Targeted disruption of luteinizing hormone beta-subunit leads to hypogonadism, defects in gonadal steroidogenesis, and infertility - sprookjes - yvandenbroek - TriplyDB

Targeted disruption of luteinizing hormone beta-subunit leads to hypogonadism, defects in gonadal steroidogenesis, and infertility

Targeted disruption of luteinizing hormone beta-subunit leads to hypogonadism, defects in gonadal steroidogenesis, and infertility

http://www.wikidata.org/entity/Q30448413

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Genetic variants related to gap junctions and hormone secretion influence conception rates in cows Connective tissue growth factor is required for normal follicle development and ovulation Genetics of the ovarian reserve Genetic Models for the Study of Luteinizing Hormone Receptor Function Endocrine control of spermatogenesis: Role of FSH and LH/ testosterone RiboTag analysis of actively translated mRNAs in Sertoli and Leydig cells in vivo.Transcriptional regulation of steroidogenic genes: STARD1, CYP11A1 and HSD3B Regulation of male fertility by the bone-derived hormone osteocalcin Fertility in luteinizing hormone receptor-knockout mice after wild-type ovary transplantation demonstrates redundancy of extragonadal luteinizing hormone action Leptin monotherapy rescues spermatogenesis in male Akita type 1 diabetic mice Growth Arrest Specific-1 (GAS1) Is a C/EBP Target Gene That Functions in Ovulation and Corpus Luteum Formation in Mice Gonadotrope-specific deletion of Dicer results in severely suppressed gonadotropins and fertility defects.Redirecting intracellular trafficking and the secretion pattern of FSH dramatically enhances ovarian function in mice.Osteocalcin regulates murine and human fertility through a pancreas-bone-testis axis.NR5A2 regulates Lhb and Fshb transcription in gonadotrope-like cells in vitro, but is dispensable for gonadotropin synthesis and fertility in vivo.Androgen-responsive microRNAs in mouse Sertoli cells.Granulosa cell-expressed BMPR1A and BMPR1B have unique functions in regulating fertility but act redundantly to suppress ovarian tumor development.Luteinizing hormone promotes gonadal tumorigenesis in inhibin-deficient mice Essential roles of COUP-TFII in Leydig cell differentiation and male fertility.Sertoli cells control peritubular myoid cell fate and support adult Leydig cell development in the prepubertal testis The effects of type 1 diabetes on the hypothalamic, pituitary and testes axis Fetal Leydig cell origin and development.Hormones in synergy: regulation of the pituitary gonadotropin genes.Phenotyping male infertility in the mouse: how to get the most out of a 'non-performer'Fetal Leydig Cells Persist as an Androgen-Independent Subpopulation in the Postnatal Testis.Genetic analysis of zebrafish gonadotropin (FSH and LH) functions by TALEN-mediated gene disruption Targeted gene disruption in zebrafish reveals noncanonical functions of LH signaling in reproduction Posthatching development of Alligator mississippiensis ovary and testis.The forkhead transcription factor, FOXP3: a critical role in male fertility in mice Insights into the Development of the Adult Leydig Cell Lineage from Stem Leydig Cells.Male germ cell apoptosis: regulation and biology.The hormonal composition of follicular fluid and its implications for ovarian cancer pathogenesis The power of mouse genetics to study spermatogenesis.Integration mechanisms of transgenes and population fitness of GH transgenic fish.The menstrual cycle: basic biology Oocyte-somatic cell communication and microRNA function in the ovary.Sertoli cells maintain Leydig cell number and peritubular myoid cell activity in the adult mouse testis Activin regulates luteinizing hormone beta-subunit gene expression through Smad-binding and homeobox elements.Androgens, progestins, and glucocorticoids induce follicle-stimulating hormone beta-subunit gene expression at the level of the gonadotrope Defective gonadotropin-dependent ovarian folliculogenesis and granulosa cell gene expression in inhibin-deficient mice.

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P2860

Targeted disruption of luteinizing hormone beta-subunit leads to hypogonadism, defects in gonadal steroidogenesis, and infertility

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2004 nî lūn-bûn

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2004 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2004年の論文

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Targeted disruption of luteini ...... eroidogenesis, and infertility

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Targeted disruption of luteini ...... eroidogenesis, and infertility

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Targeted disruption of luteini ...... eroidogenesis, and infertility

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Targeted disruption of luteini ...... eroidogenesis, and infertility

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Proceedings of the National Academy of Sciences of the United States of America

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Targeted disruption of luteini ...... eroidogenesis, and infertility

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P2093

Martin M Matzuk

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T Rajendra Kumar

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Xiaoping Ma

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Yanlan Dong

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P2860

Mutation in the follicle-stimulating hormone receptor gene causes hereditary hypergonadotropic ovarian failure

Clinical features of primary ovarian failure caused by a point mutation in the follicle-stimulating hormone receptor gene

Pituitary hormones are not required for sexual differentiation of male mice: phenotype of the T/ebp/Nkx2.1 null mutant mice

Impairing follicle-stimulating hormone (FSH) signaling in vivo: targeted disruption of the FSH receptor leads to aberrant gametogenesis and hormonal imbalance

Follicle stimulating hormone is required for ovarian follicle maturation but not male fertility

The effect of a null mutation in the follicle-stimulating hormone receptor gene on mouse reproduction

Normal prenatal but arrested postnatal sexual development of luteinizing hormone receptor knockout (LuRKO) mice

Targeted disruption of luteinizing hormone/human chorionic gonadotropin receptor gene.

Infertility with defective spermatogenesis and hypotestosteronemia in male mice lacking the androgen receptor in Sertoli cells.

Mutations of gonadotropins and gonadotropin receptors: elucidating the physiology and pathophysiology of pituitary-gonadal function.

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