Genome-wide association study of recurrent early-onset major depressive disorder - sprookjes - yvandenbroek - TriplyDB

Genome-wide association study of recurrent early-onset major depressive disorder

Genome-wide association study of recurrent early-onset major depressive disorder

http://www.wikidata.org/entity/Q28943558

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Polymorphisms in melatonin synthesis pathways: possible influences on depression The neuronal transporter gene SLC6A15 confers risk to major depression Proteoglycans and neuronal migration in the cerebral cortex during development and disease Neurogenetics of depression: a focus on reward processing and stress sensitivity The genetics of major depression Toward an integration of cognitive and genetic models of risk for depression Human genetic disorders and knockout mice deficient in glycosaminoglycan Genetic determinants of depression: recent findings and future directions Restoration of Sp4 in Forebrain GABAergic Neurons Rescues Hypersensitivity to Ketamine in Sp4 Hypomorphic Mice.Genome-wide linkage on chromosome 10q26 for a dimensional scale of major depression The association between lower educational attainment and depression owing to shared genetic effects? Results in ~25,000 subjects MK4MDD: a multi-level knowledge base and analysis platform for major depressive disorder Novel loci for major depression identified by genome-wide association study of Sequenced Treatment Alternatives to Relieve Depression and meta-analysis of three studies Genome-wide association analysis accounting for environmental factors through propensity-score matching: application to stressful live events in major depressive disorder A genome-wide association study of depressive symptoms A mega-analysis of genome-wide association studies for major depressive disorder Electrophysiological Endophenotypes for Schizophrenia Prolonged Ketamine Effects in Sp4 Hypomorphic Mice: Mimicking Phenotypes of Schizophrenia.Mood disorder susceptibility gene CACNA1C modifies mood-related behaviors in mice and interacts with sex to influence behavior in mice and diagnosis in humans.Reduced NMDAR1 expression in the Sp4 hypomorphic mouse may contribute to endophenotypes of human psychiatric disorders GlyT-1 Inhibition Attenuates Attentional But Not Learning or Motivational Deficits of the Sp4 Hypomorphic Mouse Model Relevant to Psychiatric Disorders Genomic structural variation in psychiatric disorders.Posttraumatic stress disorder increases risk for suicide attempt in adults with recurrent major depression The genetics of major depression: moving beyond the monoamine hypothesis.Prioritization and evaluation of depression candidate genes by combining multidimensional data resources.Overview of the genetics of major depressive disorder Evidence-based psychiatric genetics, AKA the false dichotomy between common and rare variant hypotheses.A fine-mapping study of 7 top scoring genes from a GWAS for major depressive disorder.Copy number variation in subjects with major depressive disorder who attempted suicide.Genome-wide and gene-based association studies of anxiety disorders in European and African American samples Allelic association, DNA resequencing and copy number variation at the metabotropic glutamate receptor GRM7 gene locus in bipolar disorder.A recessive genetic model and runs of homozygosity in major depressive disorder.Premorbid risk factors for major depressive disorder: are they associated with early onset and recurrent course?Discovering schizophrenia endophenotypes in randomly ascertained pedigrees.Joint analysis of psychiatric disorders increases accuracy of risk prediction for schizophrenia, bipolar disorder, and major depressive disorder.A conserved BDNF, glutamate- and GABA-enriched gene module related to human depression identified by coexpression meta-analysis and DNA variant genome-wide association studies.Applying polygenic risk scores to postpartum depression Microarray profiling and co-expression network analysis of circulating lncRNAs and mRNAs associated with major depressive disorder Problematic assumptions have slowed down depression research: why symptoms, not syndromes are the way forward.Depression sum-scores don't add up: why analyzing specific depression symptoms is essential

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P2860

Genome-wide association study of recurrent early-onset major depressive disorder

http://www.wikidata.org/entity/Q28943558

description

2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

artículu científicu espublizáu en 2011

@ast

im Februar 2010 veröffentlichter wissenschaftlicher Artikel

@de

scientific journal article

@en

wetenschappelijk artikel (gepubliceerd op 2011/02/01)

@nl

наукова стаття, опублікована в лютому 2011

@uk

مقالة علمية (نشرت في فبراير 2011)

@ar

name

Genome-wide association study of recurrent early-onset major depressive disorder

@ast

Genome-wide association study of recurrent early-onset major depressive disorder

@en

type

label

Genome-wide association study of recurrent early-onset major depressive disorder

@ast

Genome-wide association study of recurrent early-onset major depressive disorder

@en

prefLabel

Genome-wide association study of recurrent early-onset major depressive disorder

@ast

Genome-wide association study of recurrent early-onset major depressive disorder

@en

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Molecular Psychiatry

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Genome-wide association study of recurrent early-onset major depressive disorder

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A Zvinyatskovskiy

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D Jancic

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J Shi

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K Neimanas

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Common variants at 30 loci contribute to polygenic dyslipidemia

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Collaborative genome-wide association analysis supports a role for ANK3 and CACNA1C in bipolar disorder

The molecular neurobiology of depression

PLINK: a tool set for whole-genome association and population-based linkage analyses

Principal components analysis corrects for stratification in genome-wide association studies

Newly identified loci that influence lipid concentrations and risk of coronary artery disease

No significant association of 14 candidate genes with schizophrenia in a large European ancestry sample: implications for psychiatric genetics

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193-201

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Myrna M. Weissman

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41400127

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20125088

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