Hantavirus infection induces the expression of RANTES and IP-10 without causing increased permeability in human lung microvascular endothelial cells
about
Depletion of Alveolar Macrophages Does Not Prevent Hantavirus Disease Pathogenesis in Golden Syrian HamstersHantaviral Proteins: Structure, Functions, and Role in Hantavirus InfectionChoclo virus infection in the Syrian golden hamsterA comprehensive analysis of cytokine-induced and nuclear factor-kappa B-dependent genes in primary rat pancreatic beta-cellsImmunological mechanisms mediating hantavirus persistence in rodent reservoirs.Cytokine and chemokine levels in patients with severe fever with thrombocytopenia syndrome virus.Hypoxia induces permeability and giant cell responses of Andes virus-infected pulmonary endothelial cells by activating the mTOR-S6K signaling pathway.Vascular events in viral hemorrhagic fevers: a comparative study of dengue and hantaviruses.Multiplex Analysis of Serum Cytokines in Humans with Hantavirus Pulmonary Syndrome.Andes virus recognition of human and Syrian hamster beta3 integrins is determined by an L33P substitution in the PSI domain.Endothelial cell permeability and adherens junction disruption induced by junín virus infectionSeoul virus suppresses NF-kappaB-mediated inflammatory responses of antigen presenting cells from Norway rats.HantavirusesSeoul virus enhances regulatory and reduces proinflammatory responses in male Norway rats.Pathogenesis and host response in Syrian hamsters following intranasal infection with Andes virus.Hantavirus pulmonary syndrome: the new American hemorrhagic fever.Pathogenic and nonpathogenic hantaviruses differentially regulate endothelial cell responses.Differential antiviral response of endothelial cells after infection with pathogenic and nonpathogenic hantavirusesAndes virus disrupts the endothelial cell barrier by induction of vascular endothelial growth factor and downregulation of VE-cadherin.Induction of innate immune response genes by Sin Nombre hantavirus does not require viral replication.VEGFR2 and Src kinase inhibitors suppress Andes virus-induced endothelial cell permeabilityBunyamwera bunyavirus nonstructural protein NSs counteracts the induction of alpha/beta interferon.The pathogenic NY-1 hantavirus G1 cytoplasmic tail inhibits RIG-I- and TBK-1-directed interferon responses.Involvement of the Akt/NF-κB pathways in the HTNV-mediated increase of IL-6, CCL5, ICAM-1, and VCAM-1 in HUVECs.Expression and function of chemokines during viral infections: from molecular mechanisms to in vivo function.Elevated VEGF Levels in Pulmonary Edema Fluid and PBMCs from Patients with Acute Hantavirus Pulmonary Syndrome.Peripheral Organs of Dengue Fatal Cases Present Strong Pro-Inflammatory Response with Participation of IFN-Gamma-, TNF-Alpha- and RANTES-Producing Cells.Sustained high level of serum VEGF at convalescent stage contributes to the renal recovery after HTNV infection in patients with hemorrhagic fever with renal syndrome.Seoul virus-infected rat lung endothelial cells and alveolar macrophages differ in their ability to support virus replication and induce regulatory T cell phenotypes.Sex-dependent differences in plasma cytokine responses to hantavirus infection.Hantaviruses direct endothelial cell permeability by sensitizing cells to the vascular permeability factor VEGF, while angiopoietin 1 and sphingosine 1-phosphate inhibit hantavirus-directed permeability.Hantaan virus nucleocapsid protein binds to importin alpha proteins and inhibits tumor necrosis factor alpha-induced activation of nuclear factor kappa B.Hantavirus Infection Suppresses Thrombospondin-1 Expression in Cultured Endothelial Cells in a Strain-Specific Manner.A lethal disease model for hantavirus pulmonary syndrome in immunosuppressed Syrian hamsters infected with Sin Nombre virus.Hantaan virus infection induces CXCL10 expression through TLR3, RIG-I, and MDA-5 pathways correlated with the disease severityThe role of endothelial activation in dengue hemorrhagic fever and hantavirus pulmonary syndrome.T cells and pathogenesis of hantavirus cardiopulmonary syndrome and hemorrhagic fever with renal syndrome.The fundamental role of endothelial cells in hantavirus pathogenesis.Cell-type and donor-specific transcriptional responses to interferon-alpha. Use of customized gene arrays.Hantavirus-induced disruption of the endothelial barrier: neutrophils are on the payroll.
