Upward synaptic scaling is dependent on neurotransmission rather than spiking.
about
Homeostatic role of heterosynaptic plasticity: models and experimentsClosed-loop and activity-guided optogenetic controlCultured Cortical Neurons Can Perform Blind Source Separation According to the Free-Energy PrincipleOptogenetic feedback control of neural activity.One-photon and two-photon stimulation of neurons in a microfluidic culture system.Spontaneous Release Regulates Synaptic Scaling in the Embryonic Spinal Network In Vivo.β-Amyloid triggers aberrant over-scaling of homeostatic synaptic plasticity.Selective molecular impairment of spontaneous neurotransmission modulates synaptic efficacyAll for One But Not One for All: Excitatory Synaptic Scaling and Intrinsic Excitability Are Coregulated by CaMKIV, Whereas Inhibitory Synaptic Scaling Is Under Independent Control.Loss of Doc2-Dependent Spontaneous Neurotransmission Augments Glutamatergic Synaptic Strength.Integrating Hebbian and homeostatic plasticity: the current state of the field and future research directions.Multichannel activity propagation across an engineered axon network.Glutamatergic synapses are structurally and biochemically complex because of multiple plasticity processes: long-term potentiation, long-term depression, short-term potentiation and scaling.Linking Neuromodulated Spike-Timing Dependent Plasticity with the Free-Energy Principle.GluA4 subunit of AMPA receptors mediates the early synaptic response to altered network activity in the developing hippocampus.Synaptic up-scaling preserves motor circuit output after chronic, natural inactivity.Deprivation-Induced Homeostatic Spine Scaling In Vivo Is Localized to Dendritic Branches that Have Undergone Recent Spine Loss.Closed Loop Experiment Manager (CLEM)-An Open and Inexpensive Solution for Multichannel Electrophysiological Recordings and Closed Loop Experiments.Muscle nicotinic acetylcholine receptors may mediate trans-synaptic signaling at the mouse neuromuscular junction.Random neuronal ensembles can inherently do context dependent coarse conjunctive encoding of input stimulus without any specific training.Overexpression of cypin alters dendrite morphology, single neuron activity, and network properties via distinct mechanisms.Loss and recovery of functional connectivity in cultured cortical networks exposed to hypoxia.Spontaneous neurotransmission: A form of neural communication comes of age.Regulation of synaptic scaling by action potential-independent miniature neurotransmission.
P2860
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P2860
Upward synaptic scaling is dependent on neurotransmission rather than spiking.
description
2015 nî lūn-bûn
@nan
2015 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի մարտին հրատարակված գիտական հոդված
@hy
2015年の論文
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2015年論文
@yue
2015年論文
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2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Upward synaptic scaling is dependent on neurotransmission rather than spiking.
@ast
Upward synaptic scaling is dependent on neurotransmission rather than spiking.
@en
type
label
Upward synaptic scaling is dependent on neurotransmission rather than spiking.
@ast
Upward synaptic scaling is dependent on neurotransmission rather than spiking.
@en
prefLabel
Upward synaptic scaling is dependent on neurotransmission rather than spiking.
@ast
Upward synaptic scaling is dependent on neurotransmission rather than spiking.
@en
P2860
P356
P1476
Upward synaptic scaling is dependent on neurotransmission rather than spiking.
@en
P2093
Jonathan P Newman
Steve M Potter
P2860
P2888
P356
10.1038/NCOMMS7339
P407
P577
2015-03-09T00:00:00Z
P5875
P6179
1007081176