about
Buruli ulcer (M. ulcerans infection): new insights, new hope for disease control.Deciphering the genetic basis for polyketide variation among mycobacteria producing mycolactonesAnalysis of the small RNA transcriptional response in multidrug-resistant Staphylococcus aureus after antimicrobial exposureGiant plasmid-encoded polyketide synthases produce the macrolide toxin of Mycobacterium ulceransReduced vancomycin susceptibility in Staphylococcus aureus, including vancomycin-intermediate and heterogeneous vancomycin-intermediate strains: resistance mechanisms, laboratory detection, and clinical implicationsCorrection: Buruli Ulcer (M. ulcerans Infection): New Insights, New Hope for Disease ControlInvestigating the role of free-living amoebae as a reservoir for Mycobacterium ulceransEvolution of multidrug resistance during Staphylococcus aureus infection involves mutation of the essential two component regulator WalKRThe cell wall-associated mycolactone polyketide synthases are necessary but not sufficient for mycolactone biosynthesisRegulation of mycolactone, the Mycobacterium ulcerans toxin, depends on nutrient sourceAnalysis of the vaccine potential of plasmid DNA encoding nine mycolactone polyketide synthase domains in Mycobacterium ulcerans infected miceIn-vitro activity of avermectins against Mycobacterium ulceransEvolution of two distinct phylogenetic lineages of the emerging human pathogen Mycobacterium ulcerans.Different bacterial gene expression patterns and attenuated host immune responses are associated with the evolution of low-level vancomycin resistance during persistent methicillin-resistant Staphylococcus aureus bacteraemia.First isolation of Mycobacterium ulcerans from an aquatic environment: the end of a 60-year search?Comprehensive proteome analysis of Mycobacterium ulcerans and quantitative comparison of mycolactone biosynthesis.Exposure Risk for Infection and Lack of Human-to-Human Transmission of Mycobacterium ulcerans Disease, Australia.Two novel point mutations in clinical Staphylococcus aureus reduce linezolid susceptibility and switch on the stringent response to promote persistent infection.Mycobacterium ulcerans low infectious dose and mechanical transmission support insect bites and puncturing injuries in the spread of Buruli ulcer.Single nucleotide polymorphism typing of Mycobacterium ulcerans reveals focal transmission of buruli ulcer in a highly endemic region of Ghana.Mycobacterium ulcerans and other mycolactone-producing mycobacteria should be considered a single species.A major role for mammals in the ecology of Mycobacterium ulcerans.Common evolutionary origin for the unstable virulence plasmid pMUM found in geographically diverse strains of Mycobacterium ulceransSerological evaluation of Mycobacterium ulcerans antigens identified by comparative genomicsMycobacterium ulcerans DNA not detected in faecal samples from Buruli ulcer patients: results of a pilot study.Complete genome sequence of Staphylococcus aureus strain JKD6159, a unique Australian clone of ST93-IV community methicillin-resistant Staphylococcus aureusComplete genome sequence of Staphylococcus aureus strain JKD6008, an ST239 clone of methicillin-resistant Staphylococcus aureus with intermediate-level vancomycin resistance.Mycolactones and Mycobacterium ulcerans disease.On the origin of Mycobacterium ulcerans, the causative agent of Buruli ulcer.Complete genome sequence of the frog pathogen Mycobacterium ulcerans ecovar LiflandiiThe molecular epidemiology of the highly virulent ST93 Australian community Staphylococcus aureus strain.Insights from the complete genome sequence of Mycobacterium marinum on the evolution of Mycobacterium tuberculosis.Comparative analysis of the complete genome of an epidemic hospital sequence type 203 clone of vancomycin-resistant Enterococcus faeciumIsolates with low-level vancomycin resistance associated with persistent methicillin-resistant Staphylococcus aureus bacteremia.Hyperexpression of α-hemolysin explains enhanced virulence of sequence type 93 community-associated methicillin-resistant Staphylococcus aureus.Identification of the Mycobacterium ulcerans protein MUL_3720 as a promising target for the development of a diagnostic test for Buruli ulcer.Convergent adaptation in the dominant global hospital clone ST239 of methicillin-resistant Staphylococcus aureus.Whole genome comparisons suggest random distribution of Mycobacterium ulcerans genotypes in a Buruli ulcer endemic region of Ghana.Reductive evolution and niche adaptation inferred from the genome of Mycobacterium ulcerans, the causative agent of Buruli ulcerComplete Bypass of Restriction Systems for Major Staphylococcus aureus Lineages.
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Timothy Stinear
@ast
Timothy Stinear
@en
Timothy Stinear
@es
Timothy Stinear
@fr
Timothy Stinear
@nl
Timothy Stinear
@sl
type
label
Timothy Stinear
@ast
Timothy Stinear
@en
Timothy Stinear
@es
Timothy Stinear
@fr
Timothy Stinear
@nl
Timothy Stinear
@sl
prefLabel
Timothy Stinear
@ast
Timothy Stinear
@en
Timothy Stinear
@es
Timothy Stinear
@fr
Timothy Stinear
@nl
Timothy Stinear
@sl
P106
P1153
6602594923
P21
P31
P496
0000-0003-0150-123X
P569
2000-01-01T00:00:00Z