Seasonal and pandemic influenza H1N1 viruses induce differential expression of SOCS-1 and RIG-I genes and cytokine/chemokine production in macrophages.

Seasonal and pandemic influenza H1N1 viruses induce differential expression of SOCS-1 and RIG-I genes and cytokine/chemokine production in macrophages.

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http://www.wikidata.org/entity/Q30427498
Human Dendritic Cell Response Signatures Distinguish 1918, Pandemic, and Seasonal H1N1 Influenza Viruses.Proinflammatory chemokines are major mediators of exuberant immune response associated with Influenza A (H1N1) pdm09 virus infection.Systems-based approach to examine the cytokine responses in primary mouse lung macrophages infected with low pathogenic avian Influenza virus circulating in South East Asia.Prognoses of patients with acute-on-chronic hepatitis B liver failure are closely associated with altered SOCS1 mRNA expression and cytokine production following glucocorticoid treatment.Serum surfactant protein D (SP-D) is a prognostic marker of poor outcome in patients with A/H1N1 virus infection.Suppressor of cytokine signaling 1 (SOCS1) mitigates anterior uveitis and confers protection against ocular HSV-1 infectionSuppressor of cytokine signaling 4 (SOCS4) protects against severe cytokine storm and enhances viral clearance during influenza infection.The effects of H3N2 swine influenza virus infection on TLRs and RLRs signaling pathways in porcine alveolar macrophages.Galectins regulate the inflammatory response in airway epithelial cells exposed to microbial neuraminidase by modulating the expression of SOCS1 and RIG1Targeting CCL5 in inflammation.G45R on nonstructural protein 1 of influenza A virus contributes to virulence by increasing the expression of proinflammatory cytokines in mice.Antimicrobial peptides alter early immune response to influenza A virus infection in C57BL/6 mice.Orally Administered Salacia reticulata Extract Reduces H1N1 Influenza Clinical Symptoms in Murine Lung Tissues Putatively Due to Enhanced Natural Killer Cell Activity.MCPIP1 attenuates the innate immune response to influenza A viruse by suppressing RIG-I expression in lung epithelial cells.SOCS4 is dispensable for an efficient recall response to influenza despite being required for primary immunity.The Streptococcus pyogenes fibronectin/tenascin-binding protein PrtF.2 contributes to virulence in an influenza superinfection
P2860

Seasonal and pandemic influenza H1N1 viruses induce differential expression of SOCS-1 and RIG-I genes and cytokine/chemokine production in macrophages.

Item
http://www.wikidata.org/entity/Q30427498
description
2013 nî lūn-bûn
@nan
2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Seasonal and pandemic influenz ...... ine production in macrophages.
@ast
Seasonal and pandemic influenz ...... ine production in macrophages.
@en
type
label
Seasonal and pandemic influenz ...... ine production in macrophages.
@ast
Seasonal and pandemic influenz ...... ine production in macrophages.
@en
prefLabel
Seasonal and pandemic influenz ...... ine production in macrophages.
@ast
Seasonal and pandemic influenz ...... ine production in macrophages.
@en
P1433
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P2093
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Seasonal and pandemic influenz ...... kine production in macrophages
@en
P2093
P2860
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P31
P356
10.1016/J.CYTO.2013.01.018
P433
P478
P50
P577
P5875
P698
P921
P932