Mechanisms of prolonged presynaptic Ca2+ signaling and glutamate release induced by TRPV1 activation in rat sensory neurons
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Perspectives of TRPV1 Function on the Neurogenesis and Neural PlasticityUse of Capsaicin to Treat Pain: Mechanistic and Therapeutic ConsiderationsMitochondrial Ca2+ cycling facilitates activation of the transcription factor NFAT in sensory neurons.Pathological pain and the neuroimmune interface.Endocannabinoid-dependent LTD in a nociceptive synapse requires activation of a presynaptic TRPV-like receptor.Short-term increases in transient receptor potential vanilloid-1 mediate stress-induced enhancement of neuronal excitationTNF-α contributes to spinal cord synaptic plasticity and inflammatory pain: distinct role of TNF receptor subtypes 1 and 2.Nociceptor beta II, delta, and epsilon isoforms of PKC differentially mediate paclitaxel-induced spontaneous and evoked pain.The upregulation of α2δ-1 subunit modulates activity-dependent Ca2+ signals in sensory neuronsResolving TRPV1- and TNF-α-mediated spinal cord synaptic plasticity and inflammatory pain with neuroprotectin D1.Chronic pregabalin inhibits synaptic transmission between rat dorsal root ganglion and dorsal horn neurons in culture.Activation of transient receptor potential vanilloid-1 (TRPV1) influences how retinal ganglion cell neurons respond to pressure-related stress.Persistent inflammation increases GABA-induced depolarization of rat cutaneous dorsal root ganglion neurons in vitro.Molecular Targets of Cannabidiol in Neurological DisordersLong-term potentiation in hippocampal oriens interneurons: postsynaptic induction, presynaptic expression and evaluation of candidate retrograde factors.Absence of transient receptor potential vanilloid-1 accelerates stress-induced axonopathy in the optic projectionTRP channels, omega-3 fatty acids, and oxidative stress in neurodegeneration: from the cell membrane to intracellular cross-links.TRPV1: a stress response protein in the central nervous system.Understanding the role of mitochondria in the pathogenesis of chronic pain.TRP Channels Involved in Spontaneous L-Glutamate Release Enhancement in the Adult Rat Spinal Substantia Gelatinosa.Neuronal calcium signaling in chronic pain.Novel features on the regulation by mitochondria of calcium and secretion transients in chromaffin cells challenged with acetylcholine at 37°C.Long-term non-invasive interrogation of human dorsal root ganglion neuronal cultures on an integrated microfluidic multielectrode array platform.Group II/III metabotropic glutamate receptors exert endogenous activity-dependent modulation of TRPV1 receptors on peripheral nociceptorsThe quantal catecholamine release from mouse chromaffin cells challenged with repeated ACh pulses is regulated by the mitochondrial Na+ /Ca2+ exchanger.SK2 channels regulate mitochondrial respiration and mitochondrial Ca2+ uptake.Conjugated polymers mediate effective activation of the Mammalian Ion Channel Transient Receptor Potential Vanilloid 1.Functional interactions between NMDA receptors and TRPV1 in trigeminal sensory neurons mediate mechanical hyperalgesia in the rat masseter muscle.Peripheral calcium-permeable AMPA receptors regulate chronic inflammatory pain in mice.Multiple PKCε-dependent mechanisms mediating mechanical hyperalgesiaN-Arachidonoyl Dopamine: A Novel Endocannabinoid and Endovanilloid with Widespread Physiological and Pharmacological Activities.Inhibitory neurones of the spinal substantia gelatinosa mediate interaction of signals from primary afferents.Mechanism of the persistent sodium current activator veratridine-evoked Ca elevation: implication for epilepsy.Zingerone enhances glutamatergic spontaneous excitatory transmission by activating TRPA1 but not TRPV1 channels in the adult rat substantia gelatinosa.Presynaptic inhibition of transient receptor potential vanilloid type 1 (TRPV1) receptors by noradrenaline in nociceptive neurons.Dopamine modulation of transient receptor potential vanilloid type 1 (TRPV1) receptor in dorsal root ganglia neurons.
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P2860
Mechanisms of prolonged presynaptic Ca2+ signaling and glutamate release induced by TRPV1 activation in rat sensory neurons
description
2008 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի մայիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2008
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im Mai 2008 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 2008/05/14)
@sk
vědecký článek publikovaný v roce 2008
@cs
wetenschappelijk artikel (gepubliceerd op 2008/05/14)
@nl
наукова стаття, опублікована в травні 2008
@uk
مقالة علمية (نشرت في 14-5-2008)
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name
Mechanisms of prolonged presyn ...... ivation in rat sensory neurons
@ast
Mechanisms of prolonged presyn ...... ivation in rat sensory neurons
@en
Mechanisms of prolonged presyn ...... ivation in rat sensory neurons
@nl
type
label
Mechanisms of prolonged presyn ...... ivation in rat sensory neurons
@ast
Mechanisms of prolonged presyn ...... ivation in rat sensory neurons
@en
Mechanisms of prolonged presyn ...... ivation in rat sensory neurons
@nl
prefLabel
Mechanisms of prolonged presyn ...... ivation in rat sensory neurons
@ast
Mechanisms of prolonged presyn ...... ivation in rat sensory neurons
@en
Mechanisms of prolonged presyn ...... ivation in rat sensory neurons
@nl
P2860
P1476
Mechanisms of prolonged presyn ...... ivation in rat sensory neurons
@en
P2093
Man-Su Kim
Yuliya V Medvedeva
P2860
P304
P356
10.1523/JNEUROSCI.4810-07.2008
P407
P577
2008-05-01T00:00:00Z