Sp1 expression is disrupted in schizophrenia; a possible mechanism for the abnormal expression of mitochondrial complex I genes, NDUFV1 and NDUFV2
about
Gene environment interaction in periphery and brain converge to modulate behavioral outcomes: Insights from the SP1 transient early in life interference rat modelAltered functional protein networks in the prefrontal cortex and amygdala of victims of suicideTranscription factor SP4 is a susceptibility gene for bipolar disorderUncovering molecular biomarkers that correlate cognitive decline with the changes of hippocampus' gene expression profiles in Alzheimer's disease.Socially responsive effects of brain oxidative metabolism on aggression.Early-life stress links 5-hydroxymethylcytosine to anxiety-related behaviorsNeuroanatomical pattern of mitochondrial complex I pathology varies between schizophrenia, bipolar disorder and major depression.Expression profiles of mitochondrial genes in the frontal cortex and the caudate nucleus of developing humans and mice selectively bred for high and low fearCommon promoter variants of the NDUFV2 gene do not confer susceptibility to schizophrenia in Han Chinese.Transcriptome alterations of mitochondrial and coagulation function in schizophrenia by cortical sequencing analysis.The many roads to mitochondrial dysfunction in neuroimmune and neuropsychiatric disordersThe transcription factor SP4 is reduced in postmortem cerebellum of bipolar disorder subjects: control by depolarization and lithium.Do nuclear-encoded core subunits of mitochondrial complex I confer genetic susceptibility to schizophrenia in Han Chinese populations?Convergent functional genomics of schizophrenia: from comprehensive understanding to genetic risk prediction.Association study of sepiapterin reductase gene promoter polymorphisms with schizophrenia in a Han Chinese populationEpigenetic profiling of human brain differential DNA methylation networks in schizophrenia.The oncoprotein HBXIP activates transcriptional coregulatory protein LMO4 via Sp1 to promote proliferation of breast cancer cells.Platelets: A possible glance into brain biological processes in schizophrenia.Regulation of mitochondrial respiratory chain biogenesis by estrogens/estrogen receptors and physiological, pathological and pharmacological implications.Identification of rare variants in KCTD13 at the schizophrenia risk locus 16p11.2.Dexamethasone in the presence of desipramine enhances MAPK/ERK1/2 signaling possibly via its interference with β-arrestin.An emerging role for mitochondrial dynamics in schizophrenia.Antipsychotic drugs attenuate aberrant DNA methylation of DTNBP1 (dysbindin) promoter in saliva and post-mortem brain of patients with schizophrenia and Psychotic bipolar disorder.Mitochondrial Oxidative Phosphorylation System (OXPHOS) Deficits in Schizophrenia: Possible Interactions with Cellular Processes.Gene expression study of mitochondrial complex I in schizophrenia and paranoid personality disorder.
P2860
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P2860
Sp1 expression is disrupted in schizophrenia; a possible mechanism for the abnormal expression of mitochondrial complex I genes, NDUFV1 and NDUFV2
description
2007 nî lūn-bûn
@nan
2007 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Sp1 expression is disrupted in ...... lex I genes, NDUFV1 and NDUFV2
@ast
Sp1 expression is disrupted in ...... lex I genes, NDUFV1 and NDUFV2
@en
type
label
Sp1 expression is disrupted in ...... lex I genes, NDUFV1 and NDUFV2
@ast
Sp1 expression is disrupted in ...... lex I genes, NDUFV1 and NDUFV2
@en
prefLabel
Sp1 expression is disrupted in ...... lex I genes, NDUFV1 and NDUFV2
@ast
Sp1 expression is disrupted in ...... lex I genes, NDUFV1 and NDUFV2
@en
P2860
P1433
P1476
Sp1 expression is disrupted in ...... lex I genes, NDUFV1 and NDUFV2
@en
P2093
Dorit Ben-Shachar
Rachel Karry
P2860
P356
10.1371/JOURNAL.PONE.0000817
P407
P577
2007-09-05T00:00:00Z