Calcium currents in embryonic and neonatal mammalian skeletal muscle
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Spatially segregated control of Ca2+ release in developing skeletal muscle of mice.Differential effects of RGK proteins on L-type channel function in adult mouse skeletal musclePhysiological and ultrastructural features of human induced pluripotent and embryonic stem cell-derived skeletal myocytes in vitroFunctional expression of transgenic 1sDHPR channels in adult mammalian skeletal muscle fibres.Ontogenesis and localization of Ca2+ channels in mammalian skeletal muscle in culture and role in excitation-contraction coupling.Recovery of Ca2+ current, charge movements, and Ca2+ transients in myotubes deficient in dihydropyridine receptor beta 1 subunit transfected with beta 1 cDNA.The S5-S6 linker of repeat I is a critical determinant of L-type Ca2+ channel conductanceA malignant hyperthermia-inducing mutation in RYR1 (R163C): consequent alterations in the functional properties of DHPR channelsReduced Ca2+ current, charge movement, and absence of Ca2+ transients in skeletal muscle deficient in dihydropyridine receptor beta 1 subunit.Differential regulation of skeletal muscle L-type Ca2+ current and excitation-contraction coupling by the dihydropyridine receptor beta subunit.Patch-clamp recording of charge movement, Ca2+ current, and Ca2+ transients in adult skeletal muscle fibersVoltage clamp methods for the study of membrane currents and SR Ca(2+) release in adult skeletal muscle fibresAgouti regulation of intracellular calcium: role in the insulin resistance of viable yellow mice.The I4895T mutation in the type 1 ryanodine receptor induces fiber-type specific alterations in skeletal muscle that mimic premature aging.Sarcolemmal-restricted localization of functional ClC-1 channels in mouse skeletal muscleMuscle weakness in Ryr1I4895T/WT knock-in mice as a result of reduced ryanodine receptor Ca2+ ion permeation and release from the sarcoplasmic reticulum.Monovalent cationic channel activity in the inner membrane of nuclei from skeletal muscle fibers.Allele-specific gene silencing in two mouse models of autosomal dominant skeletal myopathy.Ca(2+) permeation and/or binding to CaV1.1 fine-tunes skeletal muscle Ca(2+) signaling to sustain muscle function.Apparent lack of physical or functional interaction between CaV1.1 and its distal C terminus.Membrane repolarization stops caffeine-induced Ca2+ release in skeletal muscle cells.Ca2+-sensitive inactivation of L-type Ca2+ channels depends on multiple cytoplasmic amino acid sequences of the alpha1C subunit.Muscle chloride channel dysfunction in two mouse models of myotonic dystrophy.Deficiency of triad junction and contraction in mutant skeletal muscle lacking junctophilin type 1.Contractions of dysgenic skeletal muscle triggered by a potentiated, endogenous calcium currentSingle calcium channel behavior in native skeletal muscle.Potentiated L-type Ca2+ channels rectifyEffect of postnatal development on calcium currents and slow charge movement in mammalian skeletal muscleUnitary behavior of skeletal, cardiac, and chimeric L-type Ca2+ channels expressed in dysgenic myotubes.Structural regions of the cardiac Ca channel alpha subunit involved in Ca-dependent inactivation.Role of calcium permeation in dihydropyridine receptor function. Insights into channel gating and excitation-contraction coupling.Sequence differences in the IQ motifs of CaV1.1 and CaV1.2 strongly impact calmodulin binding and calcium-dependent inactivation.Mitochondrial calcium buffering contributes to the maintenance of Basal calcium levels in mouse taste cells.De novo expression of connexin hemichannels in denervated fast skeletal muscles leads to atrophyCritical roles of the S3 segment and S3-S4 linker of repeat I in activation of L-type calcium channelsConformational activation of Ca2+ entry by depolarization of skeletal myotubes.Charge movement and transcription regulation of L-type calcium channel alpha(1S) in skeletal muscle cells.Insulin-like growth factor-1 increases skeletal muscle dihydropyridine receptor alpha 1S transcriptional activity by acting on the cAMP-response element-binding protein element of the promoter region.Cardiac T-type calcium current: pharmacology and roles in cardiac tissues.The mammalian skeletal muscle DHPR has larger Ca2+ conductance and is phylogenetically ancient to the early ray-finned fish sterlet (Acipenser ruthenus).
P2860
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P2860
Calcium currents in embryonic and neonatal mammalian skeletal muscle
description
1988 nî lūn-bûn
@nan
1988 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1988 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
name
Calcium currents in embryonic and neonatal mammalian skeletal muscle
@ast
Calcium currents in embryonic and neonatal mammalian skeletal muscle
@en
Calcium currents in embryonic and neonatal mammalian skeletal muscle
@nl
type
label
Calcium currents in embryonic and neonatal mammalian skeletal muscle
@ast
Calcium currents in embryonic and neonatal mammalian skeletal muscle
@en
Calcium currents in embryonic and neonatal mammalian skeletal muscle
@nl
prefLabel
Calcium currents in embryonic and neonatal mammalian skeletal muscle
@ast
Calcium currents in embryonic and neonatal mammalian skeletal muscle
@en
Calcium currents in embryonic and neonatal mammalian skeletal muscle
@nl
P2860
P921
P356
P1476
Calcium currents in embryonic and neonatal mammalian skeletal muscle
@en
P2093
P2860
P304
P356
10.1085/JGP.91.6.781
P407
P577
1988-06-01T00:00:00Z