Intracellular fibril formation, calcification, and enrichment of chaperones, cytoskeletal, and intermediate filament proteins in the adult hippocampus CA1 following neonatal exposure to the nonprotein amino acid BMAA

Intracellular fibril formation, calcification, and enrichment of chaperones, cytoskeletal, and intermediate filament proteins in the adult hippocampus CA1 following neonatal exposure to the nonprotein amino acid BMAA

Item
http://www.wikidata.org/entity/Q28239546
Dietary exposure to an environmental toxin triggers neurofibrillary tangles and amyloid deposits in the brainHigh resolution metabolite imaging in the hippocampus following neonatal exposure to the environmental toxin BMAA using ToF-SIMSMALDI imaging delineates hippocampal glycosphingolipid changes associated with neurotoxin induced proteopathy following neonatal BMAA exposure.The John Walton Muscular Dystrophy Research Centre in the University of Newcastle and the BMAA Theory of Motor Neuron DiseaseA critical review of the postulated role of the non-essential amino acid, β-N-methylamino-L-alanine, in neurodegenerative disease in humans.Melanin and neuromelanin binding of drugs and chemicals: toxicological implications.Imaging mass spectrometry in drug development and toxicologyEnvironmental neurotoxin interaction with proteins: Dose-dependent increase of free and protein-associated BMAA (β-N-methylamino-L-alanine) in neonatal rat brain.β-N-Methylamino-L-alanine (BMAA) perturbs alanine, aspartate and glutamate metabolism pathways in human neuroblastoma cells as determined by metabolic profiling.Metabolic profiling of zebrafish (Danio rerio) embryos by NMR spectroscopy reveals multifaceted toxicity of β-methylamino-L-alanine (BMAA).Cellular and Molecular Aspects of the β-N-Methylamino-l-alanine (BMAA) Mode of Action within the Neurodegenerative Pathway: Facts and Controversy.Perinatal Exposure to the Cyanotoxin β-N-Méthylamino-L-Alanine (BMAA) Results in Long-Lasting Behavioral Changes in Offspring-Potential Involvement of DNA Damage and Oxidative Stress.Analysis of BMAA enantiomers in cycads, cyanobacteria, and mammals: in vivo formation and toxicity of D-BMAA.Neurotoxicity of the Cyanotoxin BMAA Through Axonal Degeneration and Intercellular Spreading.
P2860

Intracellular fibril formation, calcification, and enrichment of chaperones, cytoskeletal, and intermediate filament proteins in the adult hippocampus CA1 following neonatal exposure to the nonprotein amino acid BMAA

Item
http://www.wikidata.org/entity/Q28239546
description
2015 nî lūn-bûn
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2015 թուականի Մարտին հրատարակուած գիտական յօդուած
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2015 թվականի մարտին հրատարակված գիտական հոդված
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2015年の論文
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2015年論文
@yue
2015年論文
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2015年論文
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2015年論文
@zh-mo
2015年論文
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2015年论文
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name
Intracellular fibril formation ...... the nonprotein amino acid BMAA
@ast
Intracellular fibril formation ...... the nonprotein amino acid BMAA
@en
Intracellular fibril formation ...... the nonprotein amino acid BMAA
@nl
type
label
Intracellular fibril formation ...... the nonprotein amino acid BMAA
@ast
Intracellular fibril formation ...... the nonprotein amino acid BMAA
@en
Intracellular fibril formation ...... the nonprotein amino acid BMAA
@nl
prefLabel
Intracellular fibril formation ...... the nonprotein amino acid BMAA
@ast
Intracellular fibril formation ...... the nonprotein amino acid BMAA
@en
Intracellular fibril formation ...... the nonprotein amino acid BMAA
@nl
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P407
P433
P478
P50
P577
P5875
P6179
P698
P921
P932
P356
P1433
P1476
Intracellular fibril formation ...... the nonprotein amino acid BMAA
@en
P2093
P2860
P2888
P304
P31
P356
P407
P433
P478
P50
P577
P5875
P6179
P698
P921
P932