Deletion of either CD55 or CD97 ameliorates arthritis in mouse models - sprookjes - yvandenbroek - TriplyDB

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

http://www.wikidata.org/entity/Q28272391

about

Immunohistochemical expression and prognostic value of CD97 and its ligand CD55 in primary gallbladder carcinoma A novel role for CD55 in granulocyte homeostasis and anti-bacterial host defense Activation of myeloid cell-specific adhesion class G protein-coupled receptor EMR2 via ligation-induced translocation and interaction of receptor subunits in lipid raft microdomains Sticky signaling--adhesion class G protein-coupled receptors take the stage Gpr97 is essential for the follicular versus marginal zone B-lymphocyte fate decision Saposin C coupled lipid nanovesicles specifically target arthritic mouse joints for optical imaging of disease severity Adhesion GPCRs as Modulators of Immune Cell Function.Runx1 is a tumor suppressor gene in the mouse gastrointestinal tract.Contradictory Role of CD97 in Basal and Tumor Necrosis Factor-Induced Osteoclastogenesis In Vivo.Triggering of the dsRNA sensors TLR3, MDA5, and RIG-I induces CD55 expression in synovial fibroblasts Adhesion GPCRs are widely expressed throughout the subsections of the gastrointestinal tract.Mice overexpressing CD97 in intestinal epithelial cells provide a unique model for mammalian postnatal intestinal cylindrical growth.The presence of CD55- and/or CD59-deficient erythrocytic populations in patients with rheumatic diseases reflects an immune-mediated bone-marrow derived phenomenon.Adhesion G-protein-coupled receptors: elusive hybrids come of age.CD55 deposited on synovial collagen fibers protects from immune complex-mediated arthritis.Dissecting signaling and functions of adhesion G protein-coupled receptors.Beyond the Role of CD55 as a Complement Component.Adipose tissue transcriptome changes during obesity development in female dogs.Modulation of PBMC-decay accelerating factor (PBMC-DAF) and cytokines in rheumatoid arthritis.

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Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

http://www.wikidata.org/entity/Q28272391

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

@ast

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

@en

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

@nl

type

label

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

@ast

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

@en

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

@nl

prefLabel

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

@ast

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

@en

Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

@nl

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P1433

Arthritis & Rheumatology

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Deletion of either CD55 or CD97 ameliorates arthritis in mouse models

@en

P2093

A Seda Yilmaz-Elis

http://www.w3.org/2001/XMLSchema#string

Daphne de Launay

http://www.w3.org/2001/XMLSchema#string

Else N Kop

http://www.w3.org/2001/XMLSchema#string

Feng Lin

http://www.w3.org/2001/XMLSchema#string

J Sjef Verbeek

http://www.w3.org/2001/XMLSchema#string

Jörg Hamann

http://www.w3.org/2001/XMLSchema#string

Kris A Reedquist

http://www.w3.org/2001/XMLSchema#string

M Edward Medof

http://www.w3.org/2001/XMLSchema#string

Paul P Tak

http://www.w3.org/2001/XMLSchema#string

Robert M Hoek

http://www.w3.org/2001/XMLSchema#string

P2860

The epidermal growth factor-seven transmembrane (EGF-TM7) receptor CD97 is required for neutrophil migration and host defense

Analysis of CD97 expression and manipulation: antibody treatment but not gene targeting curtails granulocyte migration

Deletion of decay-accelerating factor (CD55) exacerbates autoimmune disease development in MRL/lpr mice

Markedly enhanced susceptibility to experimental autoimmune myasthenia gravis in the absence of decay-accelerating factor protection.

Increased susceptibility of decay-accelerating factor deficient mice to anti-glomerular basement membrane glomerulonephritis.

Critical protection from renal ischemia reperfusion injury by CD55 and CD59.

Decay-accelerating factor deficiency increases susceptibility to dextran sulfate sodium-induced colitis: role for complement in inflammatory bowel disease.

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1036-42

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scholarly article

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10.1002/ART.27347

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4

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62

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2010-04-01T00:00:00Z

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P5875

41406153

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P698

20131275

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