Transcription factor Nrf2 coordinately regulates a group of oxidative stress-inducible genes in macrophages - sprookjes - yvandenbroek - TriplyDB

Transcription factor Nrf2 coordinately regulates a group of oxidative stress-inducible genes in macrophages

Transcription factor Nrf2 coordinately regulates a group of oxidative stress-inducible genes in macrophages

http://www.wikidata.org/entity/Q29614557

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Chemoprotection by phenolic antioxidants Ubiquitination of Keap1, a BTB-Kelch substrate adaptor protein for Cul3, targets Keap1 for degradation by a proteasome-independent pathway p62/SQSTM1 is a target gene for transcription factor NRF2 and creates a positive feedback loop by inducing antioxidant response element-driven gene transcription Bach1 functions as a hypoxia-inducible repressor for the heme oxygenase-1 gene in human cells Curcumin activates the haem oxygenase-1 gene via regulation of Nrf2 and the antioxidant-responsive element Differential induction of mafF, mafG and mafK expression by electrophile-response-element activators Keap1 is a redox-regulated substrate adaptor protein for a Cul3-dependent ubiquitin ligase complex.Heme regulates gene expression by triggering Crm1-dependent nuclear export of Bach1 Small Maf proteins serve as transcriptional cofactors for keratinocyte differentiation in the Keap1-Nrf2 regulatory pathway Nrf2 regulates haematopoietic stem cell function Understanding the human health effects of chemical mixtures.The importance of antioxidants which play the role in cellular response against oxidative/nitrosative stress: current state Mitochondrial Dysfunction in Alzheimer's Disease and the Rationale for Bioenergetics Based Therapies The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance Value of monitoring Nrf2 activity for the detection of chemical and oxidative stress Overview of Nrf2 as Therapeutic Target in Epilepsy The role of Nrf2: adipocyte differentiation, obesity, and insulin resistance Effect of redox modulating NRF2 activators on chronic kidney disease Redox status in mammalian cells and stem cells during culture in vitro: critical roles of Nrf2 and cystine transporter activity in the maintenance of redox balance A perspective on dietary phytochemicals and cancer chemoprevention: oxidative stress, nrf2, and epigenomics Emerging functional cross-talk between the Keap1-Nrf2 system and mitochondria Roles nrf2 plays in myeloid cells and related disorders Myc-driven overgrowth requires unfolded protein response-mediated induction of autophagy and antioxidant responses in Drosophila melanogaster Redox signaling in cardiovascular pathophysiology: A focus on hydrogen peroxide and vascular smooth muscle cells Cross Talk between Two Antioxidant Systems, Thioredoxin and DJ-1: Consequences for Cancer Small maf (MafG and MafK) proteins negatively regulate antioxidant response element-mediated expression and antioxidant induction of the NAD(P)H:Quinone oxidoreductase1 gene Nrf1 is critical for redox balance and survival of liver cells during development Small Maf compound mutants display central nervous system neuronal degeneration, aberrant transcription, and Bach protein mislocalization coincident with myoclonus and abnormal startle response Liver-specific inactivation of the Nrf1 gene in adult mouse leads to nonalcoholic steatohepatitis and hepatic neoplasia Oxidative and electrophilic stress induces multidrug resistance-associated protein transporters via the nuclear factor-E2-related factor-2 transcriptional pathway Transcription factor NF-E2-related factor 1 impairs glucose metabolism in mice Differential responses of the Nrf2-Keap1 system to laminar and oscillatory shear stresses in endothelial cells Embryonic lethality and fetal liver apoptosis in mice lacking all three small Maf proteins Genetic evidence that small maf proteins are essential for the activation of antioxidant response element-dependent genes.Nrf2-Keap1 signaling pathway regulates human UGT1A1 expression in vitro and in transgenic UGT1 mice Nrf2 transcriptionally activates the mafG gene through an antioxidant response element Selective autophagy mediated by autophagic adapter proteins PMI: a ΔΨm independent pharmacological regulator of mitophagy Nuclear Factor-Erythroid-2-Related Factor 2 in Aging and Lung Fibrosis CACUL1/CAC1 Regulates the Antioxidant Response by Stabilizing Nrf2

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P2860

Transcription factor Nrf2 coordinately regulates a group of oxidative stress-inducible genes in macrophages

http://www.wikidata.org/entity/Q29614557

description

2000 nî lūn-bûn

@nan

2000 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2000 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2000年の論文

@ja

2000年論文

@yue

2000年論文

@zh-hant

2000年論文

@zh-hk

2000年論文

@zh-mo

2000年論文

@zh-tw

2000年论文

@wuu

name

Transcription factor Nrf2 coor ...... inducible genes in macrophages

@ast

Transcription factor Nrf2 coor ...... inducible genes in macrophages

@en

type

label

Transcription factor Nrf2 coor ...... inducible genes in macrophages

@ast

Transcription factor Nrf2 coor ...... inducible genes in macrophages

@en

prefLabel

Transcription factor Nrf2 coor ...... inducible genes in macrophages

@ast

Transcription factor Nrf2 coor ...... inducible genes in macrophages

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Transcription factor Nrf2 coor ...... inducible genes in macrophages

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P2093

Bannai S

http://www.w3.org/2001/XMLSchema#string

Ishii T

http://www.w3.org/2001/XMLSchema#string

Itoh K

http://www.w3.org/2001/XMLSchema#string

Katoh Y

http://www.w3.org/2001/XMLSchema#string

Sato H

http://www.w3.org/2001/XMLSchema#string

Takahashi S

http://www.w3.org/2001/XMLSchema#string

Yamamoto M

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Yanagawa T

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16023-9

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scholarly article

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2109373

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10.1074/JBC.275.21.16023

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21

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275

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2000-05-26T00:00:00Z

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10821856

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