Generation of a prostate epithelial cell-specific Cre transgenic mouse model for tissue-specific gene ablation.
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SOX9 elevation in the prostate promotes proliferation and cooperates with PTEN loss to drive tumor formationLoss of Nkx3.1 leads to the activation of discrete downstream target genes during prostate tumorigenesismTOR complex 2 is required for the development of prostate cancer induced by Pten loss in miceModulation of prostate cancer genetic risk by omega-3 and omega-6 fatty acidsCrucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesisPten dose dictates cancer progression in the prostateStem cells in genetically-engineered mouse models of prostate cancerTranslating insights of AR signaling from mouse modelsGenetically engineered mouse models of prostate cancerTbx18 Regulates the Differentiation of Periductal Smooth Muscle Stroma and the Maintenance of Epithelial Integrity in the ProstateBrca2 and Trp53 deficiency cooperate in the progression of mouse prostate tumourigenesisMYC cooperates with AKT in prostate tumorigenesis and alters sensitivity to mTOR inhibitorsIncreased prostate cell proliferation and loss of cell differentiation in mice lacking prostate epithelial androgen receptorLoss of survivin in the prostate epithelium impedes carcinogenesis in a mouse model of prostate adenocarcinomaN-Myc Induces an EZH2-Mediated Transcriptional Program Driving Neuroendocrine Prostate CancerActivation of Akt signaling in prostate induces a TGFβ-mediated restraint on cancer progression and metastasis.Prostate cancer induced by loss of Apc is restrained by TGFβ signalingDifferential requirement for focal adhesion kinase signaling in cancer progression in the transgenic adenocarcinoma of mouse prostate modelThe dietary isothiocyanate sulforaphane modulates gene expression and alternative gene splicing in a PTEN null preclinical murine model of prostate cancer.Interactions between cells with distinct mutations in c-MYC and Pten in prostate cancer.The pace of prostatic intraepithelial neoplasia development is determined by the timing of Pten tumor suppressor gene excisionDisruption of PPARgamma signaling results in mouse prostatic intraepithelial neoplasia involving active autophagy.Exposure to maternal obesogenic diet worsens some but not all pre-cancer phenotypes in a murine genetic model of prostate cancer.Zbtb7a suppresses prostate cancer through repression of a Sox9-dependent pathway for cellular senescence bypass and tumor invasion.ETS Related Gene mediated Androgen Receptor Aggregation and Endoplasmic Reticulum Stress in Prostate Cancer Development.Gene targeting to the stroma of the prostate and bone.Dystroglycan is not required for maintenance of the luminal epithelial basement membrane or cell polarity in the mouse prostate.TPL2/COT/MAP3K8 (TPL2) activation promotes androgen depletion-independent (ADI) prostate cancer growth.Androgen receptor (AR) physiological roles in male and female reproductive systems: lessons learned from AR-knockout mice lacking AR in selective cellsIntraosseous injection of RM1 murine prostate cancer cells promotes rapid osteolysis and periosteal bone deposition.In situ vaccination combined with androgen ablation and regulatory T-cell depletion reduces castration-resistant tumor burden in prostate-specific pten knockout mice.A mouse model of heterogeneous, c-MYC-initiated prostate cancer with loss of Pten and p53.Mouse models of prostate cancer: picking the best model for the question.Mouse prostate cancer cell lines established from primary and postcastration recurrent tumors.piggyBac transposon somatic mutagenesis with an activated reporter and tracker (PB-SMART) for genetic screens in mice.Molecular genetics of prostate cancer: new prospects for old challengesSyndecan-1-dependent suppression of PDK1/Akt/bad signaling by docosahexaenoic acid induces apoptosis in prostate cancerThe phosphatidylinositol 3-kinase (PI3K) isoform dependence of tumor formation is determined by the genetic mode of PI3K pathway activation rather than by tissue type.Interleukin-17 Induces Expression of Chemokines and Cytokines in Prostatic Epithelial Cells but Does Not Stimulate Cell Growth In Vitro.Pten deletion leads to the expansion of a prostatic stem/progenitor cell subpopulation and tumor initiation
P2860
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P2860
Generation of a prostate epithelial cell-specific Cre transgenic mouse model for tissue-specific gene ablation.
description
2001 nî lūn-bûn
@nan
2001 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի մարտին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Generation of a prostate epith ...... tissue-specific gene ablation.
@ast
Generation of a prostate epith ...... tissue-specific gene ablation.
@en
type
label
Generation of a prostate epith ...... tissue-specific gene ablation.
@ast
Generation of a prostate epith ...... tissue-specific gene ablation.
@en
prefLabel
Generation of a prostate epith ...... tissue-specific gene ablation.
@ast
Generation of a prostate epith ...... tissue-specific gene ablation.
@en
P2093
P1476
Generation of a prostate epith ...... tissue-specific gene ablation.
@en
P2093
Matusik RJ
Roy-Burman P
Sangiorgi FO
P356
10.1016/S0925-4773(00)00551-7
P577
2001-03-01T00:00:00Z