NADPH oxidases in heart failure: poachers or gamekeepers?
about
Diagnostic, Prognostic, and Therapeutic Value of Circulating miRNAs in Heart Failure Patients Associated with Oxidative StressNovel Perspectives in Redox Biology and Pathophysiology of Failing Myocytes: Modulation of the Intramyocardial Redox Milieu for Therapeutic Interventions-A Review Article from the Working Group of Cardiac Cell Biology, Italian Society of CardiologyRole of redoximiRs in fibrogenesisNADPH oxidases in lung health and diseasePolymorphism of rs1836882 in NOX4 gene modifies associations between dietary caloric intake and ROS levels in peripheral blood mononuclear cellsQishenyiqi protects ligation-induced left ventricular remodeling by attenuating inflammation and fibrosis via STAT3 and NF-κB signaling pathwayAngiotensin II reduces cardiac AdipoR1 expression through AT1 receptor/ROS/ERK1/2/c-Myc pathwayPrevention of exercised induced cardiomyopathy following Pip-PMO treatment in dystrophic mdx mice.TRPC3 positively regulates reactive oxygen species driving maladaptive cardiac remodeling.The quest for selective nox inhibitors and therapeutics: challenges, triumphs and pitfallsCardiac ryanodine receptor activation by a high Ca²⁺ store load is reversed in a reducing cytoplasmic redox environment.Lipid-induced NOX2 activation inhibits autophagic flux by impairing lysosomal enzyme activityMyocardial fibrosis induced by exposure to subclinical lipopolysaccharide is associated with decreased miR-29c and enhanced NOX2 expression in miceRedox signalling and cardioprotection: translatability and mechanismHemoglobin-associated oxidative stress in the pericardial compartment of postoperative cardiac surgery patients.Contractile Function During Angiotensin-II Activation: Increased Nox2 Activity Modulates Cardiac Calcium Handling via Phospholamban Phosphorylation.Hepatocyte Nicotinamide Adenine Dinucleotide Phosphate Reduced Oxidase 4 Regulates Stress Signaling, Fibrosis, and Insulin Sensitivity During Development of Steatohepatitis in Mice.Syndecan-4 shedding is involved in the oxidative stress and inflammatory responses in left atrial tissue with valvular atrial fibrillationThe Role of Reactive Oxygen Species in β-Adrenergic Signaling in Cardiomyocytes from Mice with the Metabolic Syndrome.Intermittent hypoxia causes NOX2-dependent remodeling of atrial connexins.Pivotal role of miR-448 in the development of ROS-induced cardiomyopathy.QSYQ Attenuates Oxidative Stress and Apoptosis Induced Heart Remodeling Rats through Different Subtypes of NADPH-Oxidase.Molecular Mechanisms Linking Autonomic Dysfunction and Impaired Cardiac Contractility in Critical IllnessSestrin 2 and AMPK connect hyperglycemia to Nox4-dependent endothelial nitric oxide synthase uncoupling and matrix protein expression.Nox4 NADPH oxidase mediates peroxynitrite-dependent uncoupling of endothelial nitric-oxide synthase and fibronectin expression in response to angiotensin II: role of mitochondrial reactive oxygen species.NOX2 amplifies acetaldehyde-mediated cardiomyocyte mitochondrial dysfunction in alcoholic cardiomyopathyCell-specific effects of Nox2 on the acute and chronic response to myocardial infarction.Antioxidant therapies for the management of atrial fibrillation.Angiotensin receptor-mediated oxidative stress is associated with impaired cardiac redox signaling and mitochondrial function in insulin-resistant rats.Carbon monoxide pollution aggravates ischemic heart failure through oxidative stress pathway.p21-Activated kinase1 (Pak1) is a negative regulator of NADPH-oxidase 2 in ventricular myocytes.Targeting inflammation and oxidative stress in atrial fibrillation: role of 3-hydroxy-3-methylglutaryl-coenzyme a reductase inhibition with statins.Novel aspects of ROS signalling in heart failure.Redox balance and cardioprotection.ROS signalling between endothelial cells and cardiac cells.The human Nox4: gene, structure, physiological function and pathological significance.ZYZ-772 Prevents Cardiomyocyte Injury by Suppressing Nox4-Derived ROS Production and Apoptosis.Regulation of NADPH oxidases in skeletal muscle.Loss of myocardial retinoic acid receptor α induces diastolic dysfunction by promoting intracellular oxidative stress and calcium mishandling in adult mice.Polymorphisms in NADPH oxidase CYBA gene modify the risk of ESRD in patients with chronic glomerulonephritis.
P2860
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P2860
NADPH oxidases in heart failure: poachers or gamekeepers?
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2013 nî lūn-bûn
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2013 թուականի Մարտին հրատարակուած գիտական յօդուած
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2013 թվականի մարտին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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NADPH oxidases in heart failure: poachers or gamekeepers?
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NADPH oxidases in heart failure: poachers or gamekeepers?
@en
NADPH oxidases in heart failure: poachers or gamekeepers?
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NADPH oxidases in heart failure: poachers or gamekeepers?
@ast
NADPH oxidases in heart failure: poachers or gamekeepers?
@en
NADPH oxidases in heart failure: poachers or gamekeepers?
@nl
prefLabel
NADPH oxidases in heart failure: poachers or gamekeepers?
@ast
NADPH oxidases in heart failure: poachers or gamekeepers?
@en
NADPH oxidases in heart failure: poachers or gamekeepers?
@nl
P2860
P50
P3181
P356
P1476
NADPH oxidases in heart failure: poachers or gamekeepers?
@en
P2093
Alessia Perino
P2860
P304
P3181
P356
10.1089/ARS.2012.4550
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P577
2012-08-27T00:00:00Z