Antagonism of peripheral inflammation reduces the severity of status epilepticus. - sprookjes - yvandenbroek - TriplyDB

Antagonism of peripheral inflammation reduces the severity of status epilepticus.

Antagonism of peripheral inflammation reduces the severity of status epilepticus.

http://www.wikidata.org/entity/Q30498265

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Intracellular and circulating neuronal antinuclear antibodies in human epilepsy P2X receptors as targets for the treatment of status epilepticus What non-neuronal mechanisms should be studied to understand epileptic seizures?Inflammatory events at blood-brain barrier in neuroinflammatory and neurodegenerative disorders: implications for clinical disease Inflammatory mechanisms contribute to the neurological manifestations of tuberous sclerosis complex.Blood-brain barrier, bulk flow, and interstitial clearance in epilepsy Efficacy of anti-inflammatory therapy in a model of acute seizures and in a population of pediatric drug resistant epileptics Serum S100B: a potential biomarker for suicidality in adolescents?Somatostatin type-2 receptor activation inhibits glutamate release and prevents status epilepticus Lessons from the laboratory: the pathophysiology, and consequences of status epilepticus.Comorbidity between epilepsy and depression: role of hippocampal interleukin-1beta.Blood-brain barrier damage, but not parenchymal white blood cells, is a hallmark of seizure activity.Hippocampal FGF-2 and BDNF overexpression attenuates epileptogenesis-associated neuroinflammation and reduces spontaneous recurrent seizures.Interleukin-1β biosynthesis inhibition reduces acute seizures and drug resistant chronic epileptic activity in mice Therapeutic effects of anti-HMGB1 monoclonal antibody on pilocarpine-induced status epilepticus in mice.Blood-brain barrier dysfunction and epilepsy: pathophysiologic role and therapeutic approaches Endothelial Von Willebrand factor promotes blood-brain barrier flexibility and provides protection from hypoxia and seizures in mice.Multimodal investigations of trans-endothelial cell trafficking under condition of disrupted blood-brain barrier integrity.Cellular injury and neuroinflammation in children with chronic intractable epilepsy.Is peripheral immunity regulated by blood-brain barrier permeability changes?Nonconvulsive seizures in subarachnoid hemorrhage link inflammation and outcome.Comorbidity between epilepsy and depression: experimental evidence for the involvement of serotonergic, glucocorticoid, and neuroinflammatory mechanisms.Ablation of cyclooxygenase-2 in forebrain neurons is neuroprotective and dampens brain inflammation after status epilepticus Administration of simvastatin after kainic acid-induced status epilepticus restrains chronic temporal lobe epilepsy.Sertraline reduces IL-1β and TNF-α mRNA expression and overcomes their rise induced by seizures in the rat hippocampus.Prevention or modification of epileptogenesis after brain insults: experimental approaches and translational research.Inhibition of the NLRP3 inflammasome provides neuroprotection in rats following amygdala kindling-induced status epilepticus.Additional antiepileptic mechanisms of levetiracetam in lithium-pilocarpine treated rats.Propofol effectively inhibits lithium-pilocarpine- induced status epilepticus in rats via downregulation of N-methyl-D-aspartate receptor 2B subunit expression The roles of vitamin B12 and vitamin D in children with intractable epilepsy.Therapeutic window for cyclooxygenase-2 related anti-inflammatory therapy after status epilepticus S100B blood levels and childhood trauma in adolescent inpatients In vitro blood-brain barrier models: current and perspective technologies Acetylcholinesterase loosens the brain's cholinergic anti-inflammatory response and promotes epileptogenesis.Blood-brain barrier dysfunction in epileptogenesis of the temporal lobe Interleukin-1 receptor (IL-1R) mediates epilepsy-induced sleep disruption Protein expression profiling of inflammatory mediators in human temporal lobe epilepsy reveals co-activation of multiple chemokines and cytokines Infiltrating macrophages are key to the development of seizures following virus infection.Immunity and inflammation in status epilepticus and its sequelae: possibilities for therapeutic application.Modulation of Immunity and the Inflammatory Response: A New Target for Treating Drug-resistant Epilepsy.

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Antagonism of peripheral inflammation reduces the severity of status epilepticus.

http://www.wikidata.org/entity/Q30498265

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Antagonism of peripheral inflammation reduces the severity of status epilepticus.

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Antagonism of peripheral inflammation reduces the severity of status epilepticus.

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Antagonism of peripheral inflammation reduces the severity of status epilepticus.

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Antagonism of peripheral inflammation reduces the severity of status epilepticus.

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Antagonism of peripheral inflammation reduces the severity of status epilepticus.

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Antagonism of peripheral inflammation reduces the severity of status epilepticus.

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J.NBD.2008.10.002

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Neurobiology of Disease

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Antagonism of peripheral inflammation reduces the severity of status epilepticus

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Ayush Batra

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Francesca Bertolini

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Giulia Betto

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Qingyuan Fan

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Vincent Fazio

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P2860

Acute induction of epileptiform discharges by pilocarpine in the in vitro isolated guinea-pig brain requires enhancement of blood-brain barrier permeability

Interleukin 1 receptor antagonist is a member of the interleukin 1 gene family: evolution of a cytokine control mechanism

Perioperative complications of blood brain barrier disruption under general anesthesia: a retrospective review.

Early changes measured by magnetic resonance imaging in cerebral blood flow, blood volume, and blood-brain barrier permeability following dexamethasone treatment in patients with brain tumors.

Interleukin-1beta contributes to the generation of experimental febrile seizures

Seizure-promoting effect of blood-brain barrier disruption.

The blood-brain barrier and epilepsy.

TGF-beta receptor-mediated albumin uptake into astrocytes is involved in neocortical epileptogenesis.

Blood-brain barrier leakage may lead to progression of temporal lobe epilepsy.

Powerful anticonvulsant action of IL-1 receptor antagonist on intracerebral injection and astrocytic overexpression in mice

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10.1016/J.NBD.2008.10.002

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2

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Tiziana Granata

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23474749

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status epilepticus

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