Interleukin-1 receptor antagonist prevents murine bronchopulmonary dysplasia induced by perinatal inflammation and hyperoxia. - sprookjes - yvandenbroek - TriplyDB

Interleukin-1 receptor antagonist prevents murine bronchopulmonary dysplasia induced by perinatal inflammation and hyperoxia.

Interleukin-1 receptor antagonist prevents murine bronchopulmonary dysplasia induced by perinatal inflammation and hyperoxia.

http://www.wikidata.org/entity/Q30544045

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Phagocytosed Clofazimine Biocrystals Can Modulate Innate Immune Signaling by Inhibiting TNFα and Boosting IL-1RA Secretion Pro-inflammatory effect of a traditional Chinese medicine formula with potent anti-cancer activity in vitro impedes tumor inhibitory potential in vivo Refining anti-inflammatory therapy strategies for bronchopulmonary dysplasia.The Future of Bronchopulmonary Dysplasia: Emerging Pathophysiological Concepts and Potential New Avenues of Treatment.Protein painting reveals solvent-excluded drug targets hidden within native protein-protein interfaces.Drug therapy trials for the prevention of bronchopulmonary dysplasia: current and future targets Animal models of bronchopulmonary dysplasia. The term mouse models Epithelial β1 integrin is required for lung branching morphogenesis and alveolarization.Cumulative neonatal oxygen exposure predicts response of adult mice infected with influenza A virus.Systems approach to the study of brain damage in the very preterm newborn.Aryl hydrocarbon receptor is necessary to protect fetal human pulmonary microvascular endothelial cells against hyperoxic injury: Mechanistic roles of antioxidant enzymes and RelB Influence of prenatal hypoxia and postnatal hyperoxia on morphologic lung maturation in mice.Efficacy of Leukadherin-1 in the Prevention of Hyperoxia-Induced Lung Injury in Neonatal Rats.IL-1β and Inflammasome Activity Link Inflammation to Abnormal Fetal Airway Development.Inflammatory injury to the neonatal brain - what can we do?Recent advances in late lung development and the pathogenesis of bronchopulmonary dysplasia.Recent advances in the mechanisms of lung alveolarization and the pathogenesis of bronchopulmonary dysplasia.S-Nitrosoglutathione Attenuates Airway Hyperresponsiveness in Murine Bronchopulmonary Dysplasia.Antenatal Suppression of IL-1 Protects against Inflammation-Induced Fetal Injury and Improves Neonatal and Developmental Outcomes in Mice.The effect of prenatal maternal infection on respiratory function in mouse offspring: evidence for enhanced chemosensitivity.Endothelial Monocyte-Activating Polypeptide II Mediates Macrophage Migration in the Development of Hyperoxia-Induced Lung Disease of Prematurity.Ureaplasma Species Differentially Modulate Pro- and Anti-Inflammatory Cytokine Responses in Newborn and Adult Human Monocytes Pushing the State Toward Pro-Inflammation.Human amnion cells reverse acute and chronic pulmonary damage in experimental neonatal lung injury.The significance of IL-1β +3953C>T, IL-6 -174G>C and -596G>A, TNF-α -308G>A gene polymorphisms and 86 bp variable number tandem repeat polymorphism of IL-1RN in bronchopulmonary dysplasia in infants born before 32 weeks of gestation.Exacerbation of Ventilation-Induced Lung Injury and Inflammation in Preterm Lambs by High-Dose Nanoparticles.Recent advances in our understanding of the mechanisms of late lung development and bronchopulmonary dysplasia.Inhibition of IκBβ/NFκB signaling prevents LPS-induced IL1β expression without increasing apoptosis in the developing mouse lung.Airway Remodeling and Hyperreactivity in a Model of Bronchopulmonary Dysplasia and Their Modulation by IL-1 Receptor Antagonist.Recent Advances in Bronchopulmonary Dysplasia: Pathophysiology, Prevention, and Treatment.Inhibition of Rac1 Signaling Downregulates Inflammasome Activation and Attenuates Lung Injury in Neonatal Rats Exposed to Hyperoxia.Can We Understand the Pathobiology of Bronchopulmonary Dysplasia?Endometrial Mesenchymal Stem/Stromal Cells Modulate the Macrophage Response to Implanted Polyamide/Gelatin Composite Mesh in Immunocompromised and Immunocompetent Mice.Immunomodulation to Prevent or Treat Neonatal Sepsis: Past, Present, and Future Antenatal IL-1-dependent inflammation persists postnatally and causes retinal and sub-retinal vasculopathy in progeny FGF10 and Human Lung Disease Across the Life Spectrum

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P2860

Interleukin-1 receptor antagonist prevents murine bronchopulmonary dysplasia induced by perinatal inflammation and hyperoxia.

http://www.wikidata.org/entity/Q30544045

description

2013 nî lūn-bûn

@nan

2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի օգոստոսին հրատարակված գիտական հոդված

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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Interleukin-1 receptor antagon ...... al inflammation and hyperoxia.

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Interleukin-1 receptor antagon ...... al inflammation and hyperoxia.

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Interleukin-1 receptor antagon ...... al inflammation and hyperoxia.

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Proceedings of the National Academy of Sciences of the United States of America

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Interleukin-1 receptor antagon ...... tal inflammation and hyperoxia

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Alex Veldman

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Claudia A Nold-Petry

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Elizabeth M Skuza

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John Pedersen

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Nikeh Shariatian

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Philip J Berger

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Steven X Cho

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Thilini D Samarasinghe

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P2860

Early (< 8 days) postnatal corticosteroids for preventing chronic lung disease in preterm infants

Interleukin-1 in the pathogenesis and treatment of inflammatory diseases

IL-37 is a fundamental inhibitor of innate immunity

Transepithelial migration of neutrophils into the lung requires TREM-1

Vascular endothelial growth factor gene therapy increases survival, promotes lung angiogenesis, and prevents alveolar damage in hyperoxia-induced lung injury: evidence that angiogenesis participates in alveolarization

The new BPD: an arrest of lung development.

Chorioamnionitis stimulates angiogenesis in saccular stage fetal lungs via CC chemokines

Deficits in lung alveolarization and function after systemic maternal inflammation and neonatal hyperoxia exposure.

Cytokines associated with bronchopulmonary dysplasia or death in extremely low birth weight infants.

Inhibitors of inflammation and endogenous surfactant pool size as modulators of lung injury with initiation of ventilation in preterm sheep.

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10.1073/PNAS.1306859110

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Niamh E. Mangan

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2013-08-14T00:00:00Z

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255955520

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23946428

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bronchopulmonary dysplasia

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3761642

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