Mice lacking NT-3, and its receptor TrkC, exhibit profound deficiencies in CNS glial cells
about
Promoting myelin repair and return of function in multiple sclerosisDifferential cellular expression of neurotrophins in cortical tubers of the tuberous sclerosis complexSmall molecule activators of the Trk receptors for neuroprotectionCombination of electroacupuncture and grafted mesenchymal stem cells overexpressing TrkC improves remyelination and function in demyelinated spinal cord of rats.Proneurotrophin-3 is a neuronal apoptotic ligand: evidence for retrograde-directed cell killing.Targeting oligodendrocyte protection and remyelination in multiple sclerosis.Expression of TrkC receptors in the developing brain of the Monodelphis opossum and its effect on the development of cortical cells.Neurotrophin-3 targets the translational initiation machinery in oligodendrocytes.Chinese herbal formula Tongluo Jiunao injection protects against cerebral ischemia by activating neurotrophin 3/tropomyosin-related kinase C pathwayFibroblast growth factor receptor signaling in oligodendrocytes regulates myelin sheath thickness.ERK1/ERK2 MAPK signaling is required to increase myelin thickness independent of oligodendrocyte differentiation and initiation of myelination.Brain-derived neurotrophic factor effects on oligodendrocyte progenitors of the basal forebrain are mediated through trkB and the MAP kinase pathway.Neurotrophic factor control of adult SVZ neurogenesis.Signaling mechanisms regulating myelination in the central nervous system.Neurotrophin-3 and a CREB-mediated signaling pathway regulate Bcl-2 expression in oligodendrocyte progenitor cells.Signaling by FGF Receptor 2, Not FGF Receptor 1, Regulates Myelin Thickness through Activation of ERK1/2-MAPK, Which Promotes mTORC1 Activity in an Akt-Independent Manner.Astrocytes induce proliferation of oligodendrocyte progenitor cells via connexin 47-mediated activation of the ERK/Id4 pathway.Strategies for repair of white matter: influence of osmolarity and microglia on proliferation and apoptosis of oligodendrocyte precursor cells in different basal culture media.Myelination and mTOR.
P2860
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P2860
Mice lacking NT-3, and its receptor TrkC, exhibit profound deficiencies in CNS glial cells
description
1999 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1999
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im April 1999 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 1999/04/01)
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vědecký článek publikovaný v roce 1999
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wetenschappelijk artikel (gepubliceerd op 1999/04/01)
@nl
наукова стаття, опублікована у квітні 1999
@uk
مقالة علمية (نشرت في أبريل 1999)
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name
Mice lacking NT-3, and its rec ...... eficiencies in CNS glial cells
@ast
Mice lacking NT-3, and its rec ...... eficiencies in CNS glial cells
@en
Mice lacking NT-3, and its rec ...... eficiencies in CNS glial cells
@nl
type
label
Mice lacking NT-3, and its rec ...... eficiencies in CNS glial cells
@ast
Mice lacking NT-3, and its rec ...... eficiencies in CNS glial cells
@en
Mice lacking NT-3, and its rec ...... eficiencies in CNS glial cells
@nl
prefLabel
Mice lacking NT-3, and its rec ...... eficiencies in CNS glial cells
@ast
Mice lacking NT-3, and its rec ...... eficiencies in CNS glial cells
@en
Mice lacking NT-3, and its rec ...... eficiencies in CNS glial cells
@nl
P2093
P1433
P1476
Mice lacking NT-3, and its rec ...... eficiencies in CNS glial cells
@en
P2093
P304
P356
10.1002/(SICI)1098-1136(199904)26:2<153::AID-GLIA6>3.3.CO;2-Q
P577
1999-04-01T00:00:00Z