Toll-like receptor 2 modulates the proinflammatory milieu in Staphylococcus aureus-induced brain abscess
about
Expression and distribution of Toll-like receptors 11-13 in the brain during murine neurocysticercosisToll-like receptor 2 (TLR2)-TLR9 crosstalk dictates IL-12 family cytokine production in microglia.Neuroinflammation leads to region-dependent alterations in astrocyte gap junction communication and hemichannel activity.Scavenger receptor structure and function in health and disease.Cell-type-specific crosstalk between p38 MAPK and Rho signaling in lung micro- and macrovascular barrier dysfunction induced by Staphylococcus aureus-derived pathogens.Penetration of the blood-brain barrier by Staphylococcus aureus: contribution of membrane-anchored lipoteichoic acid.Lectin-like ox-LDL receptor-1 (LOX-1)-Toll-like receptor 4 (TLR4) interaction and autophagy in CATH.a differentiated cells exposed to angiotensin II.Roles of Toll-like receptor 2 (TLR2) and superantigens on adaptive immune responses during CNS staphylococcal infection.Toll-like receptors in health and disease in the brain: mechanisms and therapeutic potential.Toll-like receptors in brain abscess.Evaluation of capsular and acapsular strains of S. aureus in an experimental brain abscess modelRole of microtubules in attenuation of PepG-induced vascular endothelial dysfunction by atrial natriuretic peptide.MyD88 is pivotal for immune recognition of Citrobacter koseri and astrocyte activation during CNS infectionStaphylococcus aureus sarA regulates inflammation and colonization during central nervous system biofilm formation.Oxidized phospholipids protect against lung injury and endothelial barrier dysfunction caused by heat-inactivated Staphylococcus aureus.Adaptive immune defects against glycoantigens in chronic granulomatous disease via dysregulated nitric oxide productionExpression and distribution of Toll-like receptors in the brain during murine neurocysticercosisAtrial natriuretic peptide protects against Staphylococcus aureus-induced lung injury and endothelial barrier dysfunctionCentral nervous system fibrosis is associated with fibrocyte-like infiltrates.Therapeutic Developments Targeting Toll-like Receptor-4-Mediated NeuroinflammationEffects of low dose GM-CSF on microglial inflammatory profiles to diverse pathogen-associated molecular patterns (PAMPs).Th17 cells in human disease.IL-1RI (interleukin-1 receptor type I) signalling is essential for host defence and hemichannel activity during acute central nervous system bacterial infection.Minocycline modulates neuroinflammation independently of its antimicrobial activity in staphylococcus aureus-induced brain abscess.Toll-like receptor (TLR) and inflammasome actions in the central nervous systemDifferential effects of interleukin-17 receptor signaling on innate and adaptive immunity during central nervous system bacterial infection.Biofilm-infected intracerebroventricular shunts elicit inflammation within the central nervous system.MyD88-dependent signals are essential for the host immune response in experimental brain abscess.Toll-like receptors in central nervous system glial inflammation and homeostasis.Glial toll-like receptor signaling in central nervous system infection and autoimmunity.The role of Toll-like receptors in CNS response to microbial challenge.The synthetic peroxisome proliferator-activated receptor-gamma agonist ciglitazone attenuates neuroinflammation and accelerates encapsulation in bacterial brain abscesses.Tumor necrosis factor-alpha (TNF-alpha) regulates Toll-like receptor 2 (TLR2) expression in microglia.MyD88 expression by CNS-resident cells is pivotal for eliciting protective immunity in brain abscesses.TLR2 deficiency leads to increased Th17 infiltrates in experimental brain abscesses.Staphylococcus aureus induces microglial inflammation via a glycogen synthase kinase 3beta-regulated pathway.The enhanced permeability retention effect: a new paradigm for drug targeting in infection.Pattern recognition receptors and central nervous system repair.The role of IL-17 in CNS diseases.Modulation of the local neutrophil response by a novel hyaluronic acid-binding peptide reduces bacterial burden during staphylococcal wound infection.
P2860
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P2860
Toll-like receptor 2 modulates the proinflammatory milieu in Staphylococcus aureus-induced brain abscess
description
2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2005
@ast
im November 2005 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2005/11/01)
@sk
vědecký článek publikovaný v roce 2005
@cs
wetenschappelijk artikel (gepubliceerd op 2005/11/01)
@nl
наукова стаття, опублікована в листопаді 2005
@uk
مقالة علمية نشرت في نوفمبر 2005 حول موضوع: دماغ
@ar
name
Toll-like receptor 2 modulates ...... s aureus-induced brain abscess
@ast
Toll-like receptor 2 modulates ...... s aureus-induced brain abscess
@en
Toll-like receptor 2 modulates ...... s aureus-induced brain abscess
@nl
type
label
Toll-like receptor 2 modulates ...... s aureus-induced brain abscess
@ast
Toll-like receptor 2 modulates ...... s aureus-induced brain abscess
@en
Toll-like receptor 2 modulates ...... s aureus-induced brain abscess
@nl
prefLabel
Toll-like receptor 2 modulates ...... s aureus-induced brain abscess
@ast
Toll-like receptor 2 modulates ...... s aureus-induced brain abscess
@en
Toll-like receptor 2 modulates ...... s aureus-induced brain abscess
@nl
P2093
P2860
P921
P1476
Toll-like receptor 2 modulates ...... s aureus-induced brain abscess
@en
P2093
Anessa Haney
Nilufer Esen
Patrick M. Mayes
Sarita Garg
Tammy Kielian
P2860
P304
P356
10.1128/IAI.73.11.7428-7435.2005
P407
P577
2005-11-01T00:00:00Z