Glutamate transporter EAAT2 splice variants occur not only in ALS, but also in AD and controls
about
A comprehensive review of amyotrophic lateral sclerosisMechanisms of glutamate transportMitochondrial proteomic analysis of a cell line model of familial amyotrophic lateral sclerosis.Glutamate transporters and the excitotoxic path to motor neuron degeneration in amyotrophic lateral sclerosis.Exon-skipping splice variants of excitatory amino acid transporter-2 (EAAT2) form heteromeric complexes with full-length EAAT2.The splice of life: alternative splicing and neurological disease.Familial amyotrophic lateral sclerosis.Impairment of neuronal glutamate uptake and modulation of the glutamate transporter GLT-1 induced by retinal ischemia.Therapeutic developments in the treatment of amyotrophic lateral sclerosis.Association of rare variation in the glutamate receptor gene SLC1A2 with susceptibility to bipolar disorder and schizophrenia.Glutamate transporter splice variant expression in an enriched pyramidal cell population in schizophreniaReversal or reduction of glutamate and GABA transport in CNS pathology and therapy.Differential expression of the glutamate transporter GLT-1 in pancreasTransporters for L-glutamate: an update on their molecular pharmacology and pathological involvement.Astrocyte dysfunction in neurological disorders: a molecular perspective.EAAT2 regulation and splicing: relevance to psychiatric and neurological disorders.Role of thiamine in Alzheimer's disease.Genetic dys-regulation of astrocytic glutamate transporter EAAT2 and its implications in neurological disorders and manganese toxicityA novel alternative splicing form of excitatory amino acid transporter 1 is a negative regulator of glutamate uptake.The role of glutamate transporters in the pathophysiology of neuropsychiatric disorders.Caffeine consumption attenuates neurochemical modifications in the hippocampus of streptozotocin-induced diabetic rats.Increases in cortical glutamate concentrations in transgenic amyotrophic lateral sclerosis mice are attenuated by creatine supplementation.Alternative splicing of the 5'-sequences of the mouse EAAT2 glutamate transporter and expression in a transgenic model for amyotrophic lateral sclerosis.Neuronal expression of splice variants of "glial" glutamate transporters in brains afflicted by Alzheimer's disease: unmasking an intrinsic neuronal property.Chemical hypoxia facilitates alternative splicing of EAAT2 in presymptomatic APP23 transgenic mice.EAAT2 and the Molecular Signature of Amyotrophic Lateral Sclerosis.
P2860
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P2860
Glutamate transporter EAAT2 splice variants occur not only in ALS, but also in AD and controls
description
2000 nî lūn-bûn
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2000 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
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2000 թվականի հոտեմբերին հրատարակված գիտական հոդված
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2000年の論文
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name
Glutamate transporter EAAT2 sp ...... S, but also in AD and controls
@ast
Glutamate transporter EAAT2 sp ...... S, but also in AD and controls
@en
Glutamate transporter EAAT2 sp ...... S, but also in AD and controls
@nl
type
label
Glutamate transporter EAAT2 sp ...... S, but also in AD and controls
@ast
Glutamate transporter EAAT2 sp ...... S, but also in AD and controls
@en
Glutamate transporter EAAT2 sp ...... S, but also in AD and controls
@nl
prefLabel
Glutamate transporter EAAT2 sp ...... S, but also in AD and controls
@ast
Glutamate transporter EAAT2 sp ...... S, but also in AD and controls
@en
Glutamate transporter EAAT2 sp ...... S, but also in AD and controls
@nl
P2093
P356
P1433
P1476
Glutamate transporter EAAT2 sp ...... S, but also in AD and controls
@en
P2093
D D Chambliss
J L Elliott
S L Carroll
P304
P356
10.1212/WNL.55.8.1082
P407
P577
2000-10-24T00:00:00Z