KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome - sprookjes - yvandenbroek - TriplyDB

KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

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Forkhead Box C1 Regulates Human Primary Keratinocyte Terminal Differentiation Recurrent and Sustained Viral Infections in Primary Immunodeficiencies.KLK5 and KLK7 Ablation Fully Rescues Lethality of Netherton Syndrome-Like Phenotype.Kallikrein-related peptidases (KLKs) as emerging therapeutic targets: focus on prostate cancer and skin pathologies.Genetics of eosinophilic esophagitis.Novel benzopyran derivatives and their therapeutic applications: a patent review (2009-2016).Inhibition of Human Kallikrein 5 Protease by Triterpenoids from Natural Sources.SPINK5(490-624) binds KLK5 Expression of serine protease inhibitors in epidermal keratinocytes is increased by calcium but not 1,25-dihydroxyvitamin D3 or retinoic acid.Daikenchuto (Da-Jian-Zhong-Tang) ameliorates intestinal fibrosis by activating myofibroblast transient receptor potential ankyrin 1 channel

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KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

http://www.wikidata.org/entity/Q27310680

description

2015 nî lūn-bûn

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2015 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

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KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

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KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

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KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

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KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

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KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

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prefLabel

KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

@ast

KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

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KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

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JOURNAL.PGEN.1005389

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KLK5 Inactivation Reverses Cutaneous Hallmarks of Netherton Syndrome

@en

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Georgia Sotiropoulou

http://www.w3.org/2001/XMLSchema#string

Georgios Pampalakis

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Laetitia Furio

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Purification, molecular cloning, and expression of a human stratum corneum trypsin-like serine protease with possible function in desquamation

Elastase 2 is expressed in human and mouse epidermis and impairs skin barrier function in Netherton syndrome through filaggrin and lipid misprocessing

Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides

Kallikrein-mediated proteolysis regulates the antimicrobial effects of cathelicidins in skin

A potential role for multiple tissue kallikrein serine proteases in epidermal desquamation

Kallikrein-related peptidase 5 functions in proteolytic processing of profilaggrin in cultured human keratinocytes

Epidermal detachment, desmosomal dissociation, and destabilization of corneodesmosin in Spink5-/- mice

Matriptase initiates activation of epidermal pro-kallikrein and disease onset in a mouse model of Netherton syndrome

Scratching the surface of skin development

LEKTI fragments specifically inhibit KLK5, KLK7, and KLK14 and control desquamation through a pH-dependent interaction

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e1005389

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scholarly article

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10.1371/JOURNAL.PGEN.1005389

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9

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11

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Alain Hovnanian

Iacovos P Michael

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2015-09-01T00:00:00Z

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282047377

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26390218

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4577096

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