P2860
Q26314485-47AE8597-9933-4350-BE89-B863BECF95E4Q26774132-85BF37C9-19AE-4236-B995-4551E1931985Q27488441-2F6576A8-EEEB-4C43-B403-0A7741DCEBDAQ28572940-B677D3A0-0957-405D-A65F-365B904E620CQ33388243-293AD64E-9092-442D-9DB1-9C7BE8C375ECQ33402913-68DAD97A-BE7B-4AC1-B129-44280F55DE15Q33410566-986F916B-8B72-406A-B3B1-7DE40CCFE83CQ33413903-2EE58F2A-4265-4AF0-889A-81D5B1560F92Q33425939-6CF92C79-277E-4720-BCEA-F5D479093CD2Q33558721-EF7A253C-A347-486C-A85E-BA2386F8D037Q33719699-454141C9-1024-49B3-98B5-7A791562CB1BQ33722418-AE847FDA-9A81-4FAA-927E-1050899A06AEQ33894549-D36DBE85-4E18-46E7-AD4A-14BCF38B778FQ34091495-75658E61-B313-4DA5-8412-A991892FA154Q34109753-14985850-E846-4874-A7C7-7C91971AD999Q34122861-F58D35E6-2FC4-49B7-9F5D-EF1555B7660AQ34161805-BD5D920D-2D83-4BFB-A001-0633D31ACCB8Q34186187-B8CA6902-260F-4C35-92C4-356A7C01A547Q34190758-038D84CB-7392-4739-B921-9B84722B90AEQ34228610-CE9FF027-CCAB-401A-A692-ED3C446D2042Q34741912-1C8B040F-066A-4290-957D-A00DD54C2719Q35000433-131CEDE8-4605-4D8B-B39E-9B9442221BF3Q35101310-1ECF2A84-3470-4E91-9F66-592CDCA06D51Q35142776-7D02AC6E-6068-4DF3-A191-4275EC4B6636Q35209368-9854FC17-C75D-4DCA-9226-DBAFD5E9E309Q36203887-CA3BE48D-459D-43D2-AD2B-6D1BF7232618Q36231756-201B5A22-E5AB-4D51-A2FC-9607136734B2Q36337314-36AC2B47-4302-40B5-86E4-F77958A268E6Q36363282-F492A035-6283-4E75-B7CF-BB4ECAB679CEQ36672514-BCD1BC9A-1667-43E7-9689-2383C97A7DE9Q36673210-D93C777F-F230-499A-86D6-245238E1A59AQ37051589-023765F4-8747-422D-B76F-D56E65459E88Q37107257-39E52C32-E99D-46F3-98BC-5EAAE0CC4914Q37547578-E8F52B49-4A81-4F71-951B-2AA95AAF4F66Q37632276-7D069EF6-820A-4417-8CDD-0C6D58E070F8Q37713190-F62A4B23-66C8-454B-9902-6AEF5AF7BF2EQ37945235-CA49A7E0-9868-4F0C-9244-FF3E9AB7F4ACQ38310223-853AA8E1-72F0-48DD-A5F3-E5E5E5562B03Q38361837-D9504FA2-2FD7-4080-8795-5055A638EF0DQ38413897-188984AC-B301-4B06-832E-498375DD764F
P2860
Hantavirus infection induces the expression of RANTES and IP-10 without causing increased permeability in human lung microvascular endothelial cells
description
2001 nî lūn-bûn
@nan
2001 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Hantavirus Infection Induces t ...... icrovascular Endothelial Cells
@nl
Hantavirus infection induces t ...... icrovascular endothelial cells
@ast
Hantavirus infection induces t ...... icrovascular endothelial cells
@en
type
label
Hantavirus Infection Induces t ...... icrovascular Endothelial Cells
@nl
Hantavirus infection induces t ...... icrovascular endothelial cells
@ast
Hantavirus infection induces t ...... icrovascular endothelial cells
@en
prefLabel
Hantavirus Infection Induces t ...... icrovascular Endothelial Cells
@nl
Hantavirus infection induces t ...... icrovascular endothelial cells
@ast
Hantavirus infection induces t ...... icrovascular endothelial cells
@en
P2093
P2860
P3181
P1433
P1476
Hantavirus infection induces t ...... icrovascular endothelial cells
@en
P2093
A A Ansari
C F Spiropoulou
C J Peters
J B Sundstrom
L K McMullan
P E Rollin
W C Hooper
P2860
P304
P3181
P356
10.1128/JVI.75.13.6070-6085.2001
P577
2001-07-01T00:00:00Z