Purkinje cell-specific males absent on the first (mMof) gene deletion results in an ataxia-telangiectasia-like neurological phenotype and backward walking in mice.
about
The many faces of histone H3K79 methylationAtaxia-telangiectasia: future prospectsSingle-strand DNA-binding protein SSB1 facilitates TERT recruitment to telomeres and maintains telomere G-overhangsEnvironmental chemical exposures and human epigeneticsStructure and function of histone acetyltransferase MOFMOF-associated complexes ensure stem cell identity and Xist repression.The role of MOF in the ionizing radiation response is conserved in Drosophila melanogaster'Relax and Repair' to restrain aging.Arsenic Trioxide Reduces Global Histone H4 Acetylation at Lysine 16 through Direct Binding to Histone Acetyltransferase hMOF in Human Cells.Role of the Exocyst Complex Component Sec6/8 in Genomic StabilityT-cell-specific deletion of Mof blocks their differentiation and results in genomic instability in mice.Genome-wide distribution of histone H4 Lysine 16 acetylation sites and their relationship to gene expression.MOF maintains transcriptional programs regulating cellular stress response.Pluripotent Stem Cells and DNA Damage Response to Ionizing Radiations.Microglial CX3CR1 promotes adult neurogenesis by inhibiting Sirt 1/p65 signaling independent of CX3CL1Lysine acetylation: enzymes, bromodomains and links to different diseases.A multifaceted role for MOF histone modifying factor in genome maintenance.MOF phosphorylation by ATM regulates 53BP1-mediated double-strand break repair pathway choice.Alteration/deficiency in activation-3 (Ada3) plays a critical role in maintaining genomic stability.Histone H4 lysine 16 acetylated isoform synthesis opens new route to biophysical studiesHistone acetyltransferase KAT8 is essential for mouse oocyte development by regulating reactive oxygen species levels.The histone acetyltransferase MOF activates hypothalamic polysialylation to prevent diet-induced obesity in mice.MOF suppresses replication stress and contribute to resolution of stalled replication forks.Differentiation of Human Induced Pluripotent or Embryonic Stem Cells Decreases the DNA Damage Repair by Homologous Recombination.MOF influences meiotic expansion of H2AX phosphorylation and spermatogenesis in mice.
P2860
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P2860
Purkinje cell-specific males absent on the first (mMof) gene deletion results in an ataxia-telangiectasia-like neurological phenotype and backward walking in mice.
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Purkinje cell-specific males a ...... and backward walking in mice.
@ast
Purkinje cell-specific males a ...... and backward walking in mice.
@en
type
label
Purkinje cell-specific males a ...... and backward walking in mice.
@ast
Purkinje cell-specific males a ...... and backward walking in mice.
@en
prefLabel
Purkinje cell-specific males a ...... and backward walking in mice.
@ast
Purkinje cell-specific males a ...... and backward walking in mice.
@en
P2093
P2860
P356
P1476
Purkinje cell-specific males a ...... and backward walking in mice.
@en
P2093
Arun Gupta
Clayton R Hunt
Dennis K Burns
Jerry W Shay
Raj K Pandita
Rakesh Kumar
Robert Bachoo
Suraj Nannepaga
Tej K Pandita
Thomas Ludwig
P2860
P304
P356
10.1073/PNAS.1016524108
P407
P577
2011-02-14T00:00:00Z