DR3 regulates negative selection during thymocyte development
about
The intriguing biology of the tumour necrosis factor/tumour necrosis factor receptor superfamily: players, rules and the gamesCytokine and anti-cytokine therapies in prevention or treatment of fibrosis in IBDMembrane trafficking of death receptors: implications on signallingHistorical perspectives on tumor necrosis factor and its superfamily: 25 years later, a golden journeyTL1A-induced NF-kappaB activation and c-IAP2 production prevent DR3-mediated apoptosis in TF-1 cellsOn death receptor 3 and its ligands…Ex vivo whole-embryo culture of caspase-8-deficient embryos normalize their aberrant phenotypes in the developing neural tube and heartThe TNF-like protein 1A-death receptor 3 pathway promotes macrophage foam cell formation in vitro.Age-dependent maintenance of motor control and corticostriatal innervation by death receptor 3.Naive and activated T cells display differential responsiveness to TL1A that affects Th17 generation, maintenance, and proliferation.Role of SODD in regulation of tumor necrosis factor responses.Apparently normal tumor necrosis factor receptor 1 signaling in the absence of the silencer of death domains.The TNF-family cytokine TL1A drives IL-13-dependent small intestinal inflammation.An essential role for death receptor 3 in experimental autoimmune uveoretinitis.The role of apoptosis in the development and function of T lymphocytes.Normal thymocyte negative selection in TRAIL-deficient mice.Essential role of TNF receptor superfamily 25 (TNFRSF25) in the development of allergic lung inflammation.TL1A both promotes and protects from renal inflammation and injury.The TNF-family receptor DR3 is essential for diverse T cell-mediated inflammatory diseasesThe role of TL1A and DR3 in autoimmune and inflammatory diseases.TL1A and DR3, a TNF family ligand-receptor pair that promotes lymphocyte costimulation, mucosal hyperplasia, and autoimmune inflammation.Death Receptor 3 Promotes Chemokine-Directed Leukocyte Recruitment in Acute Resolving Inflammation and Is Essential for Pathological Development of Mesothelial Fibrosis in Chronic Disease.CCL3 and MMP-9 are induced by TL1A during death receptor 3 (TNFRSF25)-dependent osteoclast function and systemic bone loss.T cell expression of IL-18R and DR3 is essential for non-cognate stimulation of Th1 cells and optimal clearance of intracellular bacteria.Apoptotic and nonapoptotic caspase functions in animal development.Immunobiology of TNFSF15 and TNFRSF25.Death Receptor 3 (TNFRSF25) Increases Mineral Apposition by Osteoblasts and Region Specific New Bone Formation in the Axial Skeleton of Male DBA/1 Mice.Tumor Necrosis Factor-like Cytokine TL1A and Its Receptors DR3 and DcR3: Important New Factors in Mucosal Homeostasis and Inflammation.DR3 signaling modulates the function of Foxp3+ regulatory T cells and the severity of acute graft-versus-host disease.Review: Novel Insights Into Tumor Necrosis Factor Receptor, Death Receptor 3, and Progranulin Pathways in Arthritis and Bone Remodeling.Multifaceted death receptor 3 signaling-promoting survival and triggering death.Characterization of death receptor 3-dependent aortic changes during inflammatory arthritisThe TNF-family ligand TL1A and its receptor DR3 promote T cell-mediated allergic immunopathology by enhancing differentiation and pathogenicity of IL-9-producing T cells.The Death Receptor 3-TNF-like protein 1A pathway drives adverse bone pathology in inflammatory arthritis.The death receptor 3/TL1A pathway is essential for efficient development of antiviral CD4⁺ and CD8⁺ T-cell immunity.Conditional Fas-associated death domain protein (FADD): GFP knockout mice reveal FADD is dispensable in thymic development but essential in peripheral T cell homeostasis.Sustained TL1A expression modulates effector and regulatory T-cell responses and drives intestinal goblet cell hyperplasia.Regulation of early cartilage destruction in inflammatory arthritis by death receptor 3The TNF-family cytokine TL1A promotes allergic immunopathology through group 2 innate lymphoid cells.The death domain of FADD is essential for embryogenesis, lymphocyte development, and proliferation
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P2860
DR3 regulates negative selection during thymocyte development
description
2001 nî lūn-bûn
@nan
2001 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
name
DR3 regulates negative selection during thymocyte development
@ast
DR3 regulates negative selection during thymocyte development
@en
DR3 regulates negative selection during thymocyte development.
@nl
type
label
DR3 regulates negative selection during thymocyte development
@ast
DR3 regulates negative selection during thymocyte development
@en
DR3 regulates negative selection during thymocyte development.
@nl
prefLabel
DR3 regulates negative selection during thymocyte development
@ast
DR3 regulates negative selection during thymocyte development
@en
DR3 regulates negative selection during thymocyte development.
@nl
P2093
P2860
P1476
DR3 regulates negative selection during thymocyte development
@en
P2093
P2860
P304
P356
10.1128/MCB.21.10.3451-3461.2001
P407
P577
2001-05-01T00:00:00Z