The diabetes autoantigen ICA69 and its Caenorhabditis elegans homologue, ric-19, are conserved regulators of neuroendocrine secretion
about
Islet cell autoantigen of 69 kDa is an arfaptin-related protein associated with the Golgi complex of insulinoma INS-1 cellsA gene expression fingerprint of C. elegans embryonic motor neuronsPICK1 deficiency impairs secretory vesicle biogenesis and leads to growth retardation and decreased glucose tolerancePICK1 and ICA69 control insulin granule trafficking and their deficiencies lead to impaired glucose toleranceAlternative core promoters regulate tissue-specific transcription from the autoimmune diabetes-related ICA1 (ICA69) gene locusThe twisted pharynx phenotype in C. elegans.Developments in the prediction of type 1 diabetes mellitus, with special reference to insulin autoantibodies.The function of a spindle checkpoint gene bub-1 in C. elegans development.Human fibrocytes express multiple antigens associated with autoimmune endocrine diseases.C. elegans ten-1 is synthetic lethal with mutations in cytoskeleton regulators, and enhances many axon guidance defective mutants.Genomic convergence and network analysis approach to identify candidate genes in Alzheimer's disease.EAK-7 controls development and life span by regulating nuclear DAF-16/FoxO activityUnveiling clusters of RNA transcript pairs associated with markers of Alzheimer's disease progression.An overview of C. Elegans trafficking mutants.Sequence variation in promoter of Ica1 gene, which encodes protein implicated in type 1 diabetes, causes transcription factor autoimmune regulator (AIRE) to increase its binding and down-regulate expression.Islet autoantigens: structure, function, localization, and regulation.Compromised central tolerance of ICA69 induces multiple organ autoimmunity.Insulin signaling in Caenorhabditis elegans regulates both endocrine-like and cell-autonomous outputs.Fishing lines, time-delayed guideposts, and other tricks used by developing pharyngeal neurons in Caenorhabditis elegans.UNC-108/RAB-2 and its effector RIC-19 are involved in dense core vesicle maturation in Caenorhabditis elegans.A Caenorhabditis elegans model of the myosin heavy chain IIa E706K [corrected] mutation.
P2860
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P2860
The diabetes autoantigen ICA69 and its Caenorhabditis elegans homologue, ric-19, are conserved regulators of neuroendocrine secretion
description
2000 nî lūn-bûn
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2000 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հոտեմբերին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
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2000年论文
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name
The diabetes autoantigen ICA69 ...... rs of neuroendocrine secretion
@ast
The diabetes autoantigen ICA69 ...... rs of neuroendocrine secretion
@en
The diabetes autoantigen ICA69 ...... rs of neuroendocrine secretion
@nl
type
label
The diabetes autoantigen ICA69 ...... rs of neuroendocrine secretion
@ast
The diabetes autoantigen ICA69 ...... rs of neuroendocrine secretion
@en
The diabetes autoantigen ICA69 ...... rs of neuroendocrine secretion
@nl
prefLabel
The diabetes autoantigen ICA69 ...... rs of neuroendocrine secretion
@ast
The diabetes autoantigen ICA69 ...... rs of neuroendocrine secretion
@en
The diabetes autoantigen ICA69 ...... rs of neuroendocrine secretion
@nl
P2093
P2860
P356
P1476
The diabetes autoantigen ICA69 ...... rs of neuroendocrine secretion
@en
P2093
P2860
P304
P356
10.1091/MBC.11.10.3277
P407
P577
2000-10-01T00:00:00Z