Female sex hormones regulate macrophage function after trauma-hemorrhage and prevent increased death rate from subsequent sepsis - sprookjes - yvandenbroek - TriplyDB

Female sex hormones regulate macrophage function after trauma-hemorrhage and prevent increased death rate from subsequent sepsis

Female sex hormones regulate macrophage function after trauma-hemorrhage and prevent increased death rate from subsequent sepsis

http://www.wikidata.org/entity/Q28344087

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Trauma equals danger--damage control by the immune system Relationship between age/gender-induced survival changes and the magnitude of inflammatory activation and organ dysfunction in post-traumatic sepsis Defining the effects of age and gender on immune response and outcomes to melanoma vaccination: a retrospective analysis of a single-institution clinical trials' experience.Sex- and diagnosis-dependent differences in mortality and admission cytokine levels among patients admitted for intensive care.Gender and acute respiratory distress syndrome in critically injured adults: a prospective study.Trends in estradiol during critical illness are associated with mortality independent of admission estradiol Estradiol is associated with mortality in critically ill trauma and surgical patients.High levels of endogenous estrogens are associated with death in the critically injured adult.Serum estradiol concentration as a predictor of death in critically ill and injured adults.Predictors of sepsis in moderately severely injured patients: an analysis of the National Trauma Data Bank Androgen deprivation modulates the inflammatory response induced by irradiation.Testosterone influences renal electrolyte excretion in SHR/y and WKY males.Sex-based differences in outcomes after severe injury: an analysis of blunt trauma patients in China.Sex-specific social regulation of inflammatory responses and sickness behaviors.Gender differences in abdominal aortic aneurysms Influence of physiological androgen levels on wound healing and immune status in men.Estrogenic hormone modulation abrogates changes in red blood cell deformability and neutrophil activation in trauma hemorrhagic shock.Estrogen modulates cutaneous wound healing by downregulating macrophage migration inhibitory factor Kupffer cells and their mediators: the culprits in producing distant organ damage after trauma-hemorrhage.Effects of trauma-hemorrhage and IL-6 deficiency on splenic immune function in a murine trauma model.What are predictors of mortality in patients with pelvic fractures?Impairment of renal function using hyperoncotic colloids in a two hit model of shock: a prospective randomized study.17β-Estradiol attenuates cytokine-induced nitric oxide production in rat hepatocyte.Characterization of acute coagulopathy and sexual dimorphism after injury: females and coagulopathy just do not mix.A Nationwide Analysis of Pelvic Ring Fractures: Incidence and Trends in Treatment, Length of Stay, and Mortality.Females exhibit relative resistance to depressive effects of tumor necrosis factor-alpha on the myocardium.Trauma and immune response--effect of gender differences.The influence of gender on the epidemiology of and outcome from severe sepsis.Prediction of sepsis in trauma patients Post-operative complications of gastric cancer surgery: female gender at high risk Does testosterone prevent early postoperative complications after gastrointestinal surgery?Sex differences in destructive periodontal disease: exploring the biologic basis.Hormones, heart disease, and health: individualized medicine versus throwing the baby out with the bathwater.Flutamide attenuates pro-inflammatory cytokine production and hepatic injury following trauma-hemorrhage via estrogen receptor-related pathway.Increases in serum estrogen levels during major illness are caused by increased peripheral aromatization.Potential therapeutic targets for sepsis in women.Characterization of Sepsis and Sepsis-Associated Encephalopathy.Murine Models of Sepsis and Trauma: Can We Bridge the Gap?Hormones, heart disease, and health: individualized medicine versus throwing the baby out with the bathwater.Mechanism for normal splenic T lymphocyte functions in proestrus females after trauma: enhanced local synthesis of 17beta-estradiol.

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P2860

Female sex hormones regulate macrophage function after trauma-hemorrhage and prevent increased death rate from subsequent sepsis

http://www.wikidata.org/entity/Q28344087

description

2002 nî lūn-bûn

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2002 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2002 թվականի հունվարին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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name

Female sex hormones regulate m ...... th rate from subsequent sepsis

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Female sex hormones regulate m ...... th rate from subsequent sepsis

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Female sex hormones regulate m ...... th rate from subsequent sepsis

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type

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Female sex hormones regulate m ...... th rate from subsequent sepsis

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Female sex hormones regulate m ...... th rate from subsequent sepsis

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Female sex hormones regulate m ...... th rate from subsequent sepsis

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prefLabel

Female sex hormones regulate m ...... th rate from subsequent sepsis

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Female sex hormones regulate m ...... th rate from subsequent sepsis

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Female sex hormones regulate m ...... th rate from subsequent sepsis

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Annals of Surgery

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Female sex hormones regulate m ...... th rate from subsequent sepsis

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Kirby I Bland

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Markus W Knöferl

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Martin G Schwacha

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Martin K Angele

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Michael D Diodato

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William G Cioffi

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P2860

Hormone replacement therapy and cardiovascular disease.

The effects of hormone replacement therapy on thrombin generation, fibrinolysis inhibition, and resistance to activated protein C: prospective cohort study and review of literature.

Estrogen up-regulates Bcl-2 and blocks tolerance induction of naive B cells.

Toward an epidemiology and natural history of SIRS (systemic inflammatory response syndrome)

Gastrointestinal motility in pregnancy.

Male sex steroids are responsible for depressing macrophage immune function after trauma-hemorrhage.

Differential effects of hemorrhage on Kupffer cells: decreased antigen presentation despite increased inflammatory cytokine (IL-1, IL-6 and TNF) release.

Chloroquine attenuates hemorrhagic shock-induced suppression of Kupffer cell antigen presentation and major histocompatibility complex class II antigen expression through blockade of tumor necrosis factor and prostaglandin release.

Bacteremia at Boston City Hospital: Occurrence and mortality during 12 selected years (1935-1972), with special reference to hospital-acquired cases.

Gender differences in the inflammatory response and survival following haemorrhage and subsequent sepsis.

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105-12

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10.1097/00000658-200201000-00014

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1

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Irshad H. Chaudry

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11604425

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11753049

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1422402

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