Molecular mechanisms of calcium-dependent excitotoxicity
about
Anthranilate fluorescence marks a calcium-propagated necrotic wave that promotes organismal death in C. elegansHibernating without oxygen: physiological adaptations of the painted turtleProtection of cortical cells by equine estrogens against glutamate-induced excitotoxicity is mediated through a calcium independent mechanismCytokine and Growth Factor Activation In Vivo and In Vitro after Spinal Cord InjuryGlucocorticoid and polyamine interactions in the plasticity of glutamatergic synapses that contribute to ethanol-associated dependence and neuronal injuryER Stress-Mediated Signaling: Action Potential and Ca(2+) as Key PlayersPartitioning of the plasma membrane Ca2+-ATPase into lipid rafts in primary neurons: effects of cholesterol depletionChanges in the expression of plasma membrane calcium extrusion systems during the maturation of hippocampal neuronsRapid downregulation of NCX and PMCA in hippocampal neurons following H2O2 oxidative stressMolecular aspects of glutamate dysregulation: implications for schizophrenia and its treatment.Stem cell derived basal forebrain cholinergic neurons from Alzheimer's disease patients are more susceptible to cell death.Long-term dantrolene treatment reduced intraneuronal amyloid in aged Alzheimer triple transgenic miceTranscriptomic responses in mouse brain exposed to chronic excess of the neurotransmitter glutamate.Effects of Apigenin on Glutamate-induced [Ca](i) Increases in Cultured Rat Hippocampal Neurons.The CACNA1C risk allele rs1006737 is associated with age-related prefrontal cortical thinning in bipolar I disorder.Cellular mechanisms underlying acquired epilepsy: the calcium hypothesis of the induction and maintainance of epilepsy.Cell surface translocation of annexin A2 facilitates glutamate-induced extracellular proteolysisThe dual role of calcium as messenger and stressor in cell damage, death, and survival.Neuroprotection by glutamate receptor antagonists against seizure-induced excitotoxic cell death in the aging brain.Intersection between metabolic dysfunction, high fat diet consumption, and brain aging.Prolonged Abeta treatment leads to impairment in the ability of primary cortical neurons to maintain K+ and Ca2+ homeostasis.Death by necrosis. Uncontrollable catastrophe, or is there order behind the chaos?Inhibition of NR2B-Containing N-methyl-D-Aspartate Receptors (NMDARs) in Experimental Autoimmune Encephalomyelitis, a Model of Multiple SclerosisSpecific aspartyl and calpain proteases are required for neurodegeneration in C. elegans.Corticosteroid actions in the hippocampus.Genetic models of mechanotransduction: the nematode Caenorhabditis elegans.Lysosomal biogenesis and function is critical for necrotic cell death in Caenorhabditis elegans.Intermittent hypoxia is associated with oxidative stress and spatial learning deficits in the rat.Emerging roles of canonical TRP channels in neuronal function.Intravenous administration of achyranthes bidentata polypeptides supports recovery from experimental ischemic stroke in vivo.TDP-43 toxicity proceeds via calcium dysregulation and necrosis in aging Caenorhabditis elegans motor neurons.Syntheses and pharmacological evaluations of novel N-substituted bicyclo-heptane-2-amines at N-methyl-D-aspartate receptorsEndogenous GABA(A) and GABA(B) receptor-mediated electrical suppression is critical to neuronal anoxia toleranceCalcium-permeable AMPA/kainate receptors mediate toxicity and preconditioning by oxygen-glucose deprivation in oligodendrocyte precursors.Calcium signals and calpain-dependent necrosis are essential for release of coxsackievirus B from polarized intestinal epithelial cellsGlutamate excitotoxicity is involved in the induction of paralysis in mice after infection by a human coronavirus with a single point mutation in its spike protein.Causes and consequences of methamphetamine and MDMA toxicity.Potential interactions of calcium-sensitive reagents with zinc ion in different cultured cells.Altered calcium signaling following traumatic brain injuryRaised Intracellular Calcium Contributes to Ischemia-Induced Depression of Evoked Synaptic Transmission.
P2860
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P2860
Molecular mechanisms of calcium-dependent excitotoxicity
description
2000 nî lūn-bûn
@nan
2000 թուականին հրատարակուած գիտական յօդուած
@hyw
2000 թվականին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Molecular mechanisms of calcium-dependent excitotoxicity
@ast
Molecular mechanisms of calcium-dependent excitotoxicity
@en
Molecular mechanisms of calcium-dependent excitotoxicity
@nl
type
label
Molecular mechanisms of calcium-dependent excitotoxicity
@ast
Molecular mechanisms of calcium-dependent excitotoxicity
@en
Molecular mechanisms of calcium-dependent excitotoxicity
@nl
prefLabel
Molecular mechanisms of calcium-dependent excitotoxicity
@ast
Molecular mechanisms of calcium-dependent excitotoxicity
@en
Molecular mechanisms of calcium-dependent excitotoxicity
@nl
P3181
P356
P1476
Molecular mechanisms of calcium-dependent excitotoxicity
@en
P2093
M Tymianski
P2888
P3181
P356
10.1007/S001090000077
P407
P577
2000-01-01T00:00:00Z
P6179
1045132832