An Interplay between Hypervariable Region 1 of the Hepatitis C Virus E2 Glycoprotein, the Scavenger Receptor BI, and High-Density Lipoprotein Promotes both Enhancement of Infection and Protection against Neutralizing Antibodies

An Interplay between Hypervariable Region 1 of the Hepatitis C Virus E2 Glycoprotein, the Scavenger Receptor BI, and High-Density Lipoprotein Promotes both Enhancement of Infection and Protection against Neutralizing Antibodies

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http://www.wikidata.org/entity/Q27469837

Q27469837

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Receptor complementation and mutagenesis reveal SR-BI as an essential HCV entry factor and functionally imply its intra- and extra-cellular domains Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus Entry of pseudotyped hepatitis C virus into primary human hepatocytes depends on the scavenger class B type I receptor Scavenger receptor BI and BII expression levels modulate hepatitis C virus infectivity Initiation of hepatitis C virus infection is dependent on cholesterol and cooperativity between CD81 and scavenger receptor B type I Claudin-6 and claudin-9 function as additional coreceptors for hepatitis C virus Hepatitis C virus relies on lipoproteins for its life cycle Entry inhibitors: New advances in HCV treatment Genetic Diversity Underlying the Envelope Glycoproteins of Hepatitis C Virus: Structural and Functional Consequences and the Implications for Vaccine Design Impact of lipids and lipoproteins on hepatitis C virus infection and virus neutralization Emerging concepts in immunity to hepatitis C virus infection.Viral quasispecies evolution The hepatitis C virus glycan shield and evasion of the humoral immune response Basic residues in hypervariable region 1 of hepatitis C virus envelope glycoprotein e2 contribute to virus entry.Molecular virology of hepatitis C virus (HCV): 2006 update A milestone for hepatitis C virus research: A virus generated in cell culture is fully viable in vivo Cell culture-grown hepatitis C virus is infectious in vivo and can be recultured in vitro Rapid induction of virus-neutralizing antibodies and viral clearance in a single-source outbreak of hepatitis C A focus reduction neutralization assay for hepatitis C virus neutralizing antibodies Sulfated Homologues of Heparin Inhibit Hepatitis C Virus Entry into Mammalian Cells CD81 Expression Is Important for the Permissiveness of Huh7 Cell Clones for Heterogeneous Hepatitis C Virus Infection The Neutralizing Activity of Anti-Hepatitis C Virus Antibodies Is Modulated by Specific Glycans on the E2 Envelope Protein High-Avidity Monoclonal Antibodies against the Human Scavenger Class B Type I Receptor Efficiently Block Hepatitis C Virus Infection in the Presence of High-Density Lipoprotein Characterization of Fusion Determinants Points to the Involvement of Three Discrete Regions of Both E1 and E2 Glycoproteins in the Membrane Fusion Process of Hepatitis C Virus Expression and Characterization of a Minimal Hepatitis C Virus Glycoprotein E2 Core Domain That Retains CD81 Binding Studying Hepatitis C Virus: Making the Best of a Bad Virus Human combinatorial libraries yield rare antibodies that broadly neutralize hepatitis C virus Scavenger Receptor Class B Is Required for Hepatitis C Virus Uptake and Cross-Presentation by Human Dendritic Cells Acute hepatitis C virus infection: A chronic problem Apolipoprotein C1 Association with Hepatitis C Virus Separation of Hepatitis C genotype 4a into IgG-depleted and IgG-enriched fractions reveals a unique quasispecies profile Evidence for Protection against Chronic Hepatitis C Virus Infection in Chimpanzees by Immunization with Replicating Recombinant Vaccinia Virus Identification of a Residue in Hepatitis C Virus E2 Glycoprotein That Determines Scavenger Receptor BI and CD81 Receptor Dependency and Sensitivity to Neutralizing Antibodies Combined antiviral activity of interferon-α and RNA interference directed against hepatitis C without affecting vector delivery and gene silencing Targeting host factors: A novel rationale for the management of hepatitis C virus Low pH-dependent Hepatitis C Virus Membrane Fusion Depends on E2 Integrity, Target Lipid Composition, and Density of Virus Particles Hepatitis C Virus entry: the early steps in the viral replication cycle Mutagenesis of the fusion peptide-like domain of hepatitis C virus E1 glycoprotein: involvement in cell fusion and virus entry Hepatoma Cell Density Promotes Claudin-1 and Scavenger Receptor BI Expression and Hepatitis C Virus Internalization Role of Scavenger Receptor Class B Type I in Hepatitis C Virus Entry: Kinetics and Molecular Determinants

P2860

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P2860

An Interplay between Hypervariable Region 1 of the Hepatitis C Virus E2 Glycoprotein, the Scavenger Receptor BI, and High-Density Lipoprotein Promotes both Enhancement of Infection and Protection against Neutralizing Antibodies

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http://www.wikidata.org/entity/Q27469837

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2005 nî lūn-bûn

@nan

2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2005年の論文

@ja

2005年論文

@yue

2005年論文

@zh-hant

2005年論文

@zh-hk

2005年論文

@zh-mo

2005年論文

@zh-tw

2005年论文

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