The secreted metalloprotease ADAMTS20 is required for melanoblast survival
about
Mutations in MITF and PAX3 cause "splashed white" and other white spotting phenotypes in horsesThe ADAMTS (A Disintegrin and Metalloproteinase with Thrombospondin motifs) familyA disintegrin-like and metalloprotease (reprolysin-type) with thrombospondin type 1 motif (ADAMTS) superfamily: functions and mechanismsThe Function and Roles of ADAMTS-7 in Inflammatory DiseasesLoss of C. elegans GON-1, an ADAMTS9 Homolog, Decreases Secretion Resulting in Altered Lifespan and Dauer FormationA sensitized mutagenesis screen identifies Gli3 as a modifier of Sox10 neurocristopathyBiosynthesis and expression of a disintegrin-like and metalloproteinase domain with thrombospondin-1 repeats-15: a novel versican-cleaving proteoglycanaseMolecular and developmental contributions to divergent pigment patterns in marine and freshwater sticklebacksFascin 1 is transiently expressed in mouse melanoblasts during development and promotes migration and proliferation.Cell-surface processing of the metalloprotease pro-ADAMTS9 is influenced by the chaperone GRP94/gp96ADAMTS9 is a cell-autonomously acting, anti-angiogenic metalloprotease expressed by microvascular endothelial cells.A structural variant in the 5'-flanking region of the TWIST2 gene affects melanocyte development in belted cattleA customized pigmentation SNP array identifies a novel SNP associated with melanoma predisposition in the SLC45A2 gene.ADAMTS4 and ADAMTS5 knockout mice are protected from versican but not aggrecan or brevican proteolysis during spinal cord injury.The multiple, complex roles of versican and its proteolytic turnover by ADAMTS proteases during embryogenesis.A new Adamts9 conditional mouse allele identifies its non-redundant role in interdigital web regression.Determinants of versican-V1 proteoglycan processing by the metalloproteinase ADAMTS5Mutational and functional analysis reveals ADAMTS18 metalloproteinase as a novel driver in melanoma.Age- and pregnancy-associated DNA methylation changes in mammary epithelial cells.Genome-wide association studies in dogs and humans identify ADAMTS20 as a risk variant for cleft lip and palate.Post-transcriptional Regulation of Keratinocyte Progenitor Cell Expansion, Differentiation and Hair Follicle Regression by miR-22.ADAMTS9-Mediated Extracellular Matrix Dynamics Regulates Umbilical Cord Vascular Smooth Muscle Differentiation and RotationIdentification of a novel ADAMTS9/GON-1 function for protein transport from the ER to the GolgiNeural development is dependent on the function of specificity protein 2 in cell cycle progression.The secreted AdamTS-A metalloprotease is required for collective cell migration.The EJC component Magoh regulates proliferation and expansion of neural crest-derived melanocytes.Emerging Roles of ADAMTSs in Angiogenesis and Cancer.Impaired ADAMTS9 secretion: A potential mechanism for eye defects in Peters Plus Syndrome.Adamts5, the gene encoding a proteoglycan-degrading metalloprotease, is expressed by specific cell lineages during mouse embryonic development and in adult tissues.Stripes and belly-spots -- a review of pigment cell morphogenesis in vertebrates.Kit and foxd3 genetically interact to regulate melanophore survival in zebrafish.ADAMTS metalloproteases generate active versican fragments that regulate interdigital web regression.Matrix metalloproteinases, a disintegrin and metalloproteinases, and a disintegrin and metalloproteinases with thrombospondin motifs in non-neoplastic diseases.Can mesenchymal cells undergo collective cell migration? The case of the neural crest.Modeling melanoblast development.Extracellular metalloproteinases in neural crest development and craniofacial morphogenesis.The biology of melanocyte and melanocyte stem cell.Immune-induced expression of lipocalin-2 in brain endothelial cells: relationship with interleukin-6, cyclooxygenase-2 and the febrile response.Reduced versican cleavage due to Adamts9 haploinsufficiency is associated with cardiac and aortic anomalies.Cooperation of two ADAMTS metalloproteases in closure of the mouse palate identifies a requirement for versican proteolysis in regulating palatal mesenchyme proliferation.
P2860
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P2860
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
description
2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2008
@ast
im Februar 2008 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2008/02/29)
@sk
vědecký článek publikovaný v roce 2008
@cs
wetenschappelijk artikel (gepubliceerd op 2008/02/29)
@nl
наукова стаття, опублікована в лютому 2008
@uk
مقالة علمية (نشرت في 29-2-2008)
@ar
name
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
@ast
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
@en
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
@nl
type
label
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
@ast
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
@en
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
@nl
prefLabel
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
@ast
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
@en
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
@nl
P2093
P2860
P3181
P1433
P1476
The secreted metalloprotease ADAMTS20 is required for melanoblast survival
@en
P2093
Debra L. Silver
Mary E. Young
Robert Somerville
Suneel S. Apte
William J. Pavan
P2860
P304
P3181
P356
10.1371/JOURNAL.PGEN.1000003
P50
P577
2008-02-29T00:00:00Z