Modulation of adverse cardiac remodeling by STARS, a mediator of MEF2 signaling and SRF activity - sprookjes - yvandenbroek - TriplyDB

Modulation of adverse cardiac remodeling by STARS, a mediator of MEF2 signaling and SRF activity

Modulation of adverse cardiac remodeling by STARS, a mediator of MEF2 signaling and SRF activity

http://www.wikidata.org/entity/Q24673391

about

Protein kinase C-related kinase targets nuclear localization signals in a subset of class IIa histone deacetylases MURC, a muscle-restricted coiled-coil protein that modulates the Rho/ROCK pathway, induces cardiac dysfunction and conduction disturbance miR-9 and NFATc3 regulate myocardin in cardiac hypertrophy STARS is essential to maintain cardiac development and function in vivo via a SRF pathway Mechanosensitive Molecular Networks Involved in Transducing Resistance Exercise-Signals into Muscle Protein Accretion Myozap, a novel intercalated disc protein, activates serum response factor-dependent signaling and is required to maintain cardiac function in vivo The cardiac pacemaker-specific channel Hcn4 is a direct transcriptional target of MEF2 TRPC3-GEF-H1 axis mediates pressure overload-induced cardiac fibrosis Severe myopathy in mice lacking the MEF2/SRF-dependent gene leiomodin-3 MRTF-A controls vessel growth and maturation by increasing the expression of CCN1 and CCN2.Disassociation of insulin action and Akt/FOXO signaling in skeletal muscle of older Akt-deficient mice.Comparative in silico analysis identifies bona fide MyoD binding sites within the Myocyte stress 1 gene promoter.Kainic Acid Induces mTORC1-Dependent Expression of Elmo1 in Hippocampal Neurons.Eccentric exercise activates novel transcriptional regulation of hypertrophic signaling pathways not affected by hormone changes.Lamina-associated polypeptide 2alpha loss impairs heart function and stress response in mice.Molecular basis for antagonism between PDGF and the TGFbeta family of signalling pathways by control of miR-24 expression Regulation of PI3-kinase/Akt signaling by muscle-enriched microRNA-486 Myocardin-related transcription factor-a controls myofibroblast activation and fibrosis in response to myocardial infarction.The intriguing regulators of muscle mass in sarcopenia and muscular dystrophy.Linking actin dynamics and gene transcription to drive cellular motile functions.Twist1 transcriptional targets in the developing atrio-ventricular canal of the mouse Increased SRF transcriptional activity in human and mouse skeletal muscle is a signature of insulin resistance Actin-binding Rho activating protein is expressed in the central nervous system of normal adult rats.Regulation of DMD pathology by an ankyrin-encoded miRNA.Qiliqiangxin inhibits angiotensin II-induced transdifferentiation of rat cardiac fibroblasts through suppressing interleukin-6 Functional importance of cardiac enhancer-associated noncoding RNAs in heart development and disease The Cardiac Stress Response Factor Ms1 Can Bind to DNA and Has a Function in the Nucleus Cardiac expression of ms1/STARS, a novel gene involved in cardiac development and disease, is regulated by GATA4.The regulation and function of the striated muscle activator of rho signaling (STARS) protein.Overexpression of Striated Muscle Activator of Rho Signaling (STARS) Increases C2C12 Skeletal Muscle Cell Differentiation LRRC10 is required to maintain cardiac function in response to pressure overload.Deletion of Fn14 receptor protects from right heart fibrosis and dysfunction The contribution of cellular mechanotransduction to cardiomyocyte form and function.Mechanical stress-induced sarcomere assembly for cardiac muscle growth in length and width.MicroRNAs as biomarkers of disease onset.The actin-MRTF-SRF gene regulatory axis and myofibroblast differentiation.The STARS signaling pathway: a key regulator of skeletal muscle function.Current understanding of sarcopenia: possible candidates modulating muscle mass.Regulation of STARS and its downstream targets suggest a novel pathway involved in human skeletal muscle hypertrophy and atrophy.Regulation of Connective Tissue Growth Factor and Cardiac Fibrosis by an SRF/MicroRNA-133a Axis

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Modulation of adverse cardiac remodeling by STARS, a mediator of MEF2 signaling and SRF activity

http://www.wikidata.org/entity/Q24673391

description

2007 nî lūn-bûn

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2007 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2007 թվականի մայիսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年论文

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Modulation of adverse cardiac ...... EF2 signaling and SRF activity

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Modulation of adverse cardiac ...... EF2 signaling and SRF activity

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Modulation of adverse cardiac ...... EF2 signaling and SRF activity

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Modulation of adverse cardiac ...... EF2 signaling and SRF activity

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Modulation of adverse cardiac ...... EF2 signaling and SRF activity

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Modulation of adverse cardiac ...... EF2 signaling and SRF activity

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Modulation of adverse cardiac ...... EF2 signaling and SRF activity

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Modulation of adverse cardiac ...... EF2 signaling and SRF activity

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Modulation of adverse cardiac ...... EF2 signaling and SRF activity

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Journal of Clinical Investigation

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Modulation of adverse cardiac ...... EF2 signaling and SRF activity

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P2093

Eric N Olson

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Gordon C Teg Pipes

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James A Richardson

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John McAnally

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Joseph A Hill

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Koichiro Kuwahara

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Potentiation of serum response factor activity by a family of myocardin-related transcription factors.

The cardiac mechanical stretch sensor machinery involves a Z disc complex that is defective in a subset of human dilated cardiomyopathy

Muscle-specific signaling mechanism that links actin dynamics to serum response factor.

Serum response factor-GATA ternary complex required for nuclear signaling by a G-protein-coupled receptor

The neuron-restrictive silencer element-neuron-restrictive silencer factor system regulates basal and endothelin 1-inducible atrial natriuretic peptide gene expression in ventricular myocytes

Extracellular signal-regulated kinase 2 interacts with and is negatively regulated by the LIM-only protein FHL2 in cardiomyocytes

A new myocyte-specific enhancer-binding factor that recognizes a conserved element associated with multiple muscle-specific genes

Myocardin is a master regulator of smooth muscle gene expression

Megakaryoblastic leukemia 1, a potent transcriptional coactivator for serum response factor (SRF), is required for serum induction of SRF target genes

The genetic basis for cardiomyopathy: from mutation identification to mechanistic paradigms

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1324-34

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scholarly article

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10.1172/JCI31240

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5

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117

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Rhonda Bassel-Duby

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2007-05-01T00:00:00Z

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17415416

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1838928

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