Impaired striatal D2 receptor function leads to enhanced GABA transmission in a mouse model of DYT1 dystonia.
about
Dopaminergic modulation of striatal neurons, circuits, and assembliesDtorsin, the Drosophila ortholog of the early-onset dystonia TOR1A (DYT1), plays a novel role in dopamine metabolismDevelopmental profile of the aberrant dopamine D2 receptor response in striatal cholinergic interneurons in DYT1 dystoniaOpposite effects of stimulant and antipsychotic drugs on striatal fast-spiking interneuronsMutant human torsinA, responsible for early-onset dystonia, dominantly suppresses GTPCH expression, dopamine levels and locomotion in Drosophila melanogasterTor1a+/- mice develop dystonia-like movements via a striatal dopaminergic dysregulation triggered by peripheral nerve injury.Convergent evidence for abnormal striatal synaptic plasticity in dystonia.Striatal dopamine D1-like receptor binding is unchanged in primary focal dystonia.Modulation of the basal ganglia dopaminergic system in a transgenic mouse exhibiting dystonia-like features.Decreased striatal dopamine receptor binding in primary focal dystonia: a D2 or D3 defect?Untethering the nuclear envelope and cytoskeleton: biologically distinct dystonias arising from a common cellular dysfunctionMinimal Change in the cytoplasmic calcium dynamics in striatal GABAergic neurons of a DYT1 dystonia knock-in mouse model.Deep-Brain Stimulation for Basal Ganglia Disorders.Multi-neuronal recordings in the Basal Ganglia in normal and dystonic rats.The DYT1 carrier state increases energy demand in the olivocerebellar network.Alternative approaches to modeling hereditary dystonias.Mouse models of neurodevelopmental disease of the basal ganglia and associated circuits.Modulation of striatal projection systems by dopamine.Convergent mechanisms in etiologically-diverse dystoniasTorsinA participates in endoplasmic reticulum-associated degradationMutations in GNAL cause primary torsion dystonia.Deep Brain Stimulation for Movement Disorders of Basal Ganglia Origin: Restoring Function or Functionality?Engineering animal models of dystoniaMolecular pathways in dystonia.Milestones in dystonia.Emerging common molecular pathways for primary dystonia.Cholinergic dysfunction alters synaptic integration between thalamostriatal and corticostriatal inputs in DYT1 dystoniaDopamine D2 receptor dysfunction is rescued by adenosine A2A receptor antagonism in a model of DYT1 dystonia.Impairment of bidirectional synaptic plasticity in the striatum of a mouse model of DYT1 dystonia: role of endogenous acetylcholine.Anticholinergic drugs rescue synaptic plasticity in DYT1 dystonia: role of M1 muscarinic receptors.Optogenetic stimulation reveals distinct modulatory properties of thalamostriatal vs corticostriatal glutamatergic inputs to fast-spiking interneurons.Miniature release events of glutamate from hippocampal neurons are influenced by the dystonia-associated protein torsinA.Basal ganglia mechanisms in action selection, plasticity, and dystonia.
P2860
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P2860
Impaired striatal D2 receptor function leads to enhanced GABA transmission in a mouse model of DYT1 dystonia.
description
2009 nî lūn-bûn
@nan
2009 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Impaired striatal D2 receptor ...... mouse model of DYT1 dystonia.
@ast
Impaired striatal D2 receptor ...... mouse model of DYT1 dystonia.
@en
type
label
Impaired striatal D2 receptor ...... mouse model of DYT1 dystonia.
@ast
Impaired striatal D2 receptor ...... mouse model of DYT1 dystonia.
@en
prefLabel
Impaired striatal D2 receptor ...... mouse model of DYT1 dystonia.
@ast
Impaired striatal D2 receptor ...... mouse model of DYT1 dystonia.
@en
P2093
P2860
P50
P1476
Impaired striatal D2 receptor ...... a mouse model of DYT1 dystonia
@en
P2093
Antonio Pisani
Dario Cuomo
Giorgio Bernardi
Nutan Sharma
P2860
P304
P356
10.1016/J.NBD.2009.01.001
P50
P577
2009-01-13T00:00:00Z