JAK2/STAT3, not ERK1/2, mediates interleukin-6-induced activation of inducible nitric-oxide synthase and decrease in contractility of adult ventricular myocytes
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Cerium oxide nanoparticle treatment ameliorates peritonitis-induced diaphragm dysfunctionInhibition of sarcoplasmic reticular function by chronic interleukin-6 exposure via iNOS in adult ventricular myocytesAG490 prevents cell death after exposure of rat astrocytes to hydrogen peroxide or proinflammatory cytokines: involvement of the Jak2/STAT pathwayMetabolic shifts toward glutamine regulate tumor growth, invasion and bioenergetics in ovarian cancerInterleukin-6 displays lung anti-inflammatory properties and exerts protective hemodynamic effects in a double-hit murine acute lung injuryActivation of JAK-STAT and nitric oxide signaling as a mechanism for donor heart dysfunctionSTAT3 balances myocyte hypertrophy vis-à-vis autophagy in response to Angiotensin II by modulating the AMPKα/mTOR axis.IL-6 induces regionally selective spinal cord injury in patients with the neuroinflammatory disorder transverse myelitis.Absence of SOCS3 in the cardiomyocyte increases mortality in a gp130-dependent manner accompanied by contractile dysfunction and ventricular arrhythmias.Cytokines and cardiovascular disease.The varying faces of IL-6: From cardiac protection to cardiac failure.Hepatitis C virus activates Bcl-2 and MMP-2 expression through multiple cellular signaling pathways.Roles of STATs signaling in cardiovascular diseases.IL-6 plays an obligatory role in late preconditioning via JAK-STAT signaling and upregulation of iNOS and COX-2.Protein Inhibitor of Activated STAT3 (PIAS3) Is Down-Regulated in Eutopic Endometrium of Women with Endometriosis.Targeting myeloid-derived suppressor cells using a novel adenosine monophosphate-activated protein kinase (AMPK) activator.Qishen granules inhibit myocardial inflammation injury through regulating arachidonic acid metabolism.Inducible NO synthase and antibacterial host defence in times of Th17/Th22/T22 immunity.Analytic review: Interleukin-6 in surgery, trauma, and critical care: part I: basic science.Interleukin-6 in surgery, trauma, and critical care part II: clinical implications.Phosphoinositide-3 kinase signaling in cardiac hypertrophy and heart failureNeutrophil roles in left ventricular remodeling following myocardial infarction.Cytokine Effects on Mechano-Induced Electrical Activity in Atrial Myocardium.Pro-inflammatory cytokines mediate GPCR dysfunction.Role of interleukin-6 in cardiac inflammation and dysfunction after burn complicated by sepsis.Interleukin-6 impairs endothelium-dependent NO-cGMP-mediated relaxation and enhances contraction in systemic vessels of pregnant rats.Mechanism of cardiac depression after trauma-hemorrhage: increased cardiomyocyte IL-6 and effect of sex steroids on IL-6 regulation and cardiac function.Both cGMP and peroxynitrite mediate chronic interleukin-6-induced negative inotropy in adult rat ventricular myocytes.Skeletal Muscle Function During the Progression of Cancer Cachexia in the Male ApcMin/+ Mouse.Altered inflammatory gene expression underlies increased susceptibility to murine postoperative ileus with advancing age.Differential effects of olmesartan and ramipril on inflammatory response after myocardial infarction in rats.Epac activation inhibits IL-6-induced cardiac myocyte dysfunction.Estrus cycle: influence on cardiac function following trauma-hemorrhage.Temporal cardiac remodeling post-myocardial infarction: dynamics and prognostic implications in personalized medicine.Neostigmine and pilocarpine attenuated tumour necrosis factor alpha expression and cardiac hypertrophy in the heart with pressure overload.Interleukin-6 contributes to the paracrine effects of cardiospheres cultured from human, murine and rat hearts.Inducible nitric oxide synthase is a major intermediate in signaling pathways for the survival of plasma cells.Cardiac Remodeling: Endothelial Cells Have More to Say Than Just NO.Cardiomyocytes and Macrophages Discourse on the Method to Govern Cardiac Repair
P2860
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P2860
JAK2/STAT3, not ERK1/2, mediates interleukin-6-induced activation of inducible nitric-oxide synthase and decrease in contractility of adult ventricular myocytes
description
2003 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի մայիսին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique Journal of Biological Chemistry
@fr
artículu científicu espublizáu en 2003
@ast
im Mai 2003 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2003/05/02)
@sk
vědecký článek publikovaný v roce 2003
@cs
wetenschappelijk artikel (gepubliceerd op 2003/05/02)
@nl
наукова стаття, опублікована в травні 2003
@uk
name
JAK2/STAT3, not ERK1/2, mediat ...... of adult ventricular myocytes
@ast
JAK2/STAT3, not ERK1/2, mediat ...... of adult ventricular myocytes
@en
JAK2/STAT3, not ERK1/2, mediat ...... of adult ventricular myocytes
@nl
type
label
JAK2/STAT3, not ERK1/2, mediat ...... of adult ventricular myocytes
@ast
JAK2/STAT3, not ERK1/2, mediat ...... of adult ventricular myocytes
@en
JAK2/STAT3, not ERK1/2, mediat ...... of adult ventricular myocytes
@nl
prefLabel
JAK2/STAT3, not ERK1/2, mediat ...... of adult ventricular myocytes
@ast
JAK2/STAT3, not ERK1/2, mediat ...... of adult ventricular myocytes
@en
JAK2/STAT3, not ERK1/2, mediat ...... of adult ventricular myocytes
@nl
P2093
P2860
P3181
P356
P1476
JAK2/STAT3, not ERK1/2, mediat ...... of adult ventricular myocytes
@en
P2093
Richard H. Kennedy
Shi J. Liu
P2860
P304
16304–16309
P3181
P356
10.1074/JBC.M212321200
P407
P577
2003-05-02T00:00:00Z