Changes in expression and distribution of claudin 2, 5 and 8 lead to discontinuous tight junctions and barrier dysfunction in active Crohn's disease.
about
Central role of the gut epithelial barrier in the pathogenesis of chronic intestinal inflammation: lessons learned from animal models and human geneticsThe junctional proteins cingulin and paracingulin modulate the expression of tight junction protein genes through GATA-4Notch-1 signaling regulates intestinal epithelial barrier function, through interaction with CD4+ T cells, in mice and humansTight junctions in inflammatory bowel diseases and inflammatory bowel disease associated colorectal cancerInfection Strategies of Intestinal Parasite Pathogens and Host Cell ResponsesProtein tyrosine phosphatase non-receptor type 2 and inflammatory bowel diseaseBreaking down the barriers: the gut microbiome, intestinal permeability and stress-related psychiatric disordersIntestinal Microbiota and the Innate Immune System - A Crosstalk in Crohn's Disease PathogenesisMitochondrial dysfunction in inflammatory bowel diseaseBacterial translocation - impact on the adipocyte compartmentIntestinal permeability regulation by tight junction: implication on inflammatory bowel diseasesMolecular aspects of tight junction barrier functionClaudins in intestines: Distribution and functional significance in health and diseasesInflammatory pathways of importance for management of inflammatory bowel diseaseIntestinal permeability--a new target for disease prevention and therapyIntestinal epithelium in inflammatory bowel diseaseIntestinal barrier in inflammatory bowel diseaseIndoleamine 2,3 dioxygenase in intestinal diseaseK+ Channel Inhibition Differentially Regulates Migration of Intestinal Epithelial Cells in Inflamed vs. Non-Inflamed Conditions in a PI3K/Akt-Mediated MannerPeriodic Exposure of Keratinocytes to Cold Physical Plasma: An In Vitro Model for Redox-Related Diseases of the Skin.ClC-2 regulation of intestinal barrier function: Translation of basic science to therapeutic targetIntestinal Permeability in Inflammatory Bowel Disease: Pathogenesis, Clinical Evaluation, and Therapy of Leaky GutIntestinal barrier function: molecular regulation and disease pathogenesisTranscriptional regulators of claudins in epithelial tight junctionsSystems Proteomics View of the Endogenous Human Claudin Protein FamilyThe intestinal barrier as an emerging target in the toxicological assessment of mycotoxinsPancreas-specific deletion of Prox1 affects development and disrupts homeostasis of the exocrine pancreasDynamic distribution of claudin proteins in pancreatic epithelia undergoing morphogenesis or neoplastic transformationOverexpression of Ste20-related proline/alanine-rich kinase exacerbates experimental colitis in miceClaudin-2 Expression Levels in Ulcerative Colitis: Development and Validation of an In-Situ Hybridisation Assay for Therapeutic StudiesMetformin Improves Ileal Epithelial Barrier Function in Interleukin-10 Deficient MiceAltered distribution of tight junction proteins after intestinal ischaemia/reperfusion injury in ratsIncrease in the tight junction protein claudin-1 in intestinal inflammationDissociation and dispersion of claudin-3 from the tight junction could be one of the most sensitive indicators of reflux esophagitis in a rat model of the diseaseFunctional analysis of VopF activity required for colonization in Vibrio cholerae.Gut microbiota, tight junction protein expression, intestinal resistance, bacterial translocation and mortality following cholestasis depend on the genetic background of the host.SAMP1/YitFc mouse strain: a spontaneous model of Crohn's disease-like ileitis.Probiotic bacteria regulate intestinal epithelial permeability in experimental ileitis by a TNF-dependent mechanismTricellulin forms a barrier to macromolecules in tricellular tight junctions without affecting ion permeability.Local barrier dysfunction identified by confocal laser endomicroscopy predicts relapse in inflammatory bowel disease
P2860
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P2860
Changes in expression and distribution of claudin 2, 5 and 8 lead to discontinuous tight junctions and barrier dysfunction in active Crohn's disease.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2006年の論文
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2006年学术文章
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2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
name
Changes in expression and dist ...... ion in active Crohn's disease.
@ast
Changes in expression and dist ...... ion in active Crohn's disease.
@en
type
label
Changes in expression and dist ...... ion in active Crohn's disease.
@ast
Changes in expression and dist ...... ion in active Crohn's disease.
@en
prefLabel
Changes in expression and dist ...... ion in active Crohn's disease.
@ast
Changes in expression and dist ...... ion in active Crohn's disease.
@en
P2093
P2860
P356
P1433
P1476
Changes in expression and dist ...... ion in active Crohn's disease.
@en
P2093
Kroesen AJ
Mankertz J
Schulzke JD
Wahnschaffe U
P2860
P356
10.1136/GUT.2006.094375
P407
P577
2006-07-05T00:00:00Z