Induction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposure - sprookjes - yvandenbroek - TriplyDB

Induction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposure

Induction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposure

http://www.wikidata.org/entity/Q24682426

about

Plastics derived endocrine disruptors (BPA, DEHP and DBP) induce epigenetic transgenerational inheritance of obesity, reproductive disease and sperm epimutations In utero exposure to bisphenol-A and anogenital distance of male offspring Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses Impact of oral bisphenol A at reference doses on intestinal barrier function and sex differences after perinatal exposure in rats Bisphenol-A and the great divide: a review of controversies in the field of endocrine disruption Endocrine-disrupting chemicals: an Endocrine Society scientific statement Why public health agencies cannot depend on good laboratory practices as a criterion for selecting data: the case of bisphenol A Does breast cancer start in the womb?Oral exposure to bisphenol a increases dimethylbenzanthracene-induced mammary cancer in rats Prenatal exposure to bisphenol a at environmentally relevant doses adversely affects the murine female reproductive tract later in life In vivo effects of bisphenol A in laboratory rodent studies Historical review of the causes of cancer Progesterone and Overlooked Endocrine Pathways in Breast Cancer Pathogenesis Environmental endocrine disruptors: Effects on the human male reproductive system Environmental causes of cancer: endocrine disruptors as carcinogens Endocrine-disrupting chemicals: associated disorders and mechanisms of action Ranking transitive chemical-disease inferences using local network topology in the comparative toxicogenomics database Concentrations of environmental phenols and parabens in milk, urine and serum of lactating North Carolina women Differential effects of bisphenol A and diethylstilbestrol on human, rat and mouse fetal leydig cell function Estrogens in the wrong place at the wrong time: Fetal BPA exposure and mammary cancer Carcinogenetic mechanisms of endocrine disruptors in female cancers (Review)Nuclear receptors and endocrine disruptors in fetal and neonatal testes: a gapped landscape.Histological analysis of low dose NMU effects in the rat mammary gland.Predicting environmental chemical factors associated with disease-related gene expression data Bisphenol A: Perinatal exposure and body weight Neoplasia as development gone awry: the role of endocrine disruptors.Perinatal exposure to bisphenol a increases adult mammary gland progesterone response and cell number.Benzene and 2-ethyl-phthalate induce proliferation in normal rat pituitary cells.Prenatal exposure to BPA alters the epigenome of the rat mammary gland and increases the propensity to neoplastic development Female reproductive disorders: the roles of endocrine-disrupting compounds and developmental timing Designing Endocrine Disruption Out of the Next Generation of Chemicals Epigenetic influences of low-dose bisphenol A in primary human breast epithelial cells.Human excretion of bisphenol A: blood, urine, and sweat (BUS) study.In utero exposure to bisphenol A shifts the window of susceptibility for mammary carcinogenesis in the rat Endocrine-disrupting chemicals and public health protection: a statement of principles from The Endocrine Society A novel pathogenic classification of cancers.Low-dose BPA exposure alters the mesenchymal and epithelial transcriptomes of the mouse fetal mammary gland.Exposure to the Endocrine Disruptor Bisphenol A Alters Susceptibility for Mammary Cancer An investigation of the endocrine-disruptive effects of bisphenol a in human and rat fetal testes.In utero exposure to diethylstilbestrol (DES) or bisphenol-A (BPA) increases EZH2 expression in the mammary gland: an epigenetic mechanism linking endocrine disruptors to breast cancer

P2860

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P2860

Induction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposure

http://www.wikidata.org/entity/Q24682426

description

2007 nî lūn-bûn

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2007 թուականին հրատարակուած գիտական յօդուած

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2007 թվականին հրատարակված գիտական հոդված

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2007年の論文

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Induction of mammary gland duc ...... ing fetal bisphenol A exposure

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Induction of mammary gland duc ...... ing fetal bisphenol A exposure

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J.REPROTOX.2006.10.002

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Reproductive Toxicology

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Induction of mammary gland duc ...... ing fetal bisphenol A exposure

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Ana M Soto

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Angelo A Ucci

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Carlos Sonnenschein

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Maricel V Maffini

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Tessa J Murray

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P2860

Urinary concentrations of bisphenol A and 4-nonylphenol in a human reference population

Relative binding affinity-serum modified access (RBA-SMA) assay predicts the relative in vivo bioactivity of the xenoestrogens bisphenol A and octylphenol

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4

Exposure to environmentally relevant doses of the xenoestrogen bisphenol-A alters development of the fetal mouse mammary gland

Parent bisphenol A accumulation in the human maternal-fetal-placental unit

Perinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levels

Exposure to bisphenol A advances puberty

Estrogenicity of resin-based composites and sealants used in dentistry

Regulation of Msx-1, Msx-2, Bmp-2 and Bmp-4 during foetal and postnatal mammary gland development

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383-90

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10.1016/J.REPROTOX.2006.10.002

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