Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
about
Comprehensive analysis of interactions between the Src-associated protein in mitosis of 68 kDa and the human Src-homology 3 proteomeThe STAR RNA binding proteins GLD-1, QKI, SAM68 and SLM-2 bind bipartite RNA motifsThe splicing regulator Sam68 binds to a novel exonic splicing silencer and functions in SMN2 alternative splicing in spinal muscular atrophyComparison of an expanded ataxia interactome with patient medical records reveals a relationship between macular degeneration and ataxiaSAM68 regulates neuronal activity-dependent alternative splicing of neurexin-1Sam68 regulates a set of alternatively spliced exons during neurogenesisProteostasis and RNA Binding Proteins in Synaptic Plasticity and in the Pathogenesis of Neuropsychiatric DisordersThe neurogenetics of alternative splicingThe splicing regulator Rbfox2 is required for both cerebellar development and mature motor functionProteomic identification of heterogeneous nuclear ribonucleoprotein L as a novel component of SLM/Sam68 Nuclear Bodies.Transcription and post-transcriptional regulation of spermatogenesis.Histopathological and behavioral assessment of toxin-produced cerebellar lesion: a potent model for cell transplantation studies in the cerebellum.The tissue-specific RNA binding protein T-STAR controls regional splicing patterns of neurexin pre-mRNAs in the brainNetworking in a global world: establishing functional connections between neural splicing regulators and their target transcriptsSam68 relocalization into stress granules in response to oxidative stress through complexing with TIA-1Sam68 marks the transcriptionally active stages of spermatogenesis and modulates alternative splicing in male germ cells.RNA-binding protein misregulation in microsatellite expansion disordersEmerging roles for Sam68 in adipogenesis and neuronal development.RNA-binding protein Sam68 controls synapse number and local β-actin mRNA metabolism in dendrites.Role of Sam68 in post-transcriptional gene regulation.Sam68 promotes self-renewal and glycolytic metabolism in mouse neural progenitor cells by modulating Aldh1a3 pre-mRNA 3'-end processing.Developments in RNA splicing and disease.RNA-binding proteins in microsatellite expansion disorders: mediators of RNA toxicity.Sam68 sequestration and partial loss of function are associated with splicing alterations in FXTAS patients.The p53 status can influence the role of Sam68 in tumorigenesis.Coordination between Translation and Degradation Regulates Inducibility of mGluR-LTD.
P2860
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P2860
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
description
2008 nî lūn-bûn
@nan
2008 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
@ast
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
@en
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
@nl
type
label
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
@ast
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
@en
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
@nl
prefLabel
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
@ast
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
@en
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
@nl
P1476
Motor coordination defects in mice deficient for the Sam68 RNA-binding protein
@en
P2093
Kiven E Lukong
Stéphane Richard
P304
P356
10.1016/J.BBR.2008.01.010
P407
P577
2008-06-03T00:00:00Z