An ADIOL-ERβ-CtBP transrepression pathway negatively regulates microglia-mediated inflammation
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Regulation of Estrogen Receptor α Expression in the Hypothalamus by Sex Steroids: Implication in the Regulation of Energy HomeostasisMinireview: the year in review of estrogen regulation of metabolismRole of microglia in CNS autoimmunityNeuroprotective effects of estrogens and androgens in CNS inflammation and neurodegenerationEstrogen ameliorates microglial activation by inhibiting the Kir2.1 inward-rectifier K(+) channel.Resveratrol modulates the inflammatory response via an estrogen receptor-signal integration network5-AED enhances survival of irradiated mice in a G-CSF-dependent manner, stimulates innate immune cell function, reduces radiation-induced DNA damage and induces genes that modulate cell cycle progression and apoptosisInteractions between inflammation, sex steroids, and Alzheimer's disease risk factorsMinocycline modulates human social decision-making: possible impact of microglia on personality-oriented social behaviorsSuppression of neuroinflammation by astrocytic dopamine D2 receptors via αB-crystallin17β-estradiol represses myogenic differentiation by increasing ubiquitin-specific peptidase 19 through estrogen receptor αEstrogens regulate neuroinflammatory genes via estrogen receptors α and β in the frontal cortex of middle-aged female ratsOvariectomy and subsequent treatment with estrogen receptor agonists tune the innate immune system of the hippocampus in middle-aged female ratsR-flurbiprofen attenuates experimental autoimmune encephalomyelitis in micePregnancy, sex and hormonal factors in multiple sclerosis.Nudging oligodendrocyte intrinsic signaling to remyelinate and repair: Estrogen receptor ligand effects.Estrogen receptor β ligand therapy activates PI3K/Akt/mTOR signaling in oligodendrocytes and promotes remyelination in a mouse model of multiple sclerosis.Estrogen receptor (ER) β expression in oligodendrocytes is required for attenuation of clinical disease by an ERβ ligandMissing and Possible Link between Neuroendocrine Factors, Neuropsychiatric Disorders, and MicrogliaHypothalamic programming of systemic ageing involving IKK-β, NF-κB and GnRHPotent and multiple regulatory actions of microglial glucocorticoid receptors during CNS inflammation.Development of subtype-selective oestrogen receptor-based therapeutics.Diarylpropionitrile (DPN) enantiomers: synthesis and evaluation of estrogen receptor β-selective ligands.Estrogen receptors alpha (ERα) and beta (ERβ): subtype-selective ligands and clinical potential.Microglia are essential to masculinization of brain and behavior.17β-Hydroxysteroid dehydrogenase type 14 is a predictive marker for tamoxifen response in oestrogen receptor positive breast cancerVersatility or promiscuity: the estrogen receptors, control of ligand selectivity and an update on subtype selective ligands.Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.Multiple functional therapeutic effects of the estrogen receptor β agonist indazole-Cl in a mouse model of multiple sclerosisC-Terminal Binding Protein: A Molecular Link between Metabolic Imbalance and Epigenetic Regulation in Breast Cancer.The 2010 Philip S. Portoghese Medicinal Chemistry Lectureship: addressing the "core issue" in the design of estrogen receptor ligandsHypothalamic inflammation: a double-edged sword to nutritional diseases.Microglia: scapegoat, saboteur, or something else?Sex-related factors in multiple sclerosis susceptibility and progression.Estrogen receptor β and 17β-hydroxysteroid dehydrogenase type 6, a growth regulatory pathway that is lost in prostate cancerSelective oestrogen receptor modulators differentially potentiate brain mitochondrial function.Reduction of dendritic spines and elevation of GABAergic signaling in the brains of mice treated with an estrogen receptor β ligandOvariectomy shortens the life span of female miceA local paracrine and endocrine network involving TGFβ, Cox-2, ROS, and estrogen receptor β influences reactive stromal cell regulation of prostate cancer cell motility.A lack of ovarian function increases neuroinflammation in aged mice.
P2860
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P2860
An ADIOL-ERβ-CtBP transrepression pathway negatively regulates microglia-mediated inflammation
description
2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2011
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im Mai 2011 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
@en
vedecký článok (publikovaný 2011/05/13)
@sk
vědecký článek publikovaný v roce 2011
@cs
wetenschappelijk artikel (gepubliceerd op 2011/05/13)
@nl
наукова стаття, опублікована в травні 2011
@uk
مقالة علمية (نشرت في 13-5-2011)
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name
An ADIOL-ERβ-CtBP transrepress ...... icroglia-mediated inflammation
@ast
An ADIOL-ERβ-CtBP transrepress ...... icroglia-mediated inflammation
@en
An ADIOL-ERβ-CtBP transrepress ...... icroglia-mediated inflammation
@nl
type
label
An ADIOL-ERβ-CtBP transrepress ...... icroglia-mediated inflammation
@ast
An ADIOL-ERβ-CtBP transrepress ...... icroglia-mediated inflammation
@en
An ADIOL-ERβ-CtBP transrepress ...... icroglia-mediated inflammation
@nl
prefLabel
An ADIOL-ERβ-CtBP transrepress ...... icroglia-mediated inflammation
@ast
An ADIOL-ERβ-CtBP transrepress ...... icroglia-mediated inflammation
@en
An ADIOL-ERβ-CtBP transrepress ...... icroglia-mediated inflammation
@nl
P2093
P2860
P921
P3181
P1433
P1476
An ADIOL-ERβ-CtBP transrepress ...... icroglia-mediated inflammation
@en
P2093
Andrew C. Li
Christopher K. Glass
Jana G. Collier
John A. Katzenellenbogen
Kaoru Saijo
P2860
P304
P3181
P356
10.1016/J.CELL.2011.03.050
P407
P577
2011-05-13T00:00:00Z