Huntington's disease induced cardiac amyloidosis is reversed by modulating protein folding and oxidative stress pathways in the Drosophila heart
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Role for the Unfolded Protein Response in Heart Disease and Cardiac ArrhythmiasMethods to assess Drosophila heart development, function and agingTime-restricted feeding attenuates age-related cardiac decline in DrosophilaAge-dependent electrical and morphological remodeling of the Drosophila heart caused by hERG/seizure mutations.Neurocardiovascular deficits in the Q175 mouse model of Huntington's disease.Getting folded: chaperone proteins in muscle development, maintenance and disease.The Huntington's disease-related cardiomyopathy prevents a hypertrophic response in the R6/2 mouse model.The UNC-45 myosin chaperone: from worms to flies to vertebrates.Dysfunction of the CNS-heart axis in mouse models of Huntington's disease.PiZ mouse liver accumulates polyubiquitin conjugates that associate with catalytically active 26S proteasomesChallenging muscle homeostasis uncovers novel chaperone interactions in Caenorhabditis elegansCardiac Dysfunction in the BACHD Mouse Model of Huntington's Disease.Molecular mechanisms of heart failure: insights from DrosophilaAzadiradione ameliorates polyglutamine expansion disease in Drosophila by potentiating DNA binding activity of heat shock factor 1.Do glial cells play an anti-oxidative role in Huntington's disease?Drosophila as a potential model to ameliorate mutant Huntington-mediated cardiac amyloidosisGenetic manipulation of cardiac ageing.Ayurvedic Amalaki Rasayana promotes improved stress tolerance and thus has anti-aging effects in Drosophila melanogaster.Proteostasis in cardiac health and disease.Huntington's disease is a multi-system disorder.Cardiac Fas-Dependent and Mitochondria-Dependent Apoptotic Pathways in a Transgenic Mouse Model of Huntington's Disease.A Statistical Framework for Pathway and Gene Identification from Integrative Analysis.Molecular Chaperones in Neurodegenerative Diseases: A Short Review.TRiC/CCT chaperonins are essential for maintaining myofibril organization, cardiac physiological rhythm, and lifespan.Time-restricted feeding for prevention and treatment of cardiometabolic disorders.Drosophila in the Heart of Understanding Cardiac Diseases: Modeling Channelopathies and Cardiomyopathies in the Fruitfly.Increasing autophagy and blocking Nrf2 suppress laminopathy-induced age-dependent cardiac dysfunction and shortened lifespan.Cardiac mTORC1 Dysregulation Impacts Stress Adaptation and Survival in Huntington's Disease.Neuro-Cardio Mechanisms in Huntington's Disease and Other Neurodegenerative Disorders.The Tiny for the Biggest Answers in Huntington's Disease
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P2860
Huntington's disease induced cardiac amyloidosis is reversed by modulating protein folding and oxidative stress pathways in the Drosophila heart
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2013 nî lūn-bûn
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2013 թուականին հրատարակուած գիտական յօդուած
@hyw
2013 թվականին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
@zh-mo
2013年論文
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2013年论文
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name
Huntington's disease induced c ...... thways in the Drosophila heart
@ast
Huntington's disease induced c ...... thways in the Drosophila heart
@en
Huntington's disease induced c ...... thways in the Drosophila heart
@nl
type
label
Huntington's disease induced c ...... thways in the Drosophila heart
@ast
Huntington's disease induced c ...... thways in the Drosophila heart
@en
Huntington's disease induced c ...... thways in the Drosophila heart
@nl
prefLabel
Huntington's disease induced c ...... thways in the Drosophila heart
@ast
Huntington's disease induced c ...... thways in the Drosophila heart
@en
Huntington's disease induced c ...... thways in the Drosophila heart
@nl
P2093
P2860
P921
P1433
P1476
Huntington's disease induced c ...... thways in the Drosophila heart
@en
P2093
Adriana S Trujillo
Girish C Melkani
Karen Ocorr
Raul Ramos
Rolf Bodmer
P2860
P304
P356
10.1371/JOURNAL.PGEN.1004024
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P577
2013-01-01T00:00:00Z