about
White matter hyperintensities, cognitive impairment and dementia: an update.Study of Enhanced Depth Imaging Optical Coherence Tomography in Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and LeukoencephalopathyThe pathobiology of vascular malformations: insights from human and model organism genetics.Recognizing CADASIL: a Secondary Cause of Migraine with Aura.The defining pathology of the new clinical and histopathologic entity ACTA2-related cerebrovascular disease.Consensus statement for diagnosis of subcortical small vessel disease.Perturbations of the cerebrovascular matrisome: A convergent mechanism in small vessel disease of the brain?Clinical-pathologic correlations in vascular cognitive impairment and dementiaRole of physical exercise in Alzheimer's disease.The comparisons of phenotype and genotype between CADASIL and CADASIL-like patients and population-specific evaluation of CADASIL scale in China.Pathogenesis and treatment of vascular cognitive impairment.CADASIL: Treatment and Management Options.CADASIL mutant NOTCH3(R90C) decreases the viability of HS683 oligodendrocytes via apoptosis.CADASIL Presenting as Acute Bilateral Multiple Subcortical Infarcts without a Characteristic Temporal Pole or Any External Capsule Lesions.Neoplastic lesions in CADASIL syndrome: report of an autopsied Japanese case.Loss of HtrA1-induced attenuation of TGF-β signaling in fibroblasts might not be the main mechanism of CARASIL pathogenesis.Genotypic and phenotypic spectrum of CADASIL in Japan: the experience at a referral center in Kumamoto University from 1997 to 2014.CADASIL and multiple sclerosis: A case report of prolonged misdiagnosis.Histopathologic Analysis of Cerebral Autosomal Recessive Arteriopathy with Subcortical Infarcts and Leukoencephalopathy (CARASIL): A Report of a New Genetically Confirmed Case and Comparison to 2 Previous Cases.Differences in proliferation rate between CADASIL and control vascular smooth muscle cells are related to increased TGFβ expression.A Next-Generation Sequencing of the NOTCH3 and HTRA1 Genes in CADASIL Patients.Connective tissue growth factor (CTGF) in age-related vascular pathologies.Proteomics analysis identifies new markers associated with capillary cerebral amyloid angiopathy in Alzheimer's disease.
P2860
Q30893715-4A5866C6-FDAB-4B3E-9CE7-0972408ACB22Q33646649-0BD2B62F-EA9E-4F57-8C18-3C7EBD2A52ABQ34544980-751813FE-696D-401A-93E1-BADEB7B556E4Q36303794-726FB7C5-5B2D-4941-90D9-7C711D82756FQ36348596-BE630FB2-3CCB-49EF-9CE4-B463C5B0A3AEQ36593206-48A73627-0C81-4FA2-94D4-AD1586546636Q36593214-841EB2AA-4E94-4591-9234-D4C1B0BED735Q36627110-41B9DDB4-0FAC-4421-BE9C-EDF27E281E4CQ36741200-95BC7E4C-07A2-4D97-8286-E07D718C96BCQ36922449-C174769E-7ED9-40A7-84E0-AB5A0AAB1A34Q38297016-EF7986F6-488C-4F35-AC03-0BB865192662Q38663098-E5738924-8C7C-4C5A-A736-EDE44E8752F9Q38672393-C7601DB9-2314-4C4C-8857-61F1A20B9EE5Q41634274-930A5967-0FC7-459E-8940-B14F202BB20AQ42249308-98ED0BC5-56C6-4E8F-BC00-FE6DE5F3A998Q43228018-71401703-9129-4CF1-8407-AA516FE5B3BAQ48173961-2490635D-080F-46EA-9FB2-8B02A85E1A4EQ48486008-D609798C-0722-4F0B-941C-24FAE9CAB6ABQ51499978-9E33882E-BCD5-49BF-9080-C691FA4254C5Q51740449-0497F7DE-3180-4FB4-BFF9-A2F22CF865F0Q53248522-F1051C8F-486D-42C0-B27A-728663405B83Q55024983-AEA1D675-A2E9-4D54-8ED0-0AE744E8E9F6Q55266481-590A5BD5-6762-4151-8624-5D75E7E0718F
P2860
description
2014 nî lūn-bûn
@nan
2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
CADASIL and CARASIL.
@ast
CADASIL and CARASIL.
@en
type
label
CADASIL and CARASIL.
@ast
CADASIL and CARASIL.
@en
prefLabel
CADASIL and CARASIL.
@ast
CADASIL and CARASIL.
@en
P2093
P2860
P50
P356
P1433
P1476
CADASIL and CARASIL.
@en
P2093
Hannu Kalimo
Liisa Myllykangas
Maija Siitonen
Matti Viitanen
Petra Pasanen
Saara Tikka
Toshio Fukutake
P2860
P304
P356
10.1111/BPA.12181
P407
P577
2014-09-01T00:00:00Z