Loss of oriented cell division does not initiate cyst formation - sprookjes - yvandenbroek - TriplyDB

Loss of oriented cell division does not initiate cyst formation

Loss of oriented cell division does not initiate cyst formation

http://www.wikidata.org/entity/Q30493527

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Olfactory receptor responding to gut microbiota-derived signals plays a role in renin secretion and blood pressure regulation Polycystic kidney disease protein fibrocystin localizes to the mitotic spindle and regulates spindle bipolarity Role of the Polycystins in Cell Migration, Polarity, and Tissue Morphogenesis Accessory proteins for heterotrimeric G-proteins in the kidney A polycystin-centric view of cyst formation and disease: the polycystins revisited Polycystin-1: a master regulator of intersecting cystic pathways The kidney and planar cell polarity.Disruption of IFT complex A causes cystic kidneys without mitotic spindle misorientation A genetic interaction network of five genes for human polycystic kidney and liver diseases defines polycystin-1 as the central determinant of cyst formation Polycystin-2 and phosphodiesterase 4C are components of a ciliary A-kinase anchoring protein complex that is disrupted in cystic kidney diseases The primary cilium: a signalling centre during vertebrate development Ciliopathies The Nek8 protein kinase, mutated in the human cystic kidney disease nephronophthisis, is both activated and degraded during ciliogenesis.The Meckel syndrome protein meckelin (TMEM67) is a key regulator of cilia function but is not required for tissue planar polarity.Vertebrate kidney tubules elongate using a planar cell polarity-dependent, rosette-based mechanism of convergent extension Molecular advances in autosomal dominant polycystic kidney disease.Planar cell polarity and the kidney.Reduced Notch signaling leads to renal cysts and papillary microadenomas.Tissue-specific regulation of the mouse Pkhd1 (ARPKD) gene promoter The cell biology of polycystic kidney disease Polycystic kidney disease: inheritance, pathophysiology, prognosis, and treatment.Downregulating hedgehog signaling reduces renal cystogenic potential of mouse models Kidney: polycystic kidney disease.Aberrant regulation of planar cell polarity in polycystic kidney disease Ciliopathies: an expanding disease spectrum.Cilium, centrosome and cell cycle regulation in polycystic kidney disease.Cell intercalation from top to bottom Kidney injury accelerates cystogenesis via pathways modulated by heme oxygenase and complement.Cystic kidney diseases: many ways to form a cyst.Cilia, Wnt signaling, and the cytoskeleton Cystic diseases of the kidney: ciliary dysfunction and cystogenic mechanisms.miR-17~92 miRNA cluster promotes kidney cyst growth in polycystic kidney disease.Heterotrimeric G protein signaling in polycystic kidney disease Primary cilia and kidney injury: current research status and future perspectives Gli-similar proteins: their mechanisms of action, physiological functions, and roles in disease.Aberrant expression of laminin-332 promotes cell proliferation and cyst growth in ARPKD.Polycystin-1 binds Par3/aPKC and controls convergent extension during renal tubular morphogenesis.Cystic kidney disease: the role of Wnt signaling.Genetics of congenital anomalies of the kidney and urinary tract.Calcium-mediated mechanisms of cystic expansion.

P2860

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P2860

Loss of oriented cell division does not initiate cyst formation

http://www.wikidata.org/entity/Q30493527

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Loss of oriented cell division does not initiate cyst formation

@ast

Loss of oriented cell division does not initiate cyst formation

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type

label

Loss of oriented cell division does not initiate cyst formation

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Loss of oriented cell division does not initiate cyst formation

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prefLabel

Loss of oriented cell division does not initiate cyst formation

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Loss of oriented cell division does not initiate cyst formation

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P356

P1433

Journal of the American Society of Nephrology

P1476

Loss of oriented cell division does not initiate cyst formation

@en

P2093

Anna Rachel Gallagher

http://www.w3.org/2001/XMLSchema#string

Saori Nishio

http://www.w3.org/2001/XMLSchema#string

Stefan Somlo

http://www.w3.org/2001/XMLSchema#string

Xin Tian

http://www.w3.org/2001/XMLSchema#string

Zhiheng Yu

http://www.w3.org/2001/XMLSchema#string

P2860

Mutations in PRKCSH cause isolated autosomal dominant polycystic liver disease.

Polaris, a protein involved in left-right axis patterning, localizes to basal bodies and cilia

Mutations in SEC63 cause autosomal dominant polycystic liver disease

Wnt9b signaling regulates planar cell polarity and kidney tubule morphogenesis

A Wnt7b-dependent pathway regulates the orientation of epithelial cell division and establishes the cortico-medullary axis of the mammalian kidney

Cystin, a novel cilia-associated protein, is disrupted in the cpk mouse model of polycystic kidney disease

Planar cell polarity signaling: from fly development to human disease

Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways

Planar cell polarity signalling couples cell division and morphogenesis during neurulation.

Polycystic kidney disease.

P304

295-302

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scholarly article

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10.1681/ASN.2009060603

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2

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21

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2009-12-03T00:00:00Z

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19959710

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2834544

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