Endogenous gamma-aminobutyric acid can excite gonadotropin-releasing hormone neurons.
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The excitatory peptide kisspeptin restores the luteinizing hormone surge and modulates amino acid neurotransmission in the medial preoptic area of middle-aged ratsNeurobiological study of fish brains gives insights into the nature of gonadotropin-releasing hormone 1-3 neuronsFXYD1, a modulator of Na,K-ATPase activity, facilitates female sexual development by maintaining gonadotrophin-releasing hormone neuronal excitabilityRetrograde endocannabinoid signaling reduces GABAergic synaptic transmission to gonadotropin-releasing hormone neurons.Anabolic androgenic steroid abuse: multiple mechanisms of regulation of GABAergic synapses in neuroendocrine control regions of the rodent forebrainEndocannabinoids and prostaglandins both contribute to GnRH neuron-GABAergic afferent local feedback circuits.Chronic exposure to anabolic androgenic steroids alters activity and synaptic function in neuroendocrine control regions of the female mouseThe neurobiology of preovulatory and estradiol-induced gonadotropin-releasing hormone surges.Cross-Talk between Metabolism and Reproduction: The Role of POMC and SF1 NeuronsProgesterone treatment inhibits and dihydrotestosterone (DHT) treatment potentiates voltage-gated calcium currents in gonadotropin-releasing hormone (GnRH) neuronsKisspeptin increases gamma-aminobutyric acidergic and glutamatergic transmission directly to gonadotropin-releasing hormone neurons in an estradiol-dependent manner.Altered GABAA receptor-mediated synaptic transmission disrupts the firing of gonadotropin-releasing hormone neurons in male mice under conditions that mimic steroid abuseEstradiol suppresses glutamatergic transmission to gonadotropin-releasing hormone neurons in a model of negative feedback in miceNeurobiological mechanisms underlying oestradiol negative and positive feedback regulation of gonadotrophin-releasing hormone neuronesClassical estrogen receptor alpha signaling mediates negative and positive feedback on gonadotropin-releasing hormone neuron firingCritical roles for fast synaptic transmission in mediating estradiol negative and positive feedback in the neural control of ovulation.Differential regulation of gonadotropin-releasing hormone neuron activity and membrane properties by acutely applied estradiol: dependence on dose and estrogen receptor subtype.Age affects spontaneous activity and depolarizing afterpotentials in isolated gonadotropin-releasing hormone neurons.Vasoactive intestinal polypeptide can excite gonadotropin-releasing hormone neurons in a manner dependent on estradiol and gated by time of dayAcquisition of spontaneous electrical activity during embryonic development of gonadotropin-releasing hormone-3 neurons located in the terminal nerve of transgenic zebrafish (Danio rerio).Estradiol modulates Kiss1 neuronal response to ghrelinHypothalamic pathways linking energy balance and reproduction.Knockdown of GABA(A) receptor signaling in GnRH neurons has minimal effects upon fertility.Chapter 2: hypothalamic neural systems controlling the female reproductive life cycle gonadotropin-releasing hormone, glutamate, and GABA.Gene networks and the neuroendocrine regulation of pubertyThe transcriptional control of female puberty.Characterization of Kiss1 neurons using transgenic mouse modelsThe role of GABA in the regulation of GnRH neuronsGhrelin decreases firing activity of gonadotropin-releasing hormone (GnRH) neurons in an estrous cycle and endocannabinoid signaling dependent mannerChloride Accumulators NKCC1 and AE2 in Mouse GnRH Neurons: Implications for GABAA Mediated Excitation.The Changes They are A-Timed: Metabolism, Endogenous Clocks, and the Timing of Puberty.Neurosteroids and GABA-A Receptor Function.Simulated GABA synaptic input and L-type calcium channels form functional microdomains in hypothalamic gonadotropin-releasing hormone neurons.Depolarising and hyperpolarising actions of GABA(A) receptor activation on gonadotrophin-releasing hormone neurones: towards an emerging consensus.GABA is excitatory in adult vasopressinergic neuroendocrine cells.Estrogen Receptor Beta and 2-arachidonoylglycerol Mediate the Suppressive Effects of Estradiol on Frequency of Postsynaptic Currents in Gonadotropin-Releasing Hormone Neurons of Metestrous Mice: An Acute Slice Electrophysiological Study.Synaptic integration in hypothalamic gonadotropin releasing hormone (GnRH) neurons.GABAergic regulation of the HPA and HPG axes and the impact of stress on reproductive function.Living or dying in three quarter time: neonatal orchestration of hippocampal cell death pathways by androgens and excitatory GABA.Glucagon-Like Peptide-1 Excites Firing and Increases GABAergic Miniature Postsynaptic Currents (mPSCs) in Gonadotropin-Releasing Hormone (GnRH) Neurons of the Male Mice via Activation of Nitric Oxide (NO) and Suppression of Endocannabinoid Signaling
P2860
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P2860
Endogenous gamma-aminobutyric acid can excite gonadotropin-releasing hormone neurons.
description
2005 nî lūn-bûn
@nan
2005 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Endogenous gamma-aminobutyric acid can excite gonadotropin-releasing hormone neurons.
@ast
Endogenous gamma-aminobutyric acid can excite gonadotropin-releasing hormone neurons.
@en
type
label
Endogenous gamma-aminobutyric acid can excite gonadotropin-releasing hormone neurons.
@ast
Endogenous gamma-aminobutyric acid can excite gonadotropin-releasing hormone neurons.
@en
prefLabel
Endogenous gamma-aminobutyric acid can excite gonadotropin-releasing hormone neurons.
@ast
Endogenous gamma-aminobutyric acid can excite gonadotropin-releasing hormone neurons.
@en
P356
P1433
P1476
Endogenous gamma-aminobutyric acid can excite gonadotropin-releasing hormone neurons.
@en
P2093
R Anthony DeFazio
Suzanne M Moenter
P304
P356
10.1210/EN.2005-0788
P407
P577
2005-08-25T00:00:00Z