Cortactin is an essential regulator of matrix metalloproteinase secretion and extracellular matrix degradation in invadopodia - sprookjes - yvandenbroek - TriplyDB

Cortactin is an essential regulator of matrix metalloproteinase secretion and extracellular matrix degradation in invadopodia

Cortactin is an essential regulator of matrix metalloproteinase secretion and extracellular matrix degradation in invadopodia

http://www.wikidata.org/entity/Q28300785

about

The regulation of MMP targeting to invadopodia during cancer metastasis Role of H-Ras/ERK signaling in carbon nanotube-induced neoplastic-like transformation of human mesothelial cells Hematopoietic lineage cell-specific protein 1 functions in concert with the Wiskott-Aldrich syndrome protein to promote podosome array organization and chemotaxis in dendritic cells Endosomal WASH and exocyst complexes control exocytosis of MT1-MMP at invadopodia.Pseudopodium-enriched atypical kinase 1 regulates the cytoskeleton and cancer progression [corrected].A role for the podosome/invadopodia scaffold protein Tks5 in tumor growth in vivo The 'ins' and 'outs' of podosomes and invadopodia: characteristics, formation and function The novel adaptor protein Tks4 (SH3PXD2B) is required for functional podosome formation Cortactin branches out: roles in regulating protrusive actin dynamics The involvement of mutant Rac1 in the formation of invadopodia in cultured melanoma cells Regulators of Actin Dynamics in Gastrointestinal Tract Tumors Tumor and stromal-based contributions to head and neck squamous cell carcinoma invasion The interplay between the proteolytic, invasive, and adhesive domains of invadopodia and their roles in cancer invasion Cells activated for wound repair have the potential to direct collective invasion of an epithelium.NEDD9/Arf6-dependent endocytic trafficking of matrix metalloproteinase 14: a novel mechanism for blocking mesenchymal cell invasion and metastasis of breast cancer.Multiparametric classification links tumor microenvironments with tumor cell phenotype Cortactin phosphorylated by ERK1/2 localizes to sites of dynamic actin regulation and is required for carcinoma lamellipodia persistence Regulation of invasive behavior by vascular endothelial growth factor is HEF1-dependent AFAP1L1, a novel associating partner with vinculin, modulates cellular morphology and motility, and promotes the progression of colorectal cancers Spatiotemporal regulation of Src and its substrates at invadosomes Cortactin regulates cofilin and N-WASp activities to control the stages of invadopodium assembly and maturation Laminin-332-beta1 integrin interactions negatively regulate invadopodia Arg interacts with cortactin to promote adhesion-dependent cell edge protrusion FAK competes for Src to promote migration against invasion in melanoma cells.Met receptor tyrosine kinase signals through a cortactin-Gab1 scaffold complex, to mediate invadopodia Nck adaptor proteins link Tks5 to invadopodia actin regulation and ECM degradation.β₁Integrin/FAK/cortactin signaling is essential for human head and neck cancer resistance to radiotherapy SRChing for the substrates of Src.Regulation of invadopodia formation and activity by CD147 Dependence of invadopodia function on collagen fiber spacing and cross-linking: computational modeling and experimental evidence.Supervillin reorganizes the actin cytoskeleton and increases invadopodial efficiency.HER2 carboxyl-terminal fragments regulate cell migration and cortactin phosphorylation The membrane-associated protein, supervillin, accelerates F-actin-dependent rapid integrin recycling and cell motility.N-WASP coordinates the delivery and F-actin-mediated capture of MT1-MMP at invasive pseudopods.Adhesion rings surround invadopodia and promote maturation.Microtubule dynamic instability controls podosome patterning in osteoclasts through EB1, cortactin, and Src.Macrophage contact induces RhoA GTPase signaling to trigger tumor cell intravasation.Control of MT1-MMP transport by atypical PKC during breast-cancer progression.Chemotaxis in cancer The Arp2/3 complex mediates multigeneration dendritic protrusions for efficient 3-dimensional cancer cell migration.

P2860

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P2860

Cortactin is an essential regulator of matrix metalloproteinase secretion and extracellular matrix degradation in invadopodia

http://www.wikidata.org/entity/Q28300785

description

2007 nî lūn-bûn

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2007 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2007 թվականի մայիսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Cortactin is an essential regu ...... rix degradation in invadopodia

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Cortactin is an essential regu ...... rix degradation in invadopodia

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Cortactin is an essential regu ...... rix degradation in invadopodia

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Cortactin is an essential regu ...... rix degradation in invadopodia

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Cortactin is an essential regu ...... rix degradation in invadopodia

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Cortactin is an essential regu ...... rix degradation in invadopodia

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Cortactin is an essential regu ...... rix degradation in invadopodia

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Cortactin is an essential regu ...... rix degradation in invadopodia

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Cortactin is an essential regu ...... rix degradation in invadopodia

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Cancer Research

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Cortactin is an essential regu ...... rix degradation in invadopodia

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Amy S Whigham

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Emily S Clark

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Wendell G Yarbrough

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scholarly article

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10.1158/0008-5472.CAN-06-3928

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9

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67

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Alissa M. Weaver

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2007-05-01T00:00:00Z

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6348733

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17483334

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extracellular matrix