Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
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Quo vadis, hair cell regeneration?Molecular mechanisms of inner ear developmentSensory hair cell development and regeneration: similarities and differencesWhere hearing starts: the development of the mammalian cochleaCLRN1 is nonessential in the mouse retina but is required for cochlear hair cell developmentMiR-210 promotes sensory hair cell formation in the organ of corti.Differentiation of the lateral compartment of the cochlea requires a temporally restricted FGF20 signalCanal cristae growth and fiber extension to the outer hair cells of the mouse ear require Prox1 activityHey2 regulation by FGF provides a Notch-independent mechanism for maintaining pillar cell fate in the organ of CortiUsher syndrome IIIA gene clarin-1 is essential for hair cell function and associated neural activationDisruption of fibroblast growth factor receptor 3 signaling results in defects in cellular differentiation, neuronal patterning, and hearing impairmentRole of chromatin remodeling gene Cecr2 in neurulation and inner ear developmentFgf20 is required for sensory epithelial specification in the developing cochleaShaping sound in space: the regulation of inner ear patterningAtoh1 as a Coordinator of Sensory Hair Cell Development and Regeneration in the Cochlea.MEKK4 Signaling Regulates Sensory Cell Development and Function in the Mouse Inner Ear.Manipulating cell fate in the cochlea: a feasible therapy for hearing loss.MAP3K1 function is essential for cytoarchitecture of the mouse organ of Corti and survival of auditory hair cells.Genetic rescue of Muenke syndrome model hearing loss reveals prolonged FGF-dependent plasticity in cochlear supporting cell fates.Inner ear supporting cells: rethinking the silent majority.Lgr5-positive supporting cells generate new hair cells in the postnatal cochlea.Postnatal development, maturation and aging in the mouse cochlea and their effects on hair cell regeneration.Notch signaling limits supporting cell plasticity in the hair cell-damaged early postnatal murine cochleaFgf signaling regulates development and transdifferentiation of hair cells and supporting cells in the basilar papillaCytoskeletal changes in actin and microtubules underlie the developing surface mechanical properties of sensory and supporting cells in the mouse cochlea.Intrinsic regenerative potential of murine cochlear supporting cells.CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.Hesr1 and Hesr2 may act as early effectors of Notch signaling in the developing cochleaHearing loss in a mouse model of Muenke syndromeExpression of ERK signaling inhibitors Dusp6, Dusp7, and Dusp9 during mouse ear development.Auditory hair cell defects as potential cause for sensorineural deafness in Wolf-Hirschhorn syndromeβ-Catenin is required for hair-cell differentiation in the cochlea.Building the world's best hearing aid; regulation of cell fate in the cochleaDelta/notch-like EGF-related receptor (DNER) is expressed in hair cells and neurons in the developing and adult mouse inner earRegulation of cell fate and patterning in the developing mammalian cochlea.Expression patterns of FGF receptors in the developing mammalian cochlea.Dynamic expression pattern of Sonic hedgehog in developing cochlear spiral ganglion neuronsRA and FGF signalling are required in the zebrafish otic vesicle to pattern and maintain ventral otic identitiesThe Fibroblast Growth Factor signaling pathwayThyroid hormone increases fibroblast growth factor receptor expression and disrupts cell mechanics in the developing organ of corti
P2860
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P2860
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
description
2007 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2007
@ast
im Februar 2007 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2007/02/01)
@sk
vědecký článek publikovaný v roce 2007
@cs
wetenschappelijk artikel (gepubliceerd op 2007/02/01)
@nl
наукова стаття, опублікована в лютому 2007
@uk
مقالة علمية (نشرت في فبراير 2007)
@ar
name
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
@ast
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
@en
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
@nl
type
label
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
@ast
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
@en
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
@nl
prefLabel
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
@ast
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
@en
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
@nl
P2093
P3181
P356
P1476
Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti
@en
P2093
Dale Cunningham
Olivia Bermingham-McDonogh
Toshinori Hayashi
P304
P3181
P356
10.1002/DVDY.21026
P577
2007-02-01T00:00:00Z