Distribution of catechol-O-methyltransferase (COMT) proteins and enzymatic activities in wild-type and soluble COMT deficient mice
about
Monoaminergic and Histaminergic Strategies and Treatments in Brain DiseasesOrientation and cellular distribution of membrane-bound catechol-O-methyltransferase in cortical neurons: implications for drug developmentMice deficient in transmembrane prostatic acid phosphatase display increased GABAergic transmission and neurological alterations.G-protein βγ subunit dimers modulate kidney repair after ischemia-reperfusion injury in rats.Noradrenergic regulation of glial activation: molecular mechanisms and therapeutic implications.Sexually dimorphic effects of catechol-O-methyltransferase (COMT) inhibition on dopamine metabolism in multiple brain regionsThe catechol-O-methyltransferase inhibitor, tolcapone, increases the bioavailability of unmethylated (-)-epigallocatechin-3-gallate in micePulse Pressure Magnifies the Effect of COMT Val(158)Met on 15 Years Episodic Memory TrajectoriesCatechol-O-methyltransferase (COMT) protein expression and activity after dopaminergic and noradrenergic lesions of the rat brain.Complexity of dopamine metabolism.Quantitative liver-specific protein fingerprint in blood: a signature for hepatotoxicity.A Novel Sensitive Method to Measure Catechol-O-Methyltransferase Activity Unravels the Presence of This Activity in Extracellular Vesicles Released by Rat Hepatocytes.Genetic variation in COMT activity impacts learning and dopamine release capacity in the striatum.Cloning and expression of a novel catechol-O-methyltransferase in common marmosetsA prolyl oligopeptidase inhibitor, KYP-2047, reduces α-synuclein protein levels and aggregates in cellular and animal models of Parkinson's disease.Characterization of non-nitrocatechol pan and isoform specific catechol-O-methyltransferase inhibitors and substratesDopamine oxidation and autophagyLack of Parkin anticipates the phenotype and affects mitochondrial morphology and mtDNA levels in a mouse model of Parkinson's Disease.Organic cation transporter 3 (OCT3) is localized to intracellular and surface membranes in select glial and neuronal cells within the basolateral amygdaloid complex of both rats and mice.
P2860
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P2860
Distribution of catechol-O-methyltransferase (COMT) proteins and enzymatic activities in wild-type and soluble COMT deficient mice
description
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2010
@ast
scientific journal article
@en
vedecký článok (publikovaný 2010/06/01)
@sk
vědecký článek publikovaný v roce 2010
@cs
wetenschappelijk artikel (gepubliceerd op 2010/06/01)
@nl
наукова стаття, опублікована в червні 2010
@uk
مقالة علمية (نشرت في يونيو 2010)
@ar
name
Distribution of catechol-O-met ...... nd soluble COMT deficient mice
@ast
Distribution of catechol-O-met ...... nd soluble COMT deficient mice
@en
Distribution of catechol-O-met ...... nd soluble COMT deficient mice
@nl
type
label
Distribution of catechol-O-met ...... nd soluble COMT deficient mice
@ast
Distribution of catechol-O-met ...... nd soluble COMT deficient mice
@en
Distribution of catechol-O-met ...... nd soluble COMT deficient mice
@nl
prefLabel
Distribution of catechol-O-met ...... nd soluble COMT deficient mice
@ast
Distribution of catechol-O-met ...... nd soluble COMT deficient mice
@en
Distribution of catechol-O-met ...... nd soluble COMT deficient mice
@nl
P2093
P1476
Distribution of catechol-O-met ...... nd soluble COMT deficient mice
@en
P2093
Nadia Schendzielorz
Pekka T. Männistö
Timo T. Myöhänen
P304
P356
10.1111/J.1471-4159.2010.06723.X
P407
P577
2010-06-01T00:00:00Z