Smooth muscle-specific expression of the smooth muscle myosin heavy chain gene in transgenic mice requires 5'-flanking and first intronic DNA sequence.
about
Myocardin is a critical serum response factor cofactor in the transcriptional program regulating smooth muscle cell differentiation.Megakaryoblastic leukemia factor-1 transduces cytoskeletal signals and induces smooth muscle cell differentiation from undifferentiated embryonic stem cellsHuman SM22 alpha BAC encompasses regulatory sequences for expression in vascular and visceral smooth muscles at fetal and adult stagesIRF-2 is involved in up-regulation of nonmuscle myosin heavy chain II-A gene expression during phorbol ester-induced promyelocytic HL-60 differentiationHepatocyte nuclear factor-3 homologue 1 (HFH-1) represses transcription of smooth muscle-specific genesThymine DNA glycosylase represses myocardin-induced smooth muscle cell differentiation by competing with serum response factor for myocardin bindingAnalysis of SM22alpha-deficient mice reveals unanticipated insights into smooth muscle cell differentiation and functionPositive- and negative-acting Kruppel-like transcription factors bind a transforming growth factor beta control element required for expression of the smooth muscle cell differentiation marker SM22alpha in vivo.Molecular mechanisms of decreased smooth muscle differentiation marker expression after vascular injury.CArG elements control smooth muscle subtype-specific expression of smooth muscle myosin in vivoThe smooth muscle myosin heavy chain gene exhibits smooth muscle subtype-selective modular regulation in vivo.Smooth muscle cell plasticity: fact or fiction?Cooperative binding of KLF4, pELK-1, and HDAC2 to a G/C repressor element in the SM22α promoter mediates transcriptional silencing during SMC phenotypic switching in vivo.Smooth muscle cell phenotypic switching in atherosclerosisPlatelet-derived growth factor-BB and Ets-1 transcription factor negatively regulate transcription of multiple smooth muscle cell differentiation marker genes.FRNK, the autonomously expressed C-terminal region of focal adhesion kinase, is uniquely regulated in vascular smooth muscle: analysis of expression in transgenic mice.Assessment of contractility of purified smooth muscle cells derived from embryonic stem cells.Multiple repressor pathways contribute to phenotypic switching of vascular smooth muscle cells.Delayed goblet cell hyperplasia, acetylcholine receptor expression, and worm expulsion in SMC-specific IL-4Ralpha-deficient miceMolecular control of vascular smooth muscle cell differentiation.Characterization of vascular mural cells during zebrafish development.Activation of the smooth muscle-specific telokin gene by thyrotroph embryonic factor (TEF).Can we differentiate between airway and vascular smooth muscle?Intronic CArG box regulates cysteine-rich protein 2 expression in the adult but not in developing vasculature.VEGF-mediated fusion in the generation of uniluminal vascular spheroidsCa2+-sensing transgenic mice: postsynaptic signaling in smooth muscle.Vascular assembly in natural and engineered tissues.Regulation of smooth muscle phenotype.Smooth muscle calponin: an unconventional CArG-dependent gene that antagonizes neointimal formationBladder function in mice with inducible smooth muscle-specific deletion of the manganese superoxide dismutase gene.Control of phenotypic plasticity of smooth muscle cells by bone morphogenetic protein signaling through the myocardin-related transcription factors.Interference with PPAR gamma function in smooth muscle causes vascular dysfunction and hypertension.Differentiation of multipotent vascular stem cells contributes to vascular diseasesGerm line activation of the Tie2 and SMMHC promoters causes noncell-specific deletion of floxed allelesSmooth muscle protein 22alpha-mediated patchy deletion of Bmpr1a impairs cardiac contractility but protects against pulmonary vascular remodeling.Intracellular Ca²⁺ signalling and phenotype of vascular smooth muscle cells.Smooth muscle cell hypertrophy, proliferation, migration and apoptosis in pulmonary hypertension.Nuclear factor of activated T cells and serum response factor cooperatively regulate the activity of an alpha-actin intronic enhancer.Smooth muscle-specific genes are differentially sensitive to inhibition by Elk-1.Hypertension: beta testing
P2860
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P2860
Smooth muscle-specific expression of the smooth muscle myosin heavy chain gene in transgenic mice requires 5'-flanking and first intronic DNA sequence.
description
1998 nî lūn-bûn
@nan
1998 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Smooth muscle-specific express ...... d first intronic DNA sequence.
@ast
Smooth muscle-specific express ...... d first intronic DNA sequence.
@en
type
label
Smooth muscle-specific express ...... d first intronic DNA sequence.
@ast
Smooth muscle-specific express ...... d first intronic DNA sequence.
@en
prefLabel
Smooth muscle-specific express ...... d first intronic DNA sequence.
@ast
Smooth muscle-specific express ...... d first intronic DNA sequence.
@en
P2093
P356
P1433
P1476
Smooth muscle-specific express ...... d first intronic DNA sequence.
@en
P2093
P304
P356
10.1161/01.RES.82.8.908
P577
1998-05-01T00:00:00Z