Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer - sprookjes - yvandenbroek - TriplyDB

Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

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Molecular mechanisms underlying resistance to androgen deprivation therapy in prostate cancer Molecular signaling involving intrinsically disordered proteins in prostate cancer A combination of sorafenib and nilotinib reduces the growth of castrate-resistant prostate cancer.BAY 1024767 blocks androgen receptor mutants found in castration-resistant prostate cancer patients Androgen/androgen receptor axis maintains and promotes cancer cell stemness through direct activation of Nanog transcription in hepatocellular carcinoma.Src promotes castration-recurrent prostate cancer through androgen receptor-dependent canonical and non-canonical transcriptional signatures.Androgen receptor CAG and GGN repeat length variation contributes more to the tumorigenesis of osteosarcoma.Suppression of miR-4735-3p in androgen receptor-expressing prostate cancer cells increases cell death during chemotherapy.The activation of OR51E1 causes growth suppression of human prostate cancer cells.Interplay between steroid hormone activation of the unfolded protein response and nuclear receptor action.The impact of interleukin-10 (IL-10) gene 4 polymorphisms on peripheral blood IL-10 variation and prostate cancer risk based on published studies.The context of prostate cancer genomics in personalized medicine.Prostate-specific membrane antigen cleavage of vitamin B9 stimulates oncogenic signaling through metabotropic glutamate receptors.Influence of Androgen Deprivation Therapy on the Uptake of PSMA-Targeted Agents: Emerging Opportunities and Challenges.Role of 20-Hydroxyeicosatetraenoic Acid (20-HETE) in Androgen-Mediated Cell Viability in Prostate Cancer Cells.Androgen receptor reverts dexamethasone‑induced inhibition of prostate cancer cell proliferation and migration.Comprehensive immunohistochemical analysis of PD-L1 shows scarce expression in castration-resistant prostate cancer.Inhibiting Multiple Deubiquitinases to Reduce Androgen Receptor Expression in Prostate Cancer Cells

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Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

http://www.wikidata.org/entity/Q26795625

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2015 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

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Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

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Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

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Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

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Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

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Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

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Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

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Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

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Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer

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Andres Jan Schrader

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Aria Baniahmad

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Helmut Klocker

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Marcus V Cronauer

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Creative Commons Attribution 2.5 Unported

P2860

Regression of castrate-recurrent prostate cancer by a small-molecule inhibitor of the amino-terminus domain of the androgen receptor

Opitz G/BBB syndrome, a defect of midline development, is due to mutations in a new RING finger gene on Xp22

EBP1, an ErbB3-binding protein, is decreased in prostate cancer and implicated in hormone resistance

The impact of microRNAs on protein output

From HER2/Neu signal cascade to androgen receptor and its coactivators: a novel pathway by induction of androgen target genes through MAP kinase in prostate cancer cells

Metformin therapy and risk of cancer in patients with type 2 diabetes: systematic review

Biologic and clinical significance of androgen receptor variants in castration resistant prostate cancer

Androgen receptor activation in castration-recurrent prostate cancer: the role of Src-family and Ack1 tyrosine kinases

Beyond T and DHT - novel steroid derivatives capable of wild type androgen receptor activation

SRC: marker or actor in prostate cancer aggressiveness

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10.18632/ONCOTARGET.4689

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intracellular receptor

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