Early correlation of microglial activation with enhanced tumor necrosis factor-alpha and monocyte chemoattractant protein-1 expression specifically within the entorhinal cortex of triple transgenic Alzheimer's disease mice - sprookjes - yvandenbroek - TriplyDB

Early correlation of microglial activation with enhanced tumor necrosis factor-alpha and monocyte chemoattractant protein-1 expression specifically within the entorhinal cortex of triple transgenic Alzheimer's disease mice

Early correlation of microglial activation with enhanced tumor necrosis factor-alpha and monocyte chemoattractant protein-1 expression specifically within the entorhinal cortex of triple transgenic Alzheimer's disease mice

http://www.wikidata.org/entity/Q24813024

about

TNF signaling inhibition in the CNS: implications for normal brain function and neurodegenerative disease Perispinal etanercept produces rapid improvement in primary progressive aphasia: identification of a novel, rapidly reversible TNF-mediated pathophysiologic mechanism Amyloidosis in Retinal Neurodegenerative Diseases TNF-α/NF-κB signaling in the CNS: possible connection to EPHB2 Tau-tubulin kinase Rodent models of neuroinflammation for Alzheimer's disease Innate immunity in Alzheimer's disease: a complex affair The flavonoid quercetin ameliorates Alzheimer's disease pathology and protects cognitive and emotional function in aged triple transgenic Alzheimer's disease model mice Rapid cognitive improvement in Alzheimer's disease following perispinal etanercept administration.Reduced pathology and improved behavioral performance in Alzheimer's disease mice vaccinated with HSV amplicons expressing amyloid-beta and interleukin-4.Treatment with a γ-ketoaldehyde scavenger prevents working memory deficits in hApoE4 mice.Intracerebroventricular streptozotocin exacerbates Alzheimer-like changes of 3xTg-AD mice Detailed immunohistochemical characterization of temporal and spatial progression of Alzheimer's disease-related pathologies in male triple-transgenic mice Tumor necrosis factor α Inhibition for Alzheimer's Disease.Reductions in amyloid-beta-derived neuroinflammation, with minocycline, restore cognition but do not significantly affect tau hyperphosphorylation.Neuroinflammation and related neuropathologies in APPSL mice: further value of this in vivo model of Alzheimer's disease.Implications of gliotransmission for the pharmacotherapy of CNS disorders.Selective vulnerability of neurons in layer II of the entorhinal cortex during aging and Alzheimer's disease.Inhibition of soluble TNF signaling in a mouse model of Alzheimer's disease prevents pre-plaque amyloid-associated neuropathology.CX3CR1 deficiency alters microglial activation and reduces beta-amyloid deposition in two Alzheimer's disease mouse models.CCR6: a biomarker for Alzheimer's-like disease in a triple transgenic mouse model Long-term treatment of thalidomide ameliorates amyloid-like pathology through inhibition of β-secretase in a mouse model of Alzheimer's disease.Hippocampal expression of murine TNFα results in attenuation of amyloid deposition in vivo.Neurovascular dysfunction, inflammation and endothelial activation: implications for the pathogenesis of Alzheimer's disease.Granulocyte colony stimulating factor decreases brain amyloid burden and reverses cognitive impairment in Alzheimer's mice.Tumour necrosis factor-α (TNF-α) and miRNA expression in frontal and temporal neocortex in Alzheimer's disease and the effect of TNF-α on miRNA expression in vitro The evolving biology of microglia in Alzheimer's disease.Ablation of TNF-RI/RII expression in Alzheimer's disease mice leads to an unexpected enhancement of pathology: implications for chronic pan-TNF-α suppressive therapeutic strategies in the brain.The classification of microglial activation phenotypes on neurodegeneration and regeneration in Alzheimer's disease brain.Regulation of cyclic AMP response element-binding protein during neuroglial interactions.Minocycline corrects early, pre-plaque neuroinflammation and inhibits BACE-1 in a transgenic model of Alzheimer's disease-like amyloid pathology.Early intervention with a small molecule inhibitor for tumor necrosis factor-α prevents cognitive deficits in a triple transgenic mouse model of Alzheimer's disease Tumor necrosis factor-α synthesis inhibitor 3,6'-dithiothalidomide attenuates markers of inflammation, Alzheimer pathology and behavioral deficits in animal models of neuroinflammation and Alzheimer's disease Targeting TNF-α to elucidate and ameliorate neuroinflammation in neurodegenerative diseases.Brain mediators of systemic oxidative stress on perceptual impairments in Parkinson's disease.Elevated plasma MCP-1 concentration following traumatic brain injury as a potential "predisposition" factor associated with an increased risk for subsequent development of Alzheimer's disease.A non-transgenic mouse model (icv-STZ mouse) of Alzheimer's disease: similarities to and differences from the transgenic model (3xTg-AD mouse).Age-related changes to tumor necrosis factor receptors affect neuron survival in the presence of beta-amyloid.Blocking soluble tumor necrosis factor signaling with dominant-negative tumor necrosis factor inhibitor attenuates loss of dopaminergic neurons in models of Parkinson's disease Microglial dysfunction and defective beta-amyloid clearance pathways in aging Alzheimer's disease mice.

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Early correlation of microglial activation with enhanced tumor necrosis factor-alpha and monocyte chemoattractant protein-1 expression specifically within the entorhinal cortex of triple transgenic Alzheimer's disease mice

http://www.wikidata.org/entity/Q24813024

description

2005 nî lūn-bûn

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2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Early correlation of microglia ...... genic Alzheimer's disease mice

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Early correlation of microglia ...... genic Alzheimer's disease mice

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Early correlation of microglia ...... genic Alzheimer's disease mice

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Early correlation of microglia ...... genic Alzheimer's disease mice

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Early correlation of microglia ...... genic Alzheimer's disease mice

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Early correlation of microglia ...... genic Alzheimer's disease mice

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Early correlation of microglia ...... genic Alzheimer's disease mice

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Early correlation of microglia ...... genic Alzheimer's disease mice

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Early correlation of microglia ...... genic Alzheimer's disease mice

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Journal of Neuroinflammation

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Early correlation of microglia ...... genic Alzheimer's disease mice

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Frank M LaFerla

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Howard J Federoff

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Michael A Mastrangelo

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Michelle C Janelsins

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Salvatore Oddo

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William J Bowers

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P2860

Down-regulation of DENN/MADD, a TNF receptor binding protein, correlates with neuronal cell death in Alzheimer's disease brain and hippocampal neurons

Microglial responses to amyloid beta peptide opsonization and indomethacin treatment.

Neuropathological stageing of Alzheimer-related changes

Alzheimer's disease is a synaptic failure

Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction

Beta-amyloid protein structure determines the nature of cytokine release from rat microglia.

The role of TNF and its receptors in Alzheimer's disease.

Microglia as a source and target of cytokines.

Plaque-independent disruption of neural circuits in Alzheimer's disease mouse models.

Overexpression of monocyte chemotactic protein-1/CCL2 in beta-amyloid precursor protein transgenic mice show accelerated diffuse beta-amyloid deposition

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10.1186/1742-2094-2-23

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16232318

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Alzheimer's disease

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1276812

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