Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
about
Identification of protein tyrosine phosphatase 1B and casein as substrates for 124-v-Mos.How eggs arrest at metaphase II: MPF stabilisation plus APC/C inhibition equals Cytostatic FactorOocyte maturation failure: a syndrome of bad eggsMos activates myogenic differentiation by promoting heterodimerization of MyoD and E12 proteinsMeiotic spindle stability depends on MAPK-interacting and spindle-stabilizing protein (MISS), a new MAPK substrateAnimal models of ovarian cancerModulation of cell cycle control during oocyte-to-embryo transitionsThe BTG4 and CAF1 complex prevents the spontaneous activation of eggs by deadenylating maternal mRNAs.Evidence of an interaction between Mos and Hsp70: a role of the Mos residue serine 3 in mediating Hsp70 associationEmi1 is required for cytostatic factor arrest in vertebrate eggsRegulated CPEB phosphorylation during meiotic progression suggests a mechanism for temporal control of maternal mRNA translationPhosphorylation of high-mobility group protein A2 by Nek2 kinase during the first meiotic division in mouse spermatocytesMammalian Emi2 mediates cytostatic arrest and transduces the signal for meiotic exit via Cdc20.Role of the inositol polyphosphate-4-phosphatase type II Inpp4b in the generation of ovarian teratomasFunctional interaction between p90Rsk2 and Emi1 contributes to the metaphase arrest of mouse oocytesRecruitment of Orc6l, a dormant maternal mRNA in mouse oocytes, is essential for DNA replication in 1-cell embryosThe Mos/mitogen-activated protein kinase (MAPK) pathway regulates the size and degradation of the first polar body in maturing mouse oocytesEmbryonic poly(A)-binding protein (EPAB) is required for oocyte maturation and female fertility in miceMice deficient in oocyte-specific oligoadenylate synthetase-like protein OAS1D display reduced fertilityMouse Emi2 is required to enter meiosis II by reestablishing cyclin B1 during interkinesisImpaired growth and fertility of cAMP-specific phosphodiesterase PDE4D-deficient miceThe Mos/MAP kinase pathway stabilizes c-Fos by phosphorylation and augments its transforming activity in NIH 3T3 cellsCyclins and cyclin-dependent kinases: a biochemical viewA morpholino phenocopy of the mouse mos mutation.Selective degradation of transcripts during meiotic maturation of mouse oocytes.Multipolar mitosis of tetraploid cells: inhibition by p53 and dependency on Mos.Expression of genes involved in mammalian meiosis during the transition from egg to embryo.Genomewide discovery and classification of candidate ovarian fertility genes in the mouseIn silico identification and molecular characterization of genes predominantly expressed in the fish oocyteSuppression of chemically induced and spontaneous mouse oocyte activation by AMP-activated protein kinase.α-endosulfine (ENSA) regulates exit from prophase I arrest in mouse oocytesKicked by Mos and tuned by MPF-the initiation of the MAPK cascade in Xenopus oocytes.Proteasomal activity is required to initiate and to sustain translational activation of messenger RNA encoding the stem-loop-binding protein during meiotic maturation in mice.Effects of thioglycolic acid on parthenogenetic activation of Xenopus oocytes.Meiotic cell cycle in Xenopus oocytes is independent of cdk2 kinase.MAP kinase links the fertilization signal transduction pathway to the G1/S-phase transition in starfish eggs.A member of the Ste20/PAK family of protein kinases is involved in both arrest of Xenopus oocytes at G2/prophase of the first meiotic cell cycle and in prevention of apoptosis.Cyclin B/cdc2 induces c-Mos stability by direct phosphorylation in Xenopus oocytes.The mitogen-activated protein kinase signaling pathway stimulates mos mRNA cytoplasmic polyadenylation during Xenopus oocyte maturationPhosphorylation of IP3R1 and the regulation of [Ca2+]i responses at fertilization: a role for the MAP kinase pathway.
P2860
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P2860
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
description
1994 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique Nature
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artículu científicu espublizáu en 1994
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scientific journal article
@en
vedecký článok (publikovaný 1994/07/07)
@sk
vědecký článek publikovaný v roce 1994
@cs
wetenschappelijk artikel (gepubliceerd op 1994/07/07)
@nl
наукова стаття, опублікована в липні 1994
@uk
مقالة علمية (نشرت في 7-7-1994)
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name
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
@ast
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
@en
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
@nl
type
label
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
@ast
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
@en
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
@nl
prefLabel
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
@ast
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
@en
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
@nl
P2093
P3181
P356
P1433
P1476
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs
@en
P2093
M. B. Carlton
M. J. Evans
W. H. Colledge
P2888
P3181
P356
10.1038/370065A0
P407
P577
1994-07-07T00:00:00Z
P5875
P6179
1007014612