and the 5' exonuclease Xrn1 in regulation of hepatitis C virus replication.Recent insights and novel bioinformatics tools to understand the role of microRNAs binding to 5' untranslated region.Hepatitis B virus mRNA-mediated miR-122 inhibition upregulates PTTG1-binding protein, which promotes hepatocellular carcinoma tumor growth and cell invasion.Virus-specific mechanisms of carcinogenesis in hepatitis C virus associated liver cancer MiR-122 in hepatic function and liver diseases.Modulation of GB virus B RNA abundance by microRNA-122: dependence on and escape from microRNA-122 restriction.Beneficial IL28B genotype associated with lower frequency of hepatic steatosis in patients with chronic hepatitis C Involvement of eukaryotic small RNA pathways in host defense and viral pathogenesis Enhancement of hepatitis C viral RNA abundance by precursor miR-122 molecules.TIM-1 Promotes Hepatitis C Virus Cell Attachment and Infection.

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P2860

Modulation of hepatitis C virus RNA abundance and the isoprenoid biosynthesis pathway by microRNA miR-122 involves distinct mechanisms

http://www.wikidata.org/entity/Q24653950

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

@zh-mo

2010年論文

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2010年论文

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name

Modulation of hepatitis C viru ...... 2 involves distinct mechanisms

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Modulation of hepatitis C viru ...... 2 involves distinct mechanisms

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Modulation of hepatitis C viru ...... 2 involves distinct mechanisms

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type

label

Modulation of hepatitis C viru ...... 2 involves distinct mechanisms

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Modulation of hepatitis C viru ...... 2 involves distinct mechanisms

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Modulation of hepatitis C viru ...... 2 involves distinct mechanisms

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Modulation of Hepatitis C Viru ...... 2 Involves Distinct Mechanisms

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Modulation of hepatitis C viru ...... 2 involves distinct mechanisms

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Modulation of hepatitis C viru ...... 2 involves distinct mechanisms

@en

Modulation of hepatitis C viru ...... 2 involves distinct mechanisms

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P1433

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Journal of Virology

P1476

Modulation of hepatitis C viru ...... 2 involves distinct mechanisms

@en

P2093

Kara L Norman

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Peter Sarnow

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P2860

Principles of microRNA-target recognition

Identification of FBL2 as a geranylgeranylated cellular protein required for hepatitis C virus RNA replication

Antagonism of microRNA-122 in mice by systemically administered LNA-antimiR leads to up-regulation of a large set of predicted target mRNAs in the liver

microRNA-122 stimulates translation of hepatitis C virus RNA

Initiation of hepatitis C virus infection is dependent on cholesterol and cooperativity between CD81 and scavenger receptor B type I

Selectable subgenomic and genome-length dicistronic RNAs derived from an infectious molecular clone of the HCV-N strain of hepatitis C virus replicate efficiently in cultured Huh7 cells.

Nonlinear partial differential equations and applications: Genomic analysis of the host response to hepatitis C virus infection

Hepatitis C virus production by human hepatocytes dependent on assembly and secretion of very low-density lipoproteins

STEATOSIS IN CHRONIC HEPATITIS C: WHY DOES IT REALLY MATTER?

Hepatitis C Virus Induces Proteolytic Cleavage of Sterol Regulatory Element Binding Proteins and Stimulates Their Phosphorylation via Oxidative Stress

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666-70

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scholarly article

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10.1128/JVI.01156-09

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1

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84

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2010-01-01T00:00:00Z

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38027403

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19846523

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P932

2798415

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