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A lymphatic defect causes ocular hypertension and glaucoma in mice

A lymphatic defect causes ocular hypertension and glaucoma in mice

http://www.wikidata.org/entity/Q28247543

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Heterogeneity in the lymphatic vascular system and its origin Angiopoietin-Tie signalling in the cardiovascular and lymphatic systems Aqueous outflow - A continuum from trabecular meshwork to episcleral veins Crosstalk in glomerular injury and repair Tie1 controls angiopoietin function in vascular remodeling and inflammation.Hematopoietic stem and progenitor cells regulate the regeneration of their niche by secreting Angiopoietin-1 Subtype-dependent Morphological and Functional Degeneration of Retinal Ganglion Cells in Mouse Models of Experimental Glaucoma Aqueous outflow regulation: Optical coherence tomography implicates pressure-dependent tissue motion.Using genetic mouse models to gain insight into glaucoma: Past results and future possibilities.Tuning the specificity of a Two-in-One Fab against three angiogenic antigens by fully utilizing the information of deep mutational scanning.Scholars and scientists in the history of the lymphatic system.Pathological lymphangiogenesis is modulated by galectin-8-dependent crosstalk between podoplanin and integrin-associated VEGFR-3.Angiopoietin receptor TEK mutations underlie primary congenital glaucoma with variable expressivity.Endothelial exocytosis of angiopoietin-2 resulting from CCM3 deficiency contributes to cerebral cavernous malformation.Angiopoietin receptor Tie2 is required for vein specification and maintenance via regulating COUP-TFII Intracellular uptake of macromolecules by brain lymphatic endothelial cells during zebrafish embryonic development.Angiopoietin receptor TEK interacts with CYP1B1 in primary congenital glaucoma.Lymphangiogenesis: fuel, smoke, or extinguisher of inflammation's fire?Toward in vivo two-photon analysis of mouse aqueous outflow structure and function.Therapeutic targeting of the angiopoietin-TIE pathway.Primary congenital and developmental glaucomas.Angiopoietins and Tie2 in vascular inflammation.Angiopoietin-1 deficiency increases tumor metastasis in mice.Cardiac Lymphatic Vessels, Transport, and Healing of the Infarcted Heart.Impaired angiopoietin/Tie2 signaling compromises Schlemm's canal integrity and induces glaucoma.Deep tissue analysis of distal aqueous drainage structures and contractile features.Organ-specific lymphatic vasculature: From development to pathophysiology.Ascending Vasa Recta Are Angiopoietin/Tie2-Dependent Lymphatic-Like Vessels.Lymphatic Endothelial Cell Plasticity in Development and Disease.Organotypic vasculature: From descriptive heterogeneity to functional pathophysiology.Context-dependent functions of angiopoietin 2 are determined by the endothelial phosphatase VEPTP.Angiopoietin-1 deficiency increases renal capillary rarefaction and tubulointerstitial fibrosis in mice.Angiopoietin-1 is required for Schlemm's canal development in mice and humans.All TIEd up: mechanisms of Schlemm's canal maintenance.A novel podoplanin-GFPCre mouse strain for gene deletion in lymphatic endothelial cells.Genome-wide analyses identify 68 new loci associated with intraocular pressure and improve risk prediction for primary open-angle glaucoma.Near-Infrared Fluorescence Lymphatic Imaging of a Toddler With Congenital Lymphedema.Physiological Perspective on Therapies of Lymphatic Vessels.

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P2860

A lymphatic defect causes ocular hypertension and glaucoma in mice

http://www.wikidata.org/entity/Q28247543

description

2014 nî lūn-bûn

@nan

2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

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2014年論文

@zh-tw

2014年论文

@wuu

name

A lymphatic defect causes ocular hypertension and glaucoma in mice

@ast

A lymphatic defect causes ocular hypertension and glaucoma in mice

@en

A lymphatic defect causes ocular hypertension and glaucoma in mice

@nl

type

label

A lymphatic defect causes ocular hypertension and glaucoma in mice

@ast

A lymphatic defect causes ocular hypertension and glaucoma in mice

@en

A lymphatic defect causes ocular hypertension and glaucoma in mice

@nl

prefLabel

A lymphatic defect causes ocular hypertension and glaucoma in mice

@ast

A lymphatic defect causes ocular hypertension and glaucoma in mice

@en

A lymphatic defect causes ocular hypertension and glaucoma in mice

@nl

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Journal of Clinical Investigation

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A lymphatic defect causes ocular hypertension and glaucoma in mice

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Ann Flenniken

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Asish K Ghosh

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Hoon-Ki Sung

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Nicholas W Gale

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Shinji Yamaguchi

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Susan E Quaggin

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Tsutomu Kume

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Xiaorong Liu

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Young-Kwon Hong

http://www.w3.org/2001/XMLSchema#string

P2860

Vascular dysmorphogenesis caused by an activating mutation in the receptor tyrosine kinase TIE2

Angiopoietin-1 is essential in mouse vasculature during development and in response to injury.

FOXC2 controls formation and maturation of lymphatic collecting vessels through cooperation with NFATc1

Schlemm's canal is a unique vessel with a combination of blood vascular and lymphatic phenotypes that forms by a novel developmental process

Dominant-negative and targeted null mutations in the endothelial receptor tyrosine kinase, tek, reveal a critical role in vasculogenesis of the embryo

Haploinsufficiency of the transcription factors FOXC1 and FOXC2 results in aberrant ocular development

Conditionals by inversion provide a universal method for the generation of conditional alleles

Changes in the inner and outer retinal layers after acute increase of the intraocular pressure in adult albino Swiss mice.

Murine Notch1 is required for lymphatic vascular morphogenesis during development

Lineage tracing demonstrates the venous origin of the mammalian lymphatic vasculature

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4320-4324

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scholarly article

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2372641

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10.1172/JCI77162

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10

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124

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Aris Economides

Benjamin R Thomson

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2014-09-09T00:00:00Z

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P698

25202984

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P921

arterial hypertension

P932

4191022

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