Direct activation of RhoA by reactive oxygen species requires a redox-sensitive motif.
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The nuclear guanine nucleotide exchange factors Ect2 and Net1 regulate RhoB-mediated cell death after DNA damageCerebral Small Vessel Disease: Targeting Oxidative Stress as a Novel Therapeutic Strategy?Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosisRho GTPases, oxidation, and cell redox controlSmall G proteins in the cardiovascular system: physiological and pathological aspectsDiabetic complications in pregnancy: is resveratrol a solution?Mouse Melanoma Cell Migration is Dependent on Production of Reactive Oxygen Species under Normoxia ConditionRegulation of signal transduction by reactive oxygen species in the cardiovascular systemMolecular and Cellular Mechanisms of Cardiovascular Disorders in DiabetesReactive oxygen species in inflammation and tissue injuryEchovirus 30 induced neuronal cell death through TRIO-RhoA signaling activationROCK1 deficiency enhances protective effects of antioxidants against apoptosis and cell detachmentDissecting the Mechanisms of Doxorubicin and Oxidative Stress-Induced Cytotoxicity: The Involvement of Actin Cytoskeleton and ROCK1Targeted subendothelial matrix oxidation by myeloperoxidase triggers myosin II-dependent de-adhesion and alters signaling in endothelial cells.Poldip2 controls vascular smooth muscle cell migration by regulating focal adhesion turnover and force polarization.RhoA mediates defective stem cell function and heterotopic ossification in dystrophic muscle of mice.Reactive oxygen species are involved in regulating alpha1-adrenoceptor-activated vascular smooth muscle contraction.8-Oxoguanine DNA glycosylase-1-mediated DNA repair is associated with Rho GTPase activation and α-smooth muscle actin polymerization.The role of redox changes in oxygen sensing.The role of mechanical force and ROS in integrin-dependent signalsThe role of reactive oxygen species in microvascular remodeling.Activation of PKN mediates survival of cardiac myocytes in the heart during ischemia/reperfusion.Genetic interference with peroxisome proliferator-activated receptor γ in smooth muscle enhances myogenic tone in the cerebrovasculature via A Rho kinase-dependent mechanism.Hyperoxia increases the elastic modulus of alveolar epithelial cells through Rho kinase.The Interplay between ROS and Ras GTPases: Physiological and Pathological Implications.Proteomic profiling of the retinas in a neonatal rat model of oxygen-induced retinopathy with a reproducible ion-current-based MS1 approach.Collapsin response mediator protein-2 phosphorylation promotes the reversible retraction of oligodendrocyte processes in response to non-lethal oxidative stress.Anti-inflammatory activity of Barleria lupulina: Identification of active compounds that activate the Nrf2 cell defense pathway, organize cortical actin, reduce stress fibers, and improve cell junctions in microvascular endothelial cells.RhoA and Rac1 GTPases Differentially Regulate Agonist-Receptor Mediated Reactive Oxygen Species Generation in PlateletsRhoA Proteolysis Regulates the Actin Cytoskeleton in Response to Oxidative Stress.The role of Nox-mediated oxidation in the regulation of cytoskeletal dynamicsRedox regulation of Rac1 by thiol oxidation.Redox regulation of Ras and Rho GTPases: mechanism and function.Effects of angiotensin II on the cerebral circulation: role of oxidative stress.Sustained adenosine exposure causes lung endothelial barrier dysfunction via nucleoside transporter-mediated signaling.Regulation of Ras proteins by reactive nitrogen species.CD98hc (SLC3A2) regulation of skin homeostasis wanes with ageCentral Rho kinase inhibition restores baroreflex sensitivity and angiotensin II type 1 receptor protein imbalance in conscious rabbits with chronic heart failure.Cucurbitacin I inhibits Rac1 activation in breast cancer cells by a reactive oxygen species-mediated mechanism and independently of Janus tyrosine kinase 2 and P-Rex1.Tumor-suppressive maspin functions as a reactive oxygen species scavenger: importance of cysteine residues.
P2860
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P2860
Direct activation of RhoA by reactive oxygen species requires a redox-sensitive motif.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Direct activation of RhoA by reactive oxygen species requires a redox-sensitive motif.
@ast
Direct activation of RhoA by reactive oxygen species requires a redox-sensitive motif.
@en
type
label
Direct activation of RhoA by reactive oxygen species requires a redox-sensitive motif.
@ast
Direct activation of RhoA by reactive oxygen species requires a redox-sensitive motif.
@en
prefLabel
Direct activation of RhoA by reactive oxygen species requires a redox-sensitive motif.
@ast
Direct activation of RhoA by reactive oxygen species requires a redox-sensitive motif.
@en
P2093
P2860
P1433
P1476
Direct activation of RhoA by reactive oxygen species requires a redox-sensitive motif.
@en
P2093
Amir Aghajanian
Erika S Wittchen
Keith Burridge
Sharon L Campbell
P2860
P356
10.1371/JOURNAL.PONE.0008045
P407
P577
2009-11-26T00:00:00Z