Regulated folding of tyrosinase in the endoplasmic reticulum demonstrates that misfolded full-length proteins are efficient substrates for class I processing and presentation.
about
DRiPs solidify: progress in understanding endogenous MHC class I antigen processingTyrosinase degradation is prevented when EDEM1 lacks the intrinsically disordered regionCellular stress response and innate immune signaling: integrating pathways in host defense and inflammation.Identification and characterization of human leukocyte antigen class I ligands in renal cell carcinoma cellsN-glycosylation enhances presentation of a MHC class I-restricted epitope from tyrosinaseMHC class II presentation of gp100 epitopes in melanoma cells requires the function of conventional endosomes and is influenced by melanosomes.Defective ribosomal products are the major source of antigenic peptides endogenously generated from influenza A virus neuraminidase.G-quadruplexes regulate Epstein-Barr virus-encoded nuclear antigen 1 mRNA translation.Optimization of a self antigen for presentation of multiple epitopes in cancer immunityHydrophobicity as a driver of MHC class I antigen processing.Viral modulation of antigen presentation: manipulation of cellular targets in the ER and beyond.Influence of translation efficiency of homologous viral proteins on the endogenous presentation of CD8+ T cell epitopes.Tyrosinase maturation through the mammalian secretory pathway: bringing color to life.Cellular stress and innate inflammation in organ-specific autoimmunity: lessons learned from vitiligo.The nature and extent of contributions by defective ribosome products to the HLA peptidome.Translating DRiPs: progress in understanding viral and cellular sources of MHC class I peptide ligands.Running the gauntlet: from peptide generation to antigen presentation by MHC class I.The interplay between endoplasmic reticulum stress and inflammation.A stressful microenvironment: opposing effects of the endoplasmic reticulum stress response in the suppression and enhancement of adaptive tumor immunity.Melanoma cells present high levels of HLA-A2-tyrosinase in association with instability and aberrant intracellular processing of tyrosinase.Epitope located N-glycans impair the MHC-I epitope generation and presentation.Prediction of cellular immune responses against CFTR in patients with cystic fibrosis after gene therapy.
P2860
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P2860
Regulated folding of tyrosinase in the endoplasmic reticulum demonstrates that misfolded full-length proteins are efficient substrates for class I processing and presentation.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մարտին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Regulated folding of tyrosinas ...... I processing and presentation.
@ast
Regulated folding of tyrosinas ...... I processing and presentation.
@en
type
label
Regulated folding of tyrosinas ...... I processing and presentation.
@ast
Regulated folding of tyrosinas ...... I processing and presentation.
@en
prefLabel
Regulated folding of tyrosinas ...... I processing and presentation.
@ast
Regulated folding of tyrosinas ...... I processing and presentation.
@en
P1476
Regulated folding of tyrosinas ...... I processing and presentation
@en
P2093
Valentina Robila
P304
P356
10.4049/JIMMUNOL.174.5.2544
P407
P577
2005-03-01T00:00:00Z