CSF-1 signaling mediates recovery from acute kidney injury. - sprookjes - yvandenbroek - TriplyDB

CSF-1 signaling mediates recovery from acute kidney injury.

CSF-1 signaling mediates recovery from acute kidney injury.

http://www.wikidata.org/entity/Q28279068

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Macrophage Phenotype in Kidney Injury and Repair Macrophage heterogeneity, phenotypes, and roles in renal fibrosis Mechanisms driving macrophage diversity and specialization in distinct tumor microenvironments and parallelisms with other tissues Interferon-regulatory factors determine macrophage phenotype polarization Dendritic cells and macrophages in the kidney: a spectrum of good and evil Systemic and Cardiac Depletion of M2 Macrophage through CSF-1R Signaling Inhibition Alters Cardiac Function Post Myocardial Infarction Proximal tubule-derived colony stimulating factor-1 mediates polarization of renal macrophages and dendritic cells, and recovery in acute kidney injury Inhibition of cyclooxygenase-2 in hematopoietic cells results in salt-sensitive hypertension Prostaglandin E receptor 4 (EP4) promotes colonic tumorigenesis.Pathogenic and protective role of macrophages in kidney disease Macrophage in chronic kidney disease Driving change: kidney proximal tubule CSF-1 polarizes macrophages Targeting Endogenous Repair Pathways after AKI.Inflammation in AKI: Current Understanding, Key Questions, and Knowledge Gaps.Sisters in arms: myeloid and tubular epithelial cells shape renal innate immunity.Therapeutic translation in acute kidney injury: the epithelial/endothelial axis Retinoic Acid Signaling Coordinates Macrophage-Dependent Injury and Repair after AKI.Myeloid PTEN deficiency protects livers from ischemia reperfusion injury by facilitating M2 macrophage differentiation.Erythropoietin protects against rhabdomyolysis-induced acute kidney injury by modulating macrophage polarization.Using 2-photon microscopy to understand albuminuria.Colony-stimulating factor-1 signaling suppresses renal crystal formation.Macrophages in atherosclerosis: a dynamic balance.Macrophages in Tissue Repair, Regeneration, and Fibrosis.Renal F4/80+ CD11c+ mononuclear phagocytes display phenotypic and functional characteristics of macrophages in health and in adriamycin nephropathy Macrophage/epithelium cross-talk regulates cell cycle progression and migration in pancreatic progenitors.Tissue-type plasminogen activator (tPA) promotes M1 macrophage survival through p90 ribosomal S6 kinase (RSK) and p38 mitogen-activated protein kinase (MAPK) pathway Vascular-resident CD169-positive monocytes and macrophages control neutrophil accumulation in the kidney with ischemia-reperfusion injury KIM-1-mediated phagocytosis reduces acute injury to the kidney.Tamm-Horsfall Protein Regulates Granulopoiesis and Systemic Neutrophil Homeostasis.Specific macrophage subtypes influence the progression of rhabdomyolysis-induced kidney injury GM-CSF Promotes Macrophage Alternative Activation after Renal Ischemia/Reperfusion Injury.Macrophage polarization in kidney diseases.Tubular cross talk in acute kidney injury: a story of sense and sensibility Kidney Regeneration: Lessons from Development.The Role of M2 Macrophages in the Progression of Chronic Kidney Disease following Acute Kidney Injury.MG53-mediated cell membrane repair protects against acute kidney injury Heme Oxygenase-1 Regulates Myeloid Cell Trafficking in AKI IL-34 mediates acute kidney injury and worsens subsequent chronic kidney disease.M2 macrophage polarisation is associated with alveolar formation during postnatal lung development.Epithelial cell TGFβ signaling induces acute tubular injury and interstitial inflammation.

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P2860

CSF-1 signaling mediates recovery from acute kidney injury.

http://www.wikidata.org/entity/Q28279068

description

2012 nî lūn-bûn

@nan

2012 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

CSF-1 signaling mediates recovery from acute kidney injury

@nl

CSF-1 signaling mediates recovery from acute kidney injury.

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CSF-1 signaling mediates recovery from acute kidney injury.

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type

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CSF-1 signaling mediates recovery from acute kidney injury

@nl

CSF-1 signaling mediates recovery from acute kidney injury.

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CSF-1 signaling mediates recovery from acute kidney injury.

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prefLabel

CSF-1 signaling mediates recovery from acute kidney injury

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CSF-1 signaling mediates recovery from acute kidney injury.

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CSF-1 signaling mediates recovery from acute kidney injury.

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CSF-1 signaling mediates recovery from acute kidney injury.

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Amar Singh

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Bing Yao

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Huiyong Yin

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Ingrid Chang

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Jianchun Chen

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Karlton Wong

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Li Jiang

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Ming-Zhi Zhang

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Raymond C Harris

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Shilin Yang

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P2860

Exploring the full spectrum of macrophage activation

The renal mononuclear phagocytic system

Macrophage Wnt7b is critical for kidney repair and regeneration

Alternative activation of macrophages: mechanism and functions

Regulation of wound healing by growth factors and cytokines

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

Evidence that fibroblasts derive from epithelium during tissue fibrosis

Inflammatory monocytes recruited after skeletal muscle injury switch into antiinflammatory macrophages to support myogenesis

Immune mechanisms and novel pharmacological therapies of acute kidney injury.

The chemokine receptors CCR2 and CX3CR1 mediate monocyte/macrophage trafficking in kidney ischemia-reperfusion injury

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4519-4532

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10.1172/JCI60363

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12

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122

